Tony Pantalleresco Radio Show Notes : Show of March 22nd 2014
In this show, Tony covers to following topics:
Recent U.S. snowstorms found to contain elements of entomological warfare
Epigenetic changes could explain type 2 diabetes
Top 5 reasons sprouted garlic is good for your health
The Gut Microbiome and Cancer
The photoprotective and antioxidative properties of luteolin are synergistically augmented by tocopherol and ubiquinone
Active extracts of black tea (Camellia Sinensis) induce apoptosis of PC-3 prostate cancer cells via mitochondrial dysfunction
Recent U.S. snowstorms found to contain elements of entomological warfare
The U.S. is now a designated military testing ground for real-time biological and entomological warfare applications using Americans as test subjects under public law 105-85
“Entomological warfare (EW) is a type of biological warfare that uses insects to attack the enemy. The concept has existed for centuries and research and development have continued into the modern era. EW has been used in battle by Japan and several other nations have developed and been accused of using an entomological warfare program.” – –[F16](INTELLIHUB) —Reporting on the “fake snow” or “polymer snow” that has been found in several U.S. cities, including Atlanta, which was mocked by some including “truther” Mark Dice, who wrote it off as a joke early on.[F17]—However, after researching nearly one-hundred accounts or claims of this “fake snow” that seemly won’t melt, burning oddly under flame, smelling like plastic– There is specualtion that there is something else to this issue and that we are not being told the real truth about the recent cold spells.—So far what we do know is that strange polymer fibers have been found in the snow in several states.–We know that snow recently found in several parts of the eastern U.S. smells like burnt plastic when melted under a flame or in a pan (using no butane).[F18] Samples of the snow have even been taken and dried by Henning Kemner, a researcher who posts videos on YouTube, under controlled circumstances. So far, at least one test conducted by Kimner using 2 pounds of fresh snow has shown a rather impressive yield of an ample amount of a dry white powdery residue which Kemner plans to get analyzed spectrally.–However, even to my surprise, new findings posted on YouTube show that America may be under attack by entomological warfare as vast quantities of fleas [insects] have now been found in new snowfall following low-altitude passes by aircraft in Craig, Montana.–Shockingly, this matches up with diabolical tests conducted on human beings by Biological Warfare Unit #731 during the World War II era.–sadly, Don Tow writes—The largest Japanese biological/chemical warfare laboratory was in Ping Fan, a small village near the city of Harbin, Heilongjiang Province in northeast China, known as Unit 731. Unit 731 was a gigantic complex covering six square kilometers and consisted of more than 150 buildings, with living quarters and amenities for up to 3,000 Japanese staff members, 300-500 of whom were medical doctors and scientists. The complex contained various factories. It had 4,500 containers for raising fleas, six giant cauldrons to produce various chemicals, and around 1,800 containers to produce biological agents. Approximately 30 kg of bubonic plague bacteria could be produced there in several days. Especially in the area of biological weapons, Unit 731 could be considered to be the largest such laboratory ever in the world. Not only that it was state of the art, it significantly extended the state of the art, partially because the Japanese had no reluctance at all to experiment with live patients, including doing autopsies while the victims were still alive.
You see under public law substantiated in 1997, all of this has been made possible, allowing the testing of chemicals and biological agents on “Civilian population” as pointed out in my documentary film SHADE the Motion Picture.
Public Law 105–85 105th Congress
PUBLIC LAW 105-85- NOV. 18, 1997: USE OF HUMAN SUBJECTS FOR TESTING OF CHEMICAL OR BIOLOGICAL AGENTSSEC. 1078. RESTRICTIONS ON THE USE OF HUMAN SUBJECTS FOR TESTING OF CHEMICAL OR BIOLOGICAL AGENTS.(a) PROHIBITED ACTIVITIES. – The Secretary of Defense may not conduct (directly or by contract)(1) any test or experiment involving the use of a chemical agent or biological agent on a civilian population; or(2) any other testing of a chemical agent or biological agent on human subjects.(b) EXCEPTIONS.- Subject to subsections (c), (d), and (e), the prohibition in subsection (a) does not apply to a test or experiment carried out for any of the following purposes:(1) Any peaceful purpose that is related to a medical, therapeutic, pharmaceutical, agricultural, industrial, or research activity.[F19]
(2) Any purpose that is directly related to protection against toxic chemicals or biological weapons and agents.
