Tony Pantellaresco Show Notes For The Week June 18 2012

Today’s Environment Influences Behavior Generations Later:

Chemical Exposure Raises Descendants’ Sensitivity to Stress

 

Genetic Mutation in African Malaria Parasite Shown to Give Resistance to Best Drugs

 

Chernobyl Deaths Top a Million Based on Real Evidence

 

Estriol- Benefits

New protein study could shake up sports nutrition market-Or Kill It!

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Today’s Environment Influences Behavior Generations Later:

 

Chemical Exposure Raises Descendants’ Sensitivity to Stress

Researchers have seen an increased reaction to stress in animals whose ancestors were exposed to an environmental compound generations earlier. —ScienceDaily (May 21, 2012) — Researchers at The University of Texas at Austin and Washington State University have seen an increased reaction to stress in animals whose ancestors were exposed to an environmental compound generations earlier.—The findings, published in the latest Proceedings of the National Academy of Sciences, put a new twist on the notions of nature and nurture, with broad implications for how certain behavioral tendencies might be inherited.[U1] –The researchers — David Crews at Texas , Michael Skinner at Washington State and colleagues — exposed gestating female rats to vinclozolin, a popular fruit and vegetable fungicide known to disrupt hormones and have effects across generations of animals.[U2] The researchers then put the rats’ third generation of offspring through a variety of behavioral tests and found they were more anxious, more sensitive to stress, and had greater activity in stress-related regions of the brain than descendants of unexposed rats.—We are now in the third human generation since the start of the chemical revolution, since humans have been exposed to these kinds of toxins,” says Crews. “This is the animal model of that.”—“The ancestral exposure of your great grandmother alters your brain development to then respond to stress differently,” says Skinner. “We did not know a stress response could be programmed by your ancestors’ environmental exposures.”[U3] The researchers had already shown exposure to vinclozolin can effect subsequent generations by affecting how genes are turned on and off, a process called epigenetics. In that case, the epigenetic transgenerational inheritance altered how rats choose mates.[U4] The new research deepens their study of the epigenetics of the brain and behavior, dealing for the first time with real-life challenges like stress. It also takes a rare systems biology approach, looking at the brain from the molecular level to the physiological level to behavior.—“We did not know a stress response could be reprogrammed by your ancestors’ environmental exposures,” says Skinner, who focused on the epigenetic transgenerational inheritance and genomics aspects of the paper. “So how well you socialize or how your anxiety levels respond to stress may be as much your ancestral epigenetic inheritance as your individual early-life events.” This could explain why some individuals have issues with post-traumatic stress syndrome while others do not, he says. Crews says that increases in other mental disorders may be attributable to the kind of “two-hit” exposure that the experiment is modeling.[U5] “There is no doubt that we have been seeing real increases in mental disorders like autism and bipolar disorder,” says Crews, who focused on the neuroscience, behavior and stress aspects of the paper. “It’s more than just a change in diagnostics. The question is why? Is it because we are living in a more frantic world, or because we are living in a more frantic world and are responding to that in a different way because we have been exposed? I favor the latter.” The researchers also saw intriguing differences in weight gain, opening the door to further research on obesity. Story Source-The above story is reprinted from materials provided by Washington State University. The original article was written by Eric Sorensen. -Journal Reference-David Crews, Ross Gillette, Samuel V. Scarpino, Mohan Manikkam, Marina I. Savenkova, and Michael K. Skinner. Epigenetic transgenerational inheritance of altered stress responses. Proceedings of the National Academy of Sciences, May 21, 2012 DOI: 10.1073/pnas.1118514109

 

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Genetic Mutation in African Malaria Parasite Shown to Give Resistance to Best Drugs

 

ScienceDaily (Apr. 27, 2012) — Scientists have identified genetic mutations[U6] in the deadliest malaria parasite in Africa that are giving it resistance to one of the most powerful anti-malarial drugs. The researchers say their findings are a further warning that the best weapons against malaria could become obsolete.–The artemisinin group of drugs are the most effective and widely used treatments for malaria. They are most powerful and less likely to be resisted by the malaria parasite when used with other drugs as artemisinin-based combination therapies (ACTs). But the new study confirms previous suggestions that mutations in a key part of the parasite can provide resistance to artemether, one of the two most effective artemisinins.—The research group, led by a team at St George’s, University of London, discovered artemether resistance in parasite samples taken from 11 of the 28 malaria-infected patients in the study. On average, artemether’s effectiveness was reduced by half. Each parasite was found to have the same genetic mutations.[U7] —The patients were infected by malaria parasite-carrying mosquitoes while travelling abroad, mostly in sub-Saharan Africa, home to 90 per cent of the one million people killed worldwide each year by malaria.—Study lead Professor Sanjeev Krishna said: “Artemether and ACTs are still very effective, but this study confirms our fears of how the parasite is mutating to develop resistance. Drug resistance could eventually become a devastating problem in Africa, and not just in south east Asia where most of the world is watching for resistance. Effective alternative treatments are currently unaffordable for most suffering from malaria[U8] . Finding new drugs is, therefore, crucial.” In the study, published online April 27, 2012 in BioMed Central’s open access journal Malaria Journal, the researchers tested samples from patients infected with the Plasmodium falciparum parasite. This parasite causes the deadliest form of malaria, and is responsible for nine out of 10 malaria deaths. [U9] The parasites were assessed for their sensitivity to four artemisinins — artemisinin itself, artemether, dihydroartemisinin and artesunate.—The 11 parasites showing artemether resistance had the same genetic mutations in an internal system called the calcium pump. This is used to transport calcium, crucial for the parasite to function. The researchers already suspected that the calcium pump — which they first showed was a target for artemisinins to work on in 2003 — had the potential to develop artemisinin resistance. But this had been difficult to confirm until now.—Artemether resistance was strongest in several cases where a separate mutation in another transport system — a protein called pfmdr1, already associated with drug resistance — also occurred.—The effectiveness of the other artemisinins was not significantly affected by the mutations. This may be because they were able to work on other transport systems in the parasite, compensating for the effects of resistance mutations in the calcium pump.—However, Professor Krishna added: “At the moment, we do not know if the other artemisinins will follow suit, but given the shared chemistry they have with artemether it is tempting to think that they would.”—He added that resistance could be a result of the increasing use of ACTs, 300 million doses of which were dispensed worldwide in 2011. Greater use could offer the parasites more opportunities to develop genetic mutations that provide resistance. This could, the researchers say, lead to a repeat of how the parasite developed resistance to pre-artemisinin drugs such as chloroquine. Incorrect use of anti-malarials, such as not completing the treatment course or taking sub-standard drugs, could aid this process.—Professor Krishna said: “New drug development is paramount, but it is vital that we also learn more about how artemisinins work so we can tailor ACT treatments to be effective for as long as possible.”—-Story Source-The above story is reprinted from materials provided by University of St George’s London, via AlphaGalileo. –Journal Reference-Dylan R Pillai, Rachel Lau, Krishna Khairnar, Rosalba Lepore, Allegra Via, Henry M Staines, Sanjeev Krishna. Artemether resistance in vitro is linked to mutations in PfATP6 that also interact with mutations in PfMDR1 in travellers returning with Plasmodium falciparum infections. Malaria Journal, 2012; 11 (1): 131 DOI: 10.1186/1475-2875-11-131