(3) Any law enforcement purpose, including any purpose related to riot control.
So section (a) prohibits these cruel and inhumane chemical and biological tests on humans. Then section (b) says that the prohibitions in section (a) do not apply to tests carried out for virtually any purpose. So section (b) completely negates the prohibitions of section (a).
Epigenetic changes could explain type 2 diabetes
People with type 2 diabetes have epigenetic changes on their DNA that healthy individuals do not have. This has been shown in a major study by researchers at Lund University. The researchers also found epigenetic changes in a large number of genes that contribute to reduced insulin production.–“This shows that the risk of developing type 2 diabetes is not only genetic, but also epigenetic,” said Charlotte Ling, who led the study.–Epigenetic changes occur as a result of factors including environment and lifestyle, and can affect the function of genes. Charlotte Ling and her colleagues have analysed insulin- producing cells of both healthy individuals and patients with type 2 diabetes. The analysis revealed epigenetic changes in approximately 800 genes in those with type 2 diabetes. Over 100 of the genes also had altered expression and many of these could contribute to reduced insulin production. Reduced insulin production is one of the underlying causes of type 2 diabetes.—In order to work out which is the chicken and which is the egg, i.e. whether the epigenetic changes are a consequence of the disease or if the disease is a result of the changes, the researchers also investigated whether healthy individuals had epigenetic changes caused by age, BMI and raised blood sugar levels.–“We were able to observe that a number of epigenetic changes had already taken place in healthy subjects as a result of age or high BMI, and were therefore able to conclude that these changes could contribute to the development of the disease,” said Charlotte Ling. “Unlike genes that can’t be changed, epigenetic changes are reversible,” added Tasnim Dayeh, first author of the publication in PLOS Genetics.–Drugs that cause epigenetic changes have long been used in the treatment of cancer and epilepsy. The new survey changes the view of epigenetics in relation to diabetes, according to Charlotte Ling.–“It shows that epigenetics is of major significance for type 2 diabetes, and can help us to understand why people develop the condition. This also opens the way for the development of future drugs.”—Story Source–The above story is based on materials provided by Lund University. —Journal Reference–Tasnim Dayeh, Petr Volkov, Sofia Salö, Elin Hall, Emma Nilsson, Anders H. Olsson, Clare L. Kirkpatrick, Claes B. Wollheim, Lena Eliasson, Tina Rönn, Karl Bacos, Charlotte Ling. Genome-Wide DNA Methylation Analysis of Human Pancreatic Islets from Type 2 Diabetic and Non-Diabetic Donors Identifies Candidate Genes That Influence Insulin Secretion. PLoS Genetics, 2014; 10 (3): e1004160 DOI: 10.1371/journal.pgen.1004160
Sprouting Garlic — it has heart-healthy antioxidants
“Garlic Sprouting Is Associated with Increased Antioxidant Activity and Concomitant Changes in the Metabolite Profile”
Journal of Agricultural and Food Chemistry
Scientists are reporting in ACS’ Journal of Agricultural and Food Chemistry that this type of garlic has even more heart-healthy antioxidant activity than its fresher counterparts.–Jong-Sang Kim and colleagues note that people have used garlic for medicinal purposes for thousands of years. Today, people still celebrate its healthful benefits. Eating garlic or taking garlic supplements is touted as a natural way to reduce cholesterol”Click to Continue > by Isaver” levels, blood pressure and heart disease risk. It even may boost the immune system and help fight cancer. But those benefits are for fresh, raw garlic. Sprouted garlic has received much less attention. When seedlings grow into green plants, they make many new compounds, including those that protect the young plant against pathogens. Kim’s group reasoned that the same thing might be happening when green shoots grow from old heads of garlic. Other studies have shown that sprouted beans and grains have increased antioxidant activity, so the team set out to see if the same is true for garlic.—They found that garlic sprouted for five days had higher antioxidant activity than fresher, younger bulbs, and it had different metabolites, suggesting that it also makes different substances. Extracts from this garlic even protected cells in a laboratory dish from certain types of damage. “Therefore, sprouting may be a useful way to improve the antioxidant potential of garlic,” they conclude.