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Chernobyl Deaths Top a Million Based on Real Evidence
Medical records from contaminated areas speak for themselves; doctors, scientists and citizens bear witness to the devastating health impacts of radioactive fallout from nuclear accidents Dr. Mae-Wan Ho
Official denial by nuclear lobby—The Chernobyl disaster occurred on 26 April 1986 at the Chernobyl Nuclear Power -Plant near the city of Prypiat in Ukraine, then part of the Soviet Union, and close to the administrative border with Belarus.  A sudden power output surge prompted an attempt at emergency shutdown; but a more extreme spike in power output led to the rupture of a reactor vessel and a series of explosions. The graphite moderator was exposed, causing it to ignite, and the resulting fire sent a plume of highly radioactive fallout over large parts of the western Soviet Union and Europe. From 1986 to 2000, 350 400 people were evacuated
and resettled from the most contaminated areas of Belarus, Russia and Ukraine. According to official post-Soviet data, about 57 % of the fallout landed in Belarus [1]. Chernobyl is widely considered to have been the worst nuclear accident in history and one of only two classified as a level 7 event on the International Nuclear Event Scale, the other being the Fukushima Daiichi nuclear meltdown in 2011 (see [2] Fukushima Nuclear Crisis, SiS 50).— From the beginning, the official nuclear safety experts were at pains to minimise the projected health impacts, as they are doing now for the Fukushima accident.  The UNSCEAR (United Nations Scientific Committee on the Effects of Atomic Radiation) estimated a “global collective dose” of radiation exposure from the accident “equivalent on average to 21 additional days of world exposure to natural background radiation”. Successive studies reported by the IAEA (International Atomic Energy Agency) continued to underestimate the level of exposure and to understate health impacts other than [3] “psychosocial effects, believed to be unrelated to radiation exposure” resulting from the lack of information immediately after the accident, “the stress and trauma of compulsory relocation to less contaminated areas, the breaking of social ties and the fear that radiation exposure could cause  health damage in the future.”——The number of deaths attributed to Chernobyl varies widely [1]. Thirty-one deaths are directly attributed to the accident, all among the reactor staff and emergency workers. An UNSCEAR report places the total confirmed deaths from radiation at 64 as of 2008. The Chernobyl Forum [4] founded in February 2003 at the IAEA Headquarters in Vienna with representatives from IAEA and UN agencies including UNSCEAR, WHO,  the World Bank, and Belarus, Russia and Ukraine, estimates that the eventual death toll could reach 4 000 among those exposed to the highest levels of radiation (200 000 emergency workers, 115 000 evacuees and 270 000 residents of the most contaminated areas); the figure includes some 50 emergency workers who died of acute radiation syndrome, 9 children who died of thyroid cancer and an estimated total of 3950 deaths from radiation-induced cancer and leukemia. The Union of Concerned Scientists based in Washington in the United States estimates another 50 000 excess cancer cases among people living in areas outside the most contaminated, and 25 000 excess deaths. A Greenpeace report puts the figure at 200 000 or more. The Russian publication, Chernobyl, by scientists Alexey V. Yablokov, Vassily B Nesterenko, and Alexey V. Nesterenko, translated and published by the New York Academy of Sciences in 2009, concludes that among the billions of people worldwide who were exposed to radioactive contamination from the disaster, nearly a million deaths had already occurred between 1986 and 2004. Most of the deaths were in Russia, Belarus and Ukraine [5] (see Truth about Chernobyl, SiS 47). The report drew on thousands of published papers and internet and printed publications. Those publications and papers, written by leading Eastern authorities, were downplayed or ignored by the IAEA and UNSCEAR. These agencies minimised their estimates by several ploys including [6]—– Underestimating the level of radiation by averaging exposure over a large regions, such as an entire country; so high exposure doses and health statistics of the most contaminated areas are lumped together with the less and least exposed

– Ignoring internal sources of radiation due to inhalation and ingestion of radioactive material from fallout
– Using an obsolete and erroneous model of linear energy transfer due to external sources of ionising radiation
– Not counting diseases and conditions other than cancers
– Overestimating the natural background radiation; today’s ‘background’ has been greatly increased by discharges from nuclear activities including tests of nuclear weapons, use of depleted uranium, and uranium mining
– Suppressing and withholding information from the public.
Nevertheless, the devastating health impacts did not escape the notice of the hundreds of doctors, scientists and other citizens who had to bear witness to the deformities, sicknesses and deaths of exposed babies, children and adults in their care. -Diversity of health impacts and their global extent over generations to come Alexei Yablokov, distinguished academician of the Russian Academy of Sciences in Moscow, spoke at the Scientific and Citizen Forum on adioprotection – From Chernobyl to Fukushima, 11-13 May 2012 in Geneva [7]. He is adamant that the consequences of the Chernobyl disaster can be clearly demonstrated by comparing the states of people’s health in areas receiving different amounts of additional radiation following the accident, instead of one based on average effective dose calculated by the ICRP and UNSCEAR which underestimates the true levels of irradiation. For example, there is a clear difference in mortality rates between highly contaminated provinces and less contaminated provinces of Russia (see Figure 1).  Yablokov is lead author of a massive report, now in its third enlarged 2011 edition [8], which has collated all the available evidence.

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Estriol- Benefits

Estriol, an estrogen that has virtually been ignored by the mainstream medical community, is one of the three principal estrogens produced by the body. Estriol was originally thought to have little significance due to its weak estrogenic activity when compared with estrone and estradiol. Nonetheless, research has found that its weakness may very well be its strength. –Studies suggest that when the lower-potency estrogen, estriol, is administered topically, it does not increase the risk of hormone-dependent cancers of the breast or endometrium (uterine lining).1-3 However, having weaker estrogenic effects does not mean that estriol has none of the benefits that come with more potent estrogens. Studies suggest that estriol reduces symptoms of menopause, such as hot flashes and vaginal dryness, but with a better safety profile compared with more potent estrogens.1,4,5 This makes estriol a better choice for bioidentical hormone-replacement treatment regimes. –That is not all this ‘weak’ hormone is good for! Research suggests that estriol has benefits for bone density, heart health, multiple sclerosis, and postmenopausal urinary tract health.6-12 In this article, we will review the attributes of this ‘weaker’ estrogen, and why this estrogen is currently in the news.

F     The body naturally makes three estrogen hormones—estradiol, estrone, and estriol. Since estriol possesses the weakest estrogenic effects of the three, it has been largely overlooked by the medical community.

  • Many studies show that estriol offers a wealth of potential health benefits—without the dangers that sometimes accompany higher-potency estrogens and synthetic or horse-derived hormones.
  • Studies suggest that estriol helps relieve menopausal symptoms while benefitting bone and urinary tract health. Estriol may also help improve cardiovascular risk factors and even shows promise in reducing the brain lesions of multiple sclerosis.
  • The most reliable way to measure estriol levels is through 24-hour urine collection.
  • Despite abundant evidence to the contrary, the FDA has recently claimed that estriol is not safe. You can act now to help preserve consumers’ access to bioidentical hormones such as estriol by visiting www.homecoalition.org.

Fear of cancer prevents many women from restoring youthful hormone levels. When applied through the topical (transdermal) route, estriol is not associated with increased cancer risk. Other methods women can use to prevent hormone-related cancers include consuming regular amounts of vitamin D,  and Bioflavonoids, Tumeric,Rosemary,Parsley,Dandelion, Hawthorn Berry, Black tea and Black Tea extracts, Celery Root, regulating meat and high-fat dairy intake.—