Top 5 reasons sprouted garlic is good for your health
A number of people believe that vegetables and fruits that have sprouted are bad for health as they release chemicals that are toxic for the body. While this might be true for certain fruits and vegetables it does not apply to garlic. That might not necessarily be true, especially in the case of sprouted garlic. While unsprouted garlic is known to help reduce cholesterol”Click to Continue > by Isaver”, keep heart disease at bay, beat cancers, heal skin infections and even help in decongesting a clogged up nose; sprouted garlic has now been found to have amazing benefits for your heart health. Read more about the health benefits of garlic. –According to a study published in the Journal of Agricultural and Food Chemistry  sprouted garlic actually has stronger health benefits than unsprouted ones. They conjectured that the pod when sprouted for five days showed the maximum ability to help the human body. -Wondering how it helps? Here are the health benefits of eating sprouted garlic:
Can fight against cancer: The process of sprouting in garlic stimulates the production of phytochemicals, that have amazing properties like the ability to block the further spread of malignant cancer cells and inhibit the activity of carcinogens (cancer causing chemicals) on the body. Apart from that, garlic also produces a large amount of anti-oxidants that are very useful in scavenging free”Click to Continue > by Isaver” oxygen radicals – one of the main reasons for the formation of cancers.
Protects your heart: The same way that phytochemicals block the activity of carcinogens, they also boost enzyme activity and block the activities that leads to plaque formation – an important factor in the formation of blockages in the heart. This thereby helps protect you from suffering from heart disease and heart attacks.
Can prevent strokes: Garlic on its own is a rich source of anjoene a substance that prevents the formation of blood clots. It also is packed with nitrites – a compound that helps dilate (or widen) the arteries. Both these activities in tandem help prevent the onset of strokes (a condition caused by the formation of a blood clot in the blood vessels of the brain). How is sprouted garlic better in preventing strokes? Well during sprouting the amount of phytochemicals present in the garlic pods helps not only enhance the activity of the already present chemicals in garlic but it also blocks the activity of blood clot forming chemicals – making it a better agent against strokes.
Helps prevent wrinkles”Click to Continue > by Isaver” and premature ageing: We all know that antioxidants help prevent premature ageing by scavenging free”Click to Continue > by Isaver” radicals in our body – the leading cause of ageing in our body. But according to the study published in the Journal of Agricultural and Food Chemistry, garlic pods sprouted for five days have the highest antioxidant activity – making them an elixir for your body. Not only will it prevent the appearance of wrinkles”Click to Continue > by Isaver” but also has the potential to stop the deterioration of organs (something that takes place as we age).
Strengthens your immune system: If you suffer from frequent cold and cough or infections its time you ate some sprouted garlic. That is because sprouted garlic – especially those that have been sprouted for five days – gives your body a strong dose of anti-oxidants. These anti-oxidants in turn strengthen your immune system by protecting the very cells that kill off infections. So, if you want to stay healthy all year round, here is your chance”Click to Continue > by Isaver” – load up on sprouted garlic.
How to sprout garlic
Garlic normally sprouts on its own if left in the open. But if you want to sprout them at home without drying them out here is how you can do it:–Take a pod of garlic and without peeling it gently poke in two tooth picks on either side of the garlic. Make sure you place the toothpicks on the more fleshy part of the pod. Now take a small and narrow glass cup and fill it with water (tap water should do. Do not add anything to the water). Next, using the two toothpicks as stands, balance the garlic pod on the rim of the glass, such that the narrow part is partly submerged in the water.–Keep this glass on a window sill where it will get enough sunlight. Allow it to stay there for five days, making sure the tip is submerged in the water. Once it is sprouted you can wash and eat the garlic
The Gut Microbiome and Cancer
Our gut microbiota influence cancer susceptibility of our digestive tract as well as distant organs including the skin, lungs, breasts and liver
Dr Eva Sirinathsinghji
Cancer is a leading disease in industrialised nations and the incidence is
rising sharply in developing nations as a result of aging demographics,
“westernised” diets, exposure to chemical carcinogens and inactivity. Though
there are genetic causes for certain cancers that give a significant risk to the
individual carrier e.g. BRCA1 mutations and breast cancer, most cancers are
linked to environmental factors. Despite this overriding environmental determinant in cancer susceptibility, we still know relatively little about the environmental factors involved. Recent research on the gut microbiota reveals a complex and important role that this ‘forgotten organ’ may play, not only in preventing or promoting carcinogenesis, but also in modifiying the efficacy of different therapies.–Clues that the bugs in our guts may influence cancer development have been around at least since the late 19th century when anti-tumour effects were observed in sarcoma patients after bacterial infections or following the
injection of heat-killed bacteria (Coley’s toxin) [1, 2]. In the 1970s, studies
of germ-free mice suggested tumour-promoting effects of the microbiota in spontaneous, carcinogen-induced and genetically-induced cancer models. Indeed, there is now a well known association of the gut microbiota with inflammation and metabolism, two hallmarks of cancer, with germ-free animals displaying decreased weight gain and resistance to obesity, hypoglycaemia and low insulin. With the advent of metabolomics and deep sequencing techniques, researchers are beginning to decipher the role of specific microbes as well as specific global microbiotic profiles associated with different cancers. These discoveries are leading to new avenues of research into cancer prevention and treatment.
One bug, two bugs, all or none?
The relationship between our gut microbiota and cancer appears to be complex,
involving both specific microbial species as well as dysregulation of the global
microbiota, called dysbiosis. Epidemiological studies have linked a number of
cancers to individual microbes, e.g. the human papillomavirus (HPV) in cervical
cancer; H.pylori in gastric cancer; chronic infection in hepatitis B virus
(HBV); hepatitis c virus (HCV) in hepatocellular carcinoma (HCC); chronic
Salmonella enterica subsp infection in gallbladder cancer; infection with
Chlamydia pneumonia in the development of lung cancer; and infection with
Haemophilus influenza and Candida albicans in the development of lower
respiratory tract malignancies [3-6]. The clearest example for bacterially
driven carcinogenesis is H.pylori infection, with epidemiological studies
suggesting it is responsible for 1-3 % of gastric cancer cases of H.pylori
infected individuals. This microbe has been widely defined as a carcinogen by public health institutions including the International Agency for Research on Cancer .—Most of the microbiome differences seen in cancer studies involve dysbiosis of the overall microbial community. Even in those ‘one microbe –one disease’ cases, the story appears more complicated than it seems. With H. pylori infection and gastric cancer, mice colonised with this species alone develop fewer gastric tumours than those colonised with a complex mixture of species . It is interesting to note that H.pylori is also linked to a lower risk of oesophageal adenocarcinoma in humans, emphasising the organ-specific effect of bacterial communities on carcinogenesis . The emerging understanding of colorectal cancer is similarly complex, with metagenomic studies showing underrepresentation of two bacterial phyla, Bacteroidetes and Firmicutes as well as overrepresentation of the invasive Fusobacterium nuculeatum species (previously associated with periodontis and appendicitis) in the tumours [10, 11]. Even though there were consistent patterns of dysbiosis between the patients in these studies, the overall microbiotas between tumours and noncancerous regions of the colon of individual patients were more similar to
each other than they were to tumours of other patients or colon samples from unaffected patients. This intricate association with cancer suggests that any therapy involving the gut microbiota many rely on individualised therapies for successful treatment. Furthermore, these associations to do not prove causation so further analysis is much needed. Dysbiosis of the gut microbiota has also been linked to cancers of distant organs, exemplified by cancers of the liver and pancreas, which do not have a known microbiota of their own. The role of the gut microbiota therefore appears wide-reaching, affecting much more than the gut itself [12-14].
How do microbiota influence cancer susceptibility?