Estriol and Hormone Replacement Therapy

In addition, several studies suggest that bioidentical estrogen has less health risk when given with low doses of bioidentical progesterone.26,27— if you are on hormone-replacement therapy (HRT) and have never heard of estriol, you might be wondering why not? Before the 1970s, estriol was thought to have significance only during pregnancy we saw the beginning of hormone-replacement therapy with patented equine estrogens such as Premarin® and synthetic progestins as found in Provera®. By the 1990s, one-third of menopausal women were taking Premarin®. Research uncovered the increased incidence of breast cancer, increased risk of blood clotting, and increased cardiovascular risk associated with the use of these horse-derived and synthetic hormones (used in combination in the patented medication Prempro®).13 The medical community began to wonder if using hormones from pregnant horses was such a good idea. In an effort to find a safer alternative, many patients and practitioners began looking into ‘natural’ hormone-replacement treatment using bioidentical hormones, which are identical to those produced naturally within the body. Bioidentical-hormone replacement was pioneered in the 1980s as a treatment for menopause by Dr. Jonathan Wright in Washington state. ——-Interest in estriol increased as it was discovered that estriol was safer than horse-derived and synthetic hormones in relation to cardiovascular health and potentially cancer risk. Unfortunately, many doctors have not adopted its use, and many bioidentical hormone-replacement regimes use only estradiol, a more potent estrogen
with increased associated risks.-
– The benefits of estriol may, in part, be explained by the mixed pro-estrogenic and anti-estrogenic effects of this interesting estrogen hormone. Scientists Melamed et al. investigated the mixture of stimulating and non-stimulating effects posed by estriol upon estrogen receptors. When estriol is given together with estradiol, the estradiol-specific stimulation to cells is decreased. This little-appreciated scientific fact helps to explain how estriol can reduce pro-carcinogenic effects of more powerful estrogens like estradiol. However, when estriol is given alone over a long period of time, it can produce a more complete pro-estrogenic effect, explaining why symptom relief is achieved when menopausal women take estriol.2 Experimental studies suggest that both estriol and tamoxifen (a synthetic anti-estrogen) have protective effects against radiation-induced cancer of the breast[U10] .14— Most of the research cited in this article used oral estrogen as the route of administration. However, for enhanced safety, topical estriol would be a better choice. Several studies have shown that transdermal estrogen confers less health risk as a route of administration than oral estrogen.3,21-25 Clinical experience of many doctors over the past 20-30 years suggests that transdermal estrogen is also more effective for some women. This is largely thought to be due to the ‘first-pass effect’—meaning that orally ingested drugs are often first metabolized in the liver, before having any activity in the body. Orally ingested estrogen hormones are among these drugs that are first metabolized in the liver before exerting their effects in the body. Physicians experienced in hormone replacement often observe that women treated with oral estrogens show high levels of estrogen metabolites in 24-hour urine specimens, suggesting that most of the orally ingested hormones are being excreted.———- In a prospective study funded by the US Army and performed at the Public Health Institute, Berkeley, California, researchers compared estriol levels during pregnancy with breast cancer incidence 40 years later. Results revealed that of the 15,000 women entered in the study, those with the highest levels of estriol relative to other estrogens during pregnancy had the lowest cancer risk. In other words, as the relative level of estriol increased during pregnancy, risk of breast cancer decreased 40 years later. In fact, women with the highest level of estriol during pregnancy had 58% lower risk for breast cancer compared with women who had the lowest serum estriol levels. The authors also noted that Asian and Hispanic women had higher estriol levels compared with other racial groups. Interestingly, Asian and Hispanic women have the lowest breast cancer rates. The authors concluded, “If confirmed, these results could lead to breast cancer prevention or treatment regimens that seek to block estradiol estrogen action using estriol, similar to treatment based on the synthetic anti-estrogen tamoxifen.”15 —In another study, Takahashi et al. studied the safety of estriol treatment for Menopausal symptoms. Fifty-three women with either surgically induced or natural menopause were given 2 mg of oral estriol/day for 12 months. Endometrial and breast assessments done with endometrial biopsy and breast ultrasound, respectively, found normal results in all women. The authors concluded that over a 12-month period, “estriol appeared to be safe and effective in relieving symptoms of menopausal women.”1—In one investigation, 52 postmenopausal women were given 2 mg, 4 mg, 6 mg, or 8 mg/day of oral estriol for six months. In all patients, vasomotor symptoms of menopause (such as hot flashes) were decreased. The most improvement was experienced by women taking the highest dose of 8 mg. There were no signs of endometrial hyperplasia confirmed by endometrial biopsy over the six-month treatment period. Mammograms were obtained on six of the patients who had mammary hyperplasia at the study’s outset, and no further changes were seen.8- Although the oral route of administration of estriol appears relatively safe over the short-term, the transdermal route is preferred for long-term use. For example, Weiderpass et al. found an increased risk of endometrial atypical hyperplasia and endometrial cancer with oral use of estriol, but not with transdermal estriol over at least a five-year period. Compared with no use of estriol, those who took oral estriol for at least five years had a significantly greater risk, compared with individuals who did not take any estriol. Women using topical estriol for at least five years did not have any increased risk.

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New protein study could shake up sports nutrition market-Or Kill It!

A study out this week could add a new player to the protein market that’s long been dominated by whey.–At the Experimental Biology meeting in San Diego on Monday, Blake Rasmussen, PhD, of the University of Texas Medical Branch, presented findings that show a blend of protein sources—50 percent casein, 25 percent whey, 25 percent soy—was superior to whey alone for prolonging muscle building and recovery after exercise.[U11] “Whey protein has been given considerable notice as the gold standard ingredient after exercise to enhance muscle growth,” Rasmussen said. “The main problem with whey is it’s fast digesting—the anabolic response in muscle is only about an hour. We wanted to prolong the anabolic response with other protein sources. We found muscle protein synthesis is elevated for a longer amount of time with a protein blend versus whey protein.”[U12] The combination of protein blends was determined in Rasmussen’s previous preclinical work with rats.–Soy, whey and casein protein are all absorbed at different rates during digestion[U13] . Whey protein is referred to as a “fast” protein because it is rapidly absorbed, between 30 and 60 minutes, Rasmussen said. Soy is an intermediate protein, taking between 60 and 120 minutes to digest. And casein is a slow protein, requiring between three and five hours to digest.-“The combination gives you a quick increase in protein synthesis, and it gets sustained,” said Rasmussen. “It’s a prolonged delivery to muscle that the muscles use for recovery.”-The double-blind, randomized clinical trial followed 19 young adults before and after ingestion of about 19 grams of protein from the blend or about 17.5 grams of whey protein alone.

“Your muscles don’t recover in 30 minutes. It takes at least 24 to 48 hours for your muscles to recover after a resistance exercise,” said Greg Paul, global marketing director for sports nutrition and weight management at Solae, a soy supplier that sponsored the study.[U14]

Not just for athletes anymore?—It was only five years ago when research showed that protein should be an important part of sports nutrition products. Before then, the game was typically provided by the likes of Gatorade-style drinks: with fast carbs and electrolytes such as potassium and sodium.–Whey became the go-to protein source in beverages because it quickly fed muscles. Whey also appears better than soy for producing muscle synthesis because of the presence in whey of the amino acid leucine, which has been shown to uniquely act as a stimulatory signal for muscle protein synthesis. But for athletes and weekend warriors alike, using a blend of protein sources that absorb in the body over time means muscles are being fed until the next meal.

The addition of soy is also important because of soy’s particular properties including as an antioxidant and as an anti-inflammatory, which are both key attributes in muscle building beyond anabolic[U15] effects.

And this could lead to the next great demographic for protein products: the elderly.–“Protein blends are useful for sports nutrition,” said Rasmussen, “but also for those interested in aging and maintaining muscle mass as we age. This could potentially be a great intervention for aging.”[U16] -To date, products targeting elderly nutrition with protein-centric value propositions are few and far between. The trend of aging baby boomers, coupled with research demonstrating the value of protein blends in maintaining muscle mass, ought to be of interest to marketers and product developers. [U17]

 

TOP D


[U1]Again Not Genetic as they perpetrate—but a preconditioning to create the anomaly intended—an experiment of control and dependency and debilitation

[U2]Disruptor of the endocrine system—then what happens when you add several different disruptors of the endocrine system? Sterilization!!