There are many mechanisms by which gut microbiota may alter susceptibility to
cancers including activation of the innate immune system, modulation of
inflammation, influencing gene expression as well as the genomic stability of
host cells. A failure of the intestinal barrier to limit host-microbiota
interactions is also thought to be important. Anatomical separation between the host and microbes is a crucial first line of defence and is maintained through an intact epithelial lining and mucosal layer, as well as a sensing system that detects and eliminates bacteria. Consistently, ulcerative colitis, a condition that disrupts the barrier, increases the risk of colon cancers. Studies that have induced barrier failure in lab animals have also shown that carcinogens are more likely to pass through a disrupted gut lining, leading to increased tumour formation in local and distant organs 
The photoprotective and antioxidative properties of luteolin are synergistically augmented by tocopherol and ubiquinone.
Planta Med. 2013 Jul;79(11):963-5
Authors: Wölfle U, Haarhaus B, Schempp CM
Ultraviolet radiation induces DNA damage and oxidative stress which can result in skin inflammation, photoaging, and photocarcinogenesis. [F20]The flavonoid luteolin that is present in high amounts in the dyers weld, Reseda luteola, is one of the most potent antioxidative plant metabolites and also has ultraviolet-absorbing properties.The aim of this study was to determine whether tocopherol and ubiquinone add synergistic antioxidative values to luteolin. None of the substances showed cytotoxic effects in concentrations from 0.25 to 4 µg/mL. The photoprotective and antioxidant effect of equivalent concentrations of luteolin, tocopherol, and ubiquinone and their combination in a ratio of 4 : 4 : 1 were studied in solar simulator irradiated human skin fibroblasts. Luteolin had a half-maximal radical scavenging concentration of 2 µg/mL, whereas tocopherol and ubiquinone were only effective at higher concentrations. None of the substances showed a phototoxic effect, and only luteolin had a moderate photoprotective effect at 2 µg/mL. The combination of luteolin, tocopherol, and ubiquinone exerted a synergistic radical scavenging effect already at a concentration of 0.25 µg/mL and a complete photoprotection at 2 µg/mL.In summary, our findings suggest that the potent antioxidant and photoprotective effect of flavonoids like luteolin may be further increased by the addition of low concentrations of other antioxidants such as tocopherol and ubiquinone. –PMID: 23839819 [PubMed – indexed for MEDLINE]
Using things like hawthorn-rosemary or sage which has a good source of Luteolin by fusing this in oil and then add Cq 10 and vitamin E ( fuse in either almond oil or olive oil which has vitamin E in them naturally) and use –the ratio is 4:4:1 4 parts luteolin-4 parts vitamin E and 1 part cq 10
100ml would be 4 ml of luteolin- 4 mil Vitamin E 1 mil of Cq 1o
Active extracts of black tea (Camellia Sinensis) induce apoptosis of PC-3 prostate cancer cells via mitochondrial dysfunction.
Oncol Rep. 2013 Aug;30(2):763-72
Authors: Sun S, Pan S, Miao A, Ling C, Pang S, Tang J, Chen D, Zhao C
Cancer of the prostate gland is the most common invasive malignancy and the second leading cause of cancer-related death in human males. Many studies have shown that black tea reduces the risk of several types of cancer. We studied the effects of active extracts of black tea and the black tea polyphenols theaflavins (TFs), on the cellular proliferation and mitochondria of the human prostate cancer cell line PC-3. Our studies revealed that Yinghong black tea extracts (YBT), Assam black tea extracts (ABT) and TFs inhibited cell proliferation in a dose-dependent manner. We also showed that TFs, YBT and ABT affected the morphology of PC-3 cells and induced apoptosis or even necrosis in PC-3 cells. In addition, it was observed that the samples significantly caused loss of the mitochondrial membrane potential, release of cytochrome c from the intermembrane space into the cytosol, decrease of the ATP content and activation of caspase-3 compared with the control. Taken together, these findings suggest that black tea could act as an effective anti-proliferative agent in PC-3 cells, and TFs, YBT and ABT induced apoptosis of PC-3 cells through mitochondrial dysfunction.—PMID: 23715786 [PubMed – indexed for MEDLINE]
Recipe—Either extract the black Tea in alcohol ( wine –brandy-cognac-vodka or a clear based rum or ethanol in a clear base) or in water and use the teas throughout the day or the use of the tinctures in 10-30 drop increments