[U3]Imagine the duress our lineage will have as a result of the –soy poisoning—chemtrail poisoning-vaccination poisoning –genetically modified food poisoning-environmental—they will be beyond xenophobic and will be told they have a genetic disposition to XYZ infections or conditions or potential invasion from a pathogen specific to there DNA structure—all lies—a direct result of intervention of a evil kind causing this to occur in 3 generations

[U4]It would appear they would possibly chose as a direct result of those who also would have hade these inherited genes –further compounding the problem and  causing  more issues

[U5]A Binary attack to hit and violate at the same time causing a breakdown

[U6]How interesting —we have a GMM= genetically  modified malaria—doe that not make you go hmmmmm wonder how it got genetically enhanced to resist what once cured it !!?—Is it not interesting that we have a new species of Malaria now—apparently the old one was not killing people off fast enough so now the pharma’ have decided to make this  a better pathogen

[U7]WARNING WARNING—Understand this—Genetic Mutation–

[U8]Interesting—if they had the  money they could be relieved but no money and they are held hostage by the IMF—what a crock

[U9] 90% kill ratio—-am I the only one seeing this is not a natural cause—weoponized malaria??

[U10]Utilizing black tea in a concentrated form  has actually the same effect as tamoxifen without the unwanted side effects

[U11]You never Mix a casein with a Soy they are highly allergenic and will cause all kinds of intestinal break down–The soy itself is an idigestible protein that cause intestinal and pancreatic cancers—avoid this

[U12]This is true—but the issue is how it is blended—the idea that you can mix a good egg or animal protein wih the whey would make this far superior to there method and without the allergens associated  with the Soy

[U13]This is true whey is in and out in about 2 hours casein in and out in about 4-6 hours-Soy can take a month due to the fact it does not digest and in fact causes intestinal disruptions and pancreatic shutdown—so they are right in the blending but wrong in how they blend

[U14]NOW YOU SEE WHO DID THE STUDY- A CONFLICT OF INTREREST SINCE THEY  TRIED TO PATENT SOY-AND THE ISRAELI GOV’T STOPPED THEM FROM ATTAINING THIS AND IN FACT GAVE SOME OF THE HARSHEST CRITIQUE ON SOY AND WHO COULD EAT IT—ESSENTIALLY NO ONE BASED ON THOSE STUDIES

[U15]This is at  best  a fabrication go to the soy links at http://augmentinforce.50webs.com

and read for your self the 4 studies that are there—even the FDA since 1904  knew Soy was toxic and not meant to consume

[U16]If this is followed this will exacerbate an already compromised colon and or pancreas—this advice Should not be followed at all—this was obviously done by someone trying to get ahead at the elderly expense

[U17]This is a form of  euphanasia—food poisoning the elderly

Radioactive buckyballs from Fukushima invade California beaches

NaturalNews) As the fallout – no pun intended – from Japan’s tsunami-damaged nuclear reactors at its Fukushima complex continues to worsen, scientists are now concerned about another related phenomenon that appears to be invading the U.S. West Coast – buckyballs.

What, exactly, are those? According to a recent University of California – Davis study, in this case the term refers to uranium-filled nanospheres which were “created from the millions of tons of fresh and salt water used to try to cool down the three molten cores of the stricken reactors,” said Michael Collins, of EnviroReporter.com – not the small, magnetic balls that cling to each other.

The tiny, stout buckyballs – which resemble soccer balls – are said to have been formed when water hit the super-heated, primarily uranium-oxide, fuel in the damaged reactors at Fukushima. “In this goo buckyballs are formed, loaded with uranium and able to move quickly through water without disintegrating,” said the report.

Worse, radiation readings in L.A. and Santa Monica during a 42-day period between December 2011 and January 2012, “strongly suggest that radiation is increasing in the region including along the coast in Ventura County,” Collins said, adding that readings both he and the Environmental Protection Agency took appeared to indicate the unnatural presence of radiation.

“Southern California is still getting hit by Fukushima radiation at alarmingly high levels that will inevitably increase as the main bulk of polluted Pacific Ocean water reaches North America over the next two years,” he asserted.

Radiation believed to be five times normal levels along parts of West Coast

Collins said he began monitoring for increased levels of radiation within four days after a devastating earthquake-caused tsunami struck the Fukushima complex, heavily damaging at least three of the site’s nuclear reactors. Today, he says, radiation levels at both the Los Angeles and Santa Monica sites are about 5.3 times the norm. The Santa Monica site transmits radiation readings to the L.A. Basin area 24/7, Collins said.

The author noted that more than 1,500 radiation tests have been conducted in the area since the disaster, including samples taken in a commercial airliner.

“[E]ven accounting for higher radiation at higher altitudes, readings were more than five times normal according to the manufacturer of our Inspector Alert nuclear radiation monitor,” Collins wrote.

The U.C. – Davis study said the radioactive buckyballs were hearty and potentially able to “transport uranium over long distances,” being stable and consistent in the absence of peroxide.

While experts in the mainstream media debate whether any toxic levels of radiation are already bombarding the U.S. West Coast or if it is on its way, what is known is that radiation, in some forms, has already made it to American waters.

NaturalNews.com reported May 30 that tuna caught off California’s coastline contained higher-than-normal levels of radiation, though most scientists said and continue to say, such levels are safe. http://www.naturalnews.com/036022_Fukushima_radiation_food_supply.html

“I wouldn’t tell anyone what’s safe to eat or what’s not safe to eat,” Daniel Madigan of Stanford University’s Hopkins Marine Station, told Reuters. “It’s become clear that some people feel that any amount of radioactivity, in their minds, is bad and they’d like to avoid it. But compared to what’s there naturally … and what’s established as safety limits, it’s not a large amount at all.”

Madigan said researchers found higher levels of two radioactive isotopes of the cesium element, 137 – which was present in the eastern Pacific before the disaster at Fukushima Daiichi – and 134, which is caused only by manmade activities and wasn’t present before the tsunami smashed into the plant.

Better off having radioactivity in the ocean?

Ken Buesseler, a Senior Scientist at the Woods Hole Oceanographic Institution, wrote on CNN.com that the fact most of Fukushima’s radioactive fallout landed in the ocean, rather than on land, was actually a good thing.

“In many ways we were fortunate that impacts were largely confined to the ocean. Certainly, the Japanese people continue to feel devastating effects of so large a release within their country, and many people may never be able to return to their homes. But in general the winds during the height of the accident at the Fukushima Daiichi nuclear power plant were blowing offshore,” he said.

“As a result, more than three-quarters of the radioactivity fell on the ocean. This is important, as any that lands on soil remains in place, resulting in the potential for greater human exposure and increased chances of contamination to food supplies and property,” Buesseler continued.

Nevertheless, some radioactive measurements show disturbing trends, he said, such as levels “of radioactivity found in fish are not decreasing and there appear to be hot spots on the seafloor that are not well mapped.”

Sources:

http://www.vcreporter.com

http://www.naturalnews.com/036022_Fukushima_radiation_food_supply.html

http://www.enviroreporter.com

Learn more: http://www.naturalnews.com/036204_Fukushima_radiation_California.html#ixzz1y9xIYMlb

Natural Antioxidant Can Protect Against Cardiovascular Disease

 

ScienceDaily (June 16, 2012) — University of Minnesota Medical School researchers have collaborated with the School of Public Health and discovered an enzyme that, when found at high levels and alongside low levels of HDL (good cholesterol), can dramatically reduce the risk of cardiovascular disease. 

The enzyme — glutathione peroxidase, or GPx3 — is a natural antioxidant that helps protect organisms from oxidant injury and helps the body naturally repair itself. Researchers have found that patients with high levels of good cholesterol, the GPx3 enzyme does not make a significant difference. However, those patients with low levels of good cholesterol, the GPx3 enzyme could potentially be a big benefit. The enzyme’s link to cardiovascular disease may also help determine cardiovascular risk in patients with low levels of good cholesterol and low levels of the protective GPx3.

The new research, published June 16 by PLoS One, supports the view that natural antioxidants may offer the human body profound benefits.

“In our study, we found that people with high levels of the GPx3 enzyme and low levels of good cholesterol were six times less likely to develop cardiovascular disease than people with low levels of both,” said lead author Jordan L. Holtzman, M.D., Ph.D., professor of pharmacology and medicine within the University of Minnesota Medical School. “This GPx3 enzyme gives us a good reason to believe that natural antioxidants like GPx3 are good for heart health.”

The combination of low HDL and low GPx3 affects an estimated 50 million people — one in four adults — in the U.S. This condition can lead to fatal heart attacks and strokes. Researchers continue to look for new ways to better predict who is at risk for these diseases and how patients can limit the impact of the disease once it’s diagnosed.

“It’s important to point out that people should not rush out to their doctors and demand testing for the GPx3 enzyme,” said Holtzman. “But in time, we hope that measuring this enzyme will be a common blood test when determining whether a patient is at risk for cardiovascular disease, including heart attacks and strokes.”

To arrive at his results, Holtzman and his colleagues studied the three major risk factors for cardiovascular disease: hypertension, smoking and high cholesterol. Data suggests that those with low levels of HDL and GPx3 were six times more likely to die from cardiovascular disease, including heart attack or stroke, than those with low levels of HDL and high levels of GPx3.

The study examined 130 stored samples from the Minnesota Heart Survey from participants who died of cardiovascular disease after 5-12 years of follow-up care. The ages of patients studied ranged from 26-85 years old. Their data was compared to 240 control samples.

“This is an important enzyme for people with low HDL cholesterol,” said Holtzman. “We think further research will be important in determining the future role of GPx3 and what drugs may serve to increase its activity in the blood.”

The research reported in this publication was supported by the National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD (RO1-HL23727), the Mayo Chair Endowment, School of Public Health, University of Minnesota (DJ), and grant no. 2005R013 from the Netherlands Heart Foundation, Den Haag, the Netherlands (BB).

Inexpensive Approach to Preventing Type 2 Diabetes Shows Promise

ScienceDaily (June 16, 2012)

A simple, inexpensive method for preventing type 2 diabetes that relies on calling people and educating them on the sort of lifestyle changes they could make to avoid developing the disease has proven effective in a study conducted by researchers at the University of California, San Francisco (UCSF) and the City of Berkeley Department of Public Health.

The study involved 230 people in poor, urban neighborhoods in the San Francisco Bay Area cities of Richmond, Oakland and Berkeley. Contacted by phone about once a month, half of them received specific dietary guidance and other lifestyle counseling. After six months, those who had received the counseling had on average lost more weight, were consuming less fat, were eating more fruits and vegetables and showed more improvements in lowering in their blood triglycerides, a key risk measure for type 2 diabetes.

Described this week in the American Journal of Public Health, the new intervention is specifically designed for urban, poor, and predominantly minority communities. It addresses the need for diabetes prevention interventions in these communities and highlights a simple fact that doctors at UCSF and elsewhere have been repeating for years — that type 2 diabetes is preventable in the first place.

“Diabetes is not something you are necessarily going to get just because it runs in your family,” said Alka Kanaya, MD, an associate professor of medicine at UCSF and one of two senior authors on the study. “It is very preventable, and lifestyle changes can really impact the onset of diabetes.”

“You can do something about it,” said Anita Stewart, PhD, a professor at the UCSF Institute for Health & Aging and the Center for Aging in Diverse Communities who is the other senior author on the paper.

How Lifestyle Changes Can Prevent Diabetes Diabetes is a chronic and complex disease marked by high levels of sugar in the blood that arise due to problems with the hormone insulin, which regulates blood sugar levels. It is usually caused by an inability to produce insulin (type 1) or an inability to respond correctly to insulin (type 2).

A major health concern in the United States, diabetes of all types affect an estimated 8.3 percent of the U.S. population — some 25.8 million Americans — and cost U.S. taxpayers more than $200 billion annually.

In California alone, an estimated 4 million people (one out of every seven adults) have type 2 diabetes and millions more are at risk of developing it. These numbers are poised to explode in the next half century if more is not done to prevent diabetes.

Previous studies have shown that counseling and other lifestyle interventions are effective at preventing type 2 diabetes, but those interventions have generally been designed for clinical settings and include separate sessions with numerous health professionals. This makes them expensive and difficult to scale to large urban populations where diabetes interventions are needed the most.

Many of this country’s urban poor face thin health insurance coverage, low literacy, and low income, and a higher risk of developing type 2 diabetes. These disparities were apparent in the UCSF study. About half of the study population was composed of immigrants, and nearly a quarter had no health insurance. Almost a third said they faced financial hardship, and 22 percent had less than a high-school education.

By focusing on a phone-based solution delivered by the Public Health department staff, the UCSF researchers designed their new intervention specifically as a low-cost community-based approach that would be relevant to poor, minority and low-literacy populations. Similar telephone interventions have been used to reach out broadly to populations in San Francisco and in other cities to spread lifestyle messages related to hypertension, smoking, high cholesterol and other issues.

“This adds to our public health toolkit of ways to do outreach and prevent diabetes,” said Kanaya.

Evening primrose oil: The king’s cure-all

NaturalNews) Evening primrose oil (EPO) comes from the seed of the evening primrose plant and is great for skin disorders such as eczema, psoriasis and acne. EPO is used in pregnancy for preventing preeclampsia, starting and shortening labor and preventing late deliveries. Women use evening primrose oil to fight PMS, breast pain, endometriosis, symptoms of menopause and hot flashes.

Evening primrose is commonly called Oenothera, “The King’s cure-all,” which is reflected in the wide range of its healing attributes that just goes on and on. In addition to the benefits listed above, EPO is used for rheumatoid arthritis, osteoporosis, Raynaud’s syndrome, multiple sclerosis, Sjogren’s syndrome, ADD, alcoholism, Alzheimer’s disease, schizophrenia, asthma, cancer, high cholesterol, chronic fatigue syndrome, diabetic nerve damage, dyspraxia, heart disease, hyperactivity, leg pain, neurodermatitis, obesity and weight loss, whooping cough, and gastrointestinal disorders including ulcerative colitis, irritable bowel syndrome and peptic ulcer disease.

EPO is converted to a hormone-like substance called prostaglandin E1 which has anti-inflammatory properties that act as both a blood thinner and blood vessel dilator.

Meet the legend

Evening primrose is a wildflower that grows throughout North America. The evening primrose plant gets its name from the flowers on it that bloom after sunset or extremely overcast days in the months of June through September. In North America, the use of Evening Primrose goes back to early Native Americans who used the seeds for food and made poultices from the whole plant to speed healing in wounds and bruises. European settlers took the root back to England and Germany and used it as a food plant. Evening primrose oil is extracted from the seeds of the evening primrose plant and EPO is an excellent source of the essential fatty acid, gamma-linolenic acid (GLA).

EPO as a medicine

At one time EPO was even an approved medicine used to treat eczema and breast pain. Wouldn’t you know that being a natural and un-patentable “drug” it didn’t last long before the Medicines Control Agency withdrew the licenses for using evening primrose oil products as prescriptions for medical treatments.

In a controlled study at the Center for Rheumatic Diseases, patients received 540 mg of EPO daily which brought less inflammation and significant improvement over 12 months compared to no improvement in the placebo group. By 12 months the patients receiving EPO and EPO/fish oil had significantly reduced or stopped their NSAID pain relievers while measures of disease activity did not worsen. Once the patients went off the EPO they all relapsed.

EPO has been studied for treating eczema, reducing symptoms of diabetic neuropathy, reducing symptoms from Raynaud’s phenomenon and it has been found to improve blood flow and nerve function by lowering blood lipid risk factors.

EPO available forms

The peppery young roots of evening primrose can be eaten like a vegetable or the shoots can be eaten as a salad. Evening primrose oil is extracted from the seeds of the plant which contain up to 25 percent essential fatty acids belonging to the omega-6 family of fatty acids. Most North Americans get too much omega-6 fatty acids in their diet and the body needs a balance of omega-6 and omega-3 fatty acids (found in fish oil) to stay healthy.

Taking EPO is a wise supplement choice. EPO is available in a liquid or in capsule form and should be kept refrigerated and out of direct sunlight to prevent rancidity. The higher- quality oil will be certified organic, bottled in light-resistant containers, refrigerated, and marked with a freshness date. The better EPO is standardized to contain 8 percent gamma-linolenic acid.

Sources for this article

http://www.umm.edu
Sinn N, Bryan J. Effect of Supplementation with Polyunsaturated Fatty Acids and Micronutrients on Learning and Behavior Problems Associated with Child ADHD. J Dev Behav Pediatr 2007.
http://www.ncbi.nlm.nih.gov/pubmed/3529370?dopt=Abstract
http://www.ncbi.nlm.nih.gov/pubmed/2833184?dopt=Abstract
http://www.ncbi.nlm.nih.gov/pubmed/12442909

About the author:
Craig Stellpflug is a Cancer Nutrition Specialist, Lifestyle Coach and Neuro Development Consultant at Healing Pathways Medical Clinic, Scottsdale, AZ. http://www.healingpathwayscancerclinic.com/ With 17 years of clinical experience working with both brain disorders and cancer, Craig has seen first-hand the devastating effects of vaccines and pharmaceuticals on the human body and has come to the conclusion that a natural lifestyle and natural remedies are the true answers to health and vibrant living. You can find his daily health blog at www.blog.realhealthtalk.com and his articles and radio show archives at www.realhealthtalk.com

Learn more: http://www.naturalnews.com/036177_evening_primrose_oil_nutrition_healthy_oils.html#ixzz1xrJvHy42

Link Between Metabolic Disorders and Alzheimer’s Disease Examined

ScienceDaily (June 14, 2012) — No effective treatments are currently available for the prevention or cure of Alzheimer’s disease (AD), the most frequent form of dementia in the elderly. The most recognized risk factors, advancing age and having the apolipoprotein E Ɛ4 gene, cannot be modified or treated. Increasingly, scientists are looking toward other risk factors to identify preventive and therapeutic strategies. Much attention recently has focused on the metabolic syndrome (MetS), with a strong and growing body of research suggesting that metabolic disorders and obesity may play a role in the development of dementia

 

A new supplement to the Journal of Alzheimer’s Disease provides a state-of-the-art assessment of research into the link between metabolic syndrome and cognitive disorders. The supplement is guest edited by Vincenza Frisardi, of the Department of Neurological and Psychiatric Sciences, University of Bari, and the Geriatric Unit and Gerontology-Geriatrics Research Laboratory, IRCCS, Foggia, Italy, and Bruno P. Imbimbo, Research and Development Department, Chiesi Farmaceutici, Parma, Italy.

The prevalence of MetS and obesity has increased over the past several decades. MetS is a cluster of vascular and metabolic risk factors including obesity, hypertension, an abnormal cholesterol profile, and impaired blood glucose regulation. “Although molecular mechanisms underlying the relationship between MetS and neurological disorders are not fully understood, it is becoming increasingly clear that cellular and biochemical alterations observed in MetS may represent a pathological bridge between MetS and various neurological disorders,” explains Dr. Frisardi.

Type 2 diabetes (T2D) has been linked with cognitive impairment in a number of studies. The risk for developing both T2D and AD increases proportionately with age, and evidence shows that individuals with T2D have a nearly twofold higher risk of AD than nondiabetic individuals.

Paula I. Moreira, Faculty of Medicine and Center for Neuroscience and Cell Biology, University of Coimbra, Portugal, outlines some of the likely mechanisms. Both AD and T2D present similar abnormalities in the mitochondria, which play a pivotal role in cellular processes that impair their ability to regulate oxidation in the cell. Human amylin, a peptide that forms deposits in the pancreatic cells of T2D patients, shares several properties with amyloid-ß plaques in the Alzheimer’s brain. Insulin resistance is another feature shared by both disorders. Impairment of insulin signalling is directly involved in the development of tau tangles and amyloid ß (Aß) plaques. “Understanding the key mechanisms underlying this deleterious interaction may provide opportunities for the design of effective therapeutic strategies,” Dr. Moreira notes.

In another article, author, José A. Luchsinger of the Division of General Medicine, Department of Medicine, Columbia University College of Physicians and Surgeons, New York, notes that while there seems to be little dispute that T2D can cause cerebrovascular disease and vascular cognitive impairment, whether T2D can cause late onset AD remains to be determined. “Although the idea is highly speculative, the association between T2D and cognitive impairment may not be causal. Several lines of evidence provide some support to the idea that late onset Alzheimer’s disease could cause T2D, or that both could share causal pathways,” he notes. He reviews epidemiological, imaging, and pathological studies and clinical trials to provide insight. “Given the epidemic of T2D in the world, it’s important to determine whether the association between T2D and cognitive impairment, particularly late onset AD, is causal and if so, what are the mechanisms underlying it.”

Dr. Frisardi notes that most efforts by the pharmaceutical industry have been directed against the production and accumulation of amyloid-ß. “Unfortunately, these efforts have not produced effective therapies yet, since the exact mechanisms of AD are largely unknown. Given that the onset of AD most likely results from the interaction of genetic and environmental factors, the research agenda should consider new platforms of study, going beyond the monolithic outlook of AD, by synthesizing epidemiological, experimental, and biological data under a unique pathophysiological model as a point of reference for further advances in the field.”

Coconut Oil – Here’s what you need to know about this amazing rejuvenation and healing oil

NaturalNews) Coconut oil is a popular nutritional oil derived from the meat of matured coconuts. Coconut has long been a primary source of food throughout the tropics. Its various industrial and cosmetic applications have made it a very viable commodity. Coconut oil is heat stable, making it suitable for cooking at high temperatures. It is slow to oxidize, resists rancidity and has a shelf life of approximately two years or more; virgin coco creme created through a wet-milling process has an indefinite shelf life.
See the NaturalNews InfoGraphic on coconut oil here (it’s beautiful!) http://www.naturalnews.com/Infographic-Benefits-of-Coconut-Oil.html

The composition of coconut oil

The coconut possesses a wide variety of health benefits due to its fiber and nutritional content, but it is the oil that makes it a remarkable source of food and medicine. It has definitely earned its reputation as the healthiest oil in the world despite the fact that its high saturated fat content was once falsely claimed to be unhealthy.
What makes coconut oil different?
Oils and fats are composed of molecules known as fatty acids. They are classified either according to saturation or based on molecular length and size of the carbon chain within each fatty acid. Monounsaturated fats and polyunsaturated fats are an example of the first class.
The second classification is based on molecular size or length of the fatty acid’s carbon chain. Long chains of carbon atoms consist of each fatty acid with an attached hydrogen atom. There are short chain fatty acids known as SCFA, medium chain fatty acids (MCFA) such as coconut oil and long chain fatty acids (LCFA). Whether unsaturated or saturated, the majority of fats and oils in our diet are composed of long chain fatty acids. In fact, a majority of the fatty acids commonly consumed are LCFA.
Coconut oil is predominantly medium-chain fatty acid (MCFA) and the effects of the MCFA in coconut oil are distinctly different from the LCFA found in other foods. In fact, the saturated and unsaturated fat in milk, eggs,meat and even in plants and most vegetable oils are made of LCFA. Why is this relevant? It is important because our bodies respond and metabolize each fatty acid differently. It is the MCFA found in coconut oil that makes it special because these fatty acids do not have a negative effect on cholesterol. In fact, they are known to lower the risk of heart disease and atherosclerosis. There are only few dietary sources of MCFA, and one of the best sources by far is coconut oil.
Theliver and gall bladder do not need to digest and emulsify MCFA, resulting in instant energy, increased metabolic rate and subsequently more heat production as well as increased circulation. Anyone with an impaired fat digestion or removed gallbladder will benefit from coconut oil as this oil is easily digested. (http://www.naturalnews.com/022313_fat_oil_coconut.html)

Lauric Acid

Coconut oil has many health benefits which are attributed to the presence of lauric acid. When it is present in the body, lauric acid is converted into monolaurin, a compound that is highly toxic to viruses, bacteria, funguses and other microorganisms because of its ability to disrupt their lipid membranes and virtually destroy them.
Monolaurin is effective for treating candida albicans, fungal infections and athlete’s foot. It also targets bacterial infections and viruses like measles, influenza, hepatitis C and even HIV. In fact, researchers from the Philippines are studying the effectiveness of lauric acid against HIV/AIDS due to its strong anti-viral properties. Moreover, lauric acid is non-toxic, making it a better alternative to modern drugs that are typically prescribed for viruses as well as fungal and bacterial infections.
Without lauric acid, monolaurin cannot be produced by the body. Breast milk is the only other source of lauric acid, which must explain the lesser incidents of infections with breast-fed infants. It has also been observed that regular consumption of coconut oil boosts immunity and reduces incidences of sickness. (http://www.naturalnews.com)

The health benefits of coconut oil

Hair care – The unique fatty acids in coconut oil have a small molecular structure and pass freely into the hair’s cell membrane, allowing for the oil to penetrate the hair’s shaft; thisliterally brings out the deep conditioning from within compared to other conditioners that work from the outside in.
Massaging the oil into the scalp can offer relief from dandruff. Dandruff is caused by dry skin or an internal fungal condition that reached the scalp. With regular use, coconut oil can kill the fungus and eliminate dandruff issues. For deep hair conditioning, a teaspoon or two on damp hair left for as long as possible can give an ultra-nice shine. Leave it on overnight and see startling results. (http://www.naturalnews.com/029120_coconut_oil_hair_conditioner.html)
Skin care – Coconut oil is an excellent skin conditioner containing medium-chain triglycerides, naturally occurring fats which deeply penetrate, moisturize and acts as a protective barrier against environmental and free radical damage. The oil also provides sun protection by screening 20 percent of ultraviolet exposure.
Coconut oil is rich in anti-oxidants and bursting with the natural microbial and antibacterial agents caphrylic and capric acids. . Its ability to smooth the skin while infusing with anti-oxidants makes it a perfect anti-aging moisturizer. Moreover, it contains vitamin E, another antioxidant popular for hastening the recovery of skin abrasions, burns and other trauma. (http://www.naturalnews.com)
Weight loss – Medium-chain fatty acids found in coconut oil can speed up metabolism faster than long-chain fatty acids because they are easily digested and converted into energy. In fact, a study reported medium-chain fatty acids to be three times more effective in raising metabolism than long-chain fatty acids, leading researchers to conclude that effective weight loss can be achieved by replacing long-chain fatty acids with medium- chain fatty acids. (http://www.naturalnews.com/026808_oil_coconut.html)
Natural remedy for pneumonia – In a study presented before The American College of Chest Physicians on October 29, 2008, coconut oil was found to offer pneumonia patients faster and more complete relief from symptoms. This could be a welcome development for many as this means a reduced stay in the hospital, lower medical expenses and lower exposure for the patient to a hospital environment. Moreover, it is an inexpensive addition to traditional antibiotics and has no known side effects. (http://www.naturalnews.com/025038_coconut_child_oil.html)
Lowers risk of diabetes, heart disease and improves cholesterol levels – In a study made on women subjects ranging from 20 to 40 years old, half of the subjects were instructed to take a 30 ml soybean oil supplement while the other half were instructed to take a 30ml coconut oil supplement while maintaining moderate exercise routine over a 12-week period. Results of the study showed that although both group of women had a decrease in body mass index (BMI), only the women who were taking coconut oil showed a notable decease in waist circumference significantly lowering the risk of conditions like type II diabetes and heart disease.
Furthermore, the study also showed that the subjects who experienced an improvement in their cholesterol profile along with higher HDL levels and higher HDL: LDL ratio were the ones taking coconut oil. Those taking soybean oil did not receive the same benefits but reflected a higher total cholesterol as well as higher LDL cholesterol lower, lower HDL cholesterol and a lower HDL: LDL ratio. (http://www.naturalnews.com/026547_oil_coconut.html)
Assists in bone health and chronic fatigue – Research has found coconut oil to help prevent osteoporosis because it helps in the nutrient absorption of minerals such as calcium and magnesium – important minerals that fight osteoporosis.
Moreover, the medium-chain fatty acids in coconut oil produce energy rather than body fat, thereby improving metabolism and preventing fatigue. The oil has also been shown to destroy organisms in the body that sap its strength and contribute to the condition of fatigue. (http://www.naturalnews.com/033718_food_miracles.html)
Alzheimer’s Disease – Dr. Mary Newport, after failing to get treatment for her husband’s dementia, discovered that coconut oil contained natural medium-chain triglyceride (MCT). The same substance was used in a drug trial her husband failed to qualify for. So, she gave her husband 1 tbsp. of coconut oil twice a day for a month and a half and saw him almost completely recovered. (http://www.naturalnews.com)
Others – aside from the health benefits mentioned earlier the following health benefits have been attributed to the beneficial use of coconut oil:
• Protects against cancer and HIV and other infectious diseases • Kills bacteria and parasites like tape worm and liver flukes • Eases acid reflux, aids in proper bowel function • Lowers incidence of hemorrhoids • Heals and relieves intestinal problems • Soothes earaches • Deals with symptoms connected with prostate enlargement • Strengthens the liver and protects against degeneration • Reduces incidence of epileptic seizures • Reduces joint and muscle inflammation • Eases neuropathies and itching from diabetes. (http://www.naturalnews.com/036028_coconu_oil_health_solutions.html)

How much oil should be taken in to enjoy its benefits?

According to researchers, an adult should consume around 3 1/2 tbsp. of coconut oil daily: an amount equal to the MCFA a nursing infant would receive in one day. The benefits of coconut oil are derived from the nutritional value of medium-chain fatty acids (MCFA’s), and the best comparison in nature as to the percentage of MCFA consumed in a diet is in human breast milk. For those who are not used to having coconut oil in their diet, it is best to start out with a lesser amount and see how the body reacts before following the recommended amount.

Does coconut oil have any adverse side effects?

Coconut oil has no known side effects. However, if you are used to a low-fat diet, a common adverse reaction would be diarrhea. It is probably not advisable to start with a large amount right away. Spreading the recommended amount over the course of one day and building up to a larger dosecan help to avoid unwanted effects.
In coconut-producing countries, it is considered beneficial for pregnant and lactating women to enjoy coconut oil;Westerners used to a low-fat diet, however, are cautioned not to experiment with coconut oil while pregnant if the body is not used to it. If you have been consuming coconut oil regularly with no adverse reactions, there is no reason to discontinue consumption
Learn more: http://www.naturalnews.com/036156_Coconut_oil_superfood_healing.html#ixzz1xoNzvi51

Infographic – Ten ways to use Apple Cider Vinegar

NaturalNews) You may not think of apple cider vinegar as something that is a boon to your overall health, but it can be, and here are 10 ways you can use it to help pave the way to a better you.
See the infographic at: http://www.naturalnews.com
1. Balance your inner ecosystem: The body constantly strives to achieve a state of homeostasis, or a state of equilibrium. One way is by keeping the body’s pH balance (or acid-base balance) within the normal range of 7.35 – 7.45. Apple cider vinegar helps the body maintain a healthy alkaline pH level. Research shows that higher acid levels (lower pH level) leads to a lack of energy, excessive production of mucous and higher incidences of infection, among other things.
2. Detoxes the body: As part of balancing the body’s pH, apple cider vinegar is useful in overall detoxification of the body. Research shows that it can help stimulate cardiovascular circulation and help detoxify the liver. It also helps break up mucous throughout the body and helps cleanse the lymph nodes.
3. Weight loss: Tired of the “fad” diets that do more damage to your wallet than your excess fat cells? Try boosting your intake of apple cider vinegar. Data shows some limited, albeit significant, weight loss benefits from sustained daily intake of acetic acid (which is prevalent in apple cider vinegar). In one study published in Bioscience, Biotechnology, and Biochemistry in 2009, it was found that subjects who consumed acetic acid for 12 weeks experienced significant declines in body weight, abdominal fat, waist circumference and triglycerides.
4. Cures: Allergies: Believe it or not, research suggests that apple cider vinegar can help reduce the effect of allergies on your body, mostly by reducing mucous and sinus congestion. A sign of a distressed immune system is an increase and thickening of mucous; apple cider vinegar can help reduce the build-up and congestion, thereby reducing your susceptibility to allergies.
5. Cures: Sinus infections, sore throats, headaches, flu: Reducing the effects of allergies can also reduce the frequency of sinus infections and the symptoms associated with them, such as sore throats and headaches. Boosting the immune system can also curb bouts of the flu.
6. Clears: Acne, dermatitis, warts and other skin problems: Some practitioners recommend mixing one part apple cider vinegar and three parts water. They recommend dabbing the solution on each pimple. For warts, some recommend soaking a cotton ball in an apple cider vinegar solution then place over the wart with a band-aid overnight. The skin will swell some as it reacts with the solution and may throb; it may even turn black after a few days. Once it falls off, though, continue the treatment for a few more days, to make sure the wart doesn’t return, according to recommendations.
7. Lower blood pressure and cholesterol: One of the most important “healing” qualities of apple cider vinegar is its ability to lower blood pressure and triglycerides, the bad cholesterol. According to a 2001 animal study published in Bioscience, Biotechnology, and Biochemistry, there was some evidence that daily consumption of apple cider vinegar could help reduce both blood pressure and cholesterol. Try 1-2 teaspoons of vinegar in about eight ounces of water; if you want, add a teaspoon of honey to make it more palatable.
8. Kills candida and fungus: This vinegar is rich in natural enzymes that can help rid your body of candida – a genus of yeasts which can cause thrush in humans and is blamed for creating symptoms of fatigue, poor memory, sugar cravings, and yeast infections.
9. Eases arthritis, stiff joints and gout: Regular use of apple cider vinegar can be tied to a decrease in joint pain and gout by reducing inflammation; data suggests the malic acid in the vinegar is able to dissolve the uric acid crystals within the joint and assist in flushing them from the body.
10. Strengthens stomach acid: Though it might seem a little odd to treat stomach acid with an acid-containing vinegar, there is research suggesting that apple cider vinegar works by treating low acid and thereby reducing heartburn. Natural remedy experts say you should begin to feel relief – unless your problem is ulcers – very shortly after taking a teaspoon of apple cider vinegar followed by a glass of water
Learn more: http://www.naturalnews.com/036142_apple_cider_vinegar_reference_graphic.html#ixzz1xoKBzxSb

Show Of The Week June 18 2012

 

Special Note

“I have sworn upon the altar of God eternal hostility against every form of tyranny over the mind of man!!!!!

- Thomas Jefferson “

 

Today’s Environment Influences Behavior Generations Later:

Chemical Exposure Raises Descendants’ Sensitivity to Stress

 

Genetic Mutation in African Malaria Parasite Shown to Give Resistance to Best Drugs

 

Chernobyl Deaths Top a Million Based on Real Evidence

 

Estriol- Benefits

New protein study could shake up sports nutrition market-Or Kill It!

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Today’s Environment Influences Behavior Generations Later:

 

Chemical Exposure Raises Descendants’ Sensitivity to Stress

Researchers have seen an increased reaction to stress in animals whose ancestors were exposed to an environmental compound generations earlier. —ScienceDaily (May 21, 2012) — Researchers at The University of Texas at Austin and Washington State University have seen an increased reaction to stress in animals whose ancestors were exposed to an environmental compound generations earlier.—The findings, published in the latest Proceedings of the National Academy of Sciences, put a new twist on the notions of nature and nurture, with broad implications for how certain behavioral tendencies might be inherited.[U1] –The researchers — David Crews at Texas , Michael Skinner at Washington State and colleagues — exposed gestating female rats to vinclozolin, a popular fruit and vegetable fungicide known to disrupt hormones and have effects across generations of animals.[U2] The researchers then put the rats’ third generation of offspring through a variety of behavioral tests and found they were more anxious, more sensitive to stress, and had greater activity in stress-related regions of the brain than descendants of unexposed rats.—We are now in the third human generation since the start of the chemical revolution, since humans have been exposed to these kinds of toxins,” says Crews. “This is the animal model of that.”—“The ancestral exposure of your great grandmother alters your brain development to then respond to stress differently,” says Skinner. “We did not know a stress response could be programmed by your ancestors’ environmental exposures.”[U3] The researchers had already shown exposure to vinclozolin can effect subsequent generations by affecting how genes are turned on and off, a process called epigenetics. In that case, the epigenetic transgenerational inheritance altered how rats choose mates.[U4] The new research deepens their study of the epigenetics of the brain and behavior, dealing for the first time with real-life challenges like stress. It also takes a rare systems biology approach, looking at the brain from the molecular level to the physiological level to behavior.—“We did not know a stress response could be reprogrammed by your ancestors’ environmental exposures,” says Skinner, who focused on the epigenetic transgenerational inheritance and genomics aspects of the paper. “So how well you socialize or how your anxiety levels respond to stress may be as much your ancestral epigenetic inheritance as your individual early-life events.” This could explain why some individuals have issues with post-traumatic stress syndrome while others do not, he says. Crews says that increases in other mental disorders may be attributable to the kind of “two-hit” exposure that the experiment is modeling.[U5] “There is no doubt that we have been seeing real increases in mental disorders like autism and bipolar disorder,” says Crews, who focused on the neuroscience, behavior and stress aspects of the paper. “It’s more than just a change in diagnostics. The question is why? Is it because we are living in a more frantic world, or because we are living in a more frantic world and are responding to that in a different way because we have been exposed? I favor the latter.” The researchers also saw intriguing differences in weight gain, opening the door to further research on obesity. Story Source-The above story is reprinted from materials provided by Washington State University. The original article was written by Eric Sorensen. -Journal Reference-David Crews, Ross Gillette, Samuel V. Scarpino, Mohan Manikkam, Marina I. Savenkova, and Michael K. Skinner. Epigenetic transgenerational inheritance of altered stress responses. Proceedings of the National Academy of Sciences, May 21, 2012 DOI: 10.1073/pnas.1118514109

 

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Genetic Mutation in African Malaria Parasite Shown to Give Resistance to Best Drugs

 

ScienceDaily (Apr. 27, 2012) — Scientists have identified genetic mutations[U6] in the deadliest malaria parasite in Africa that are giving it resistance to one of the most powerful anti-malarial drugs. The researchers say their findings are a further warning that the best weapons against malaria could become obsolete.–The artemisinin group of drugs are the most effective and widely used treatments for malaria. They are most powerful and less likely to be resisted by the malaria parasite when used with other drugs as artemisinin-based combination therapies (ACTs). But the new study confirms previous suggestions that mutations in a key part of the parasite can provide resistance to artemether, one of the two most effective artemisinins.—The research group, led by a team at St George’s, University of London, discovered artemether resistance in parasite samples taken from 11 of the 28 malaria-infected patients in the study. On average, artemether’s effectiveness was reduced by half. Each parasite was found to have the same genetic mutations.[U7] —The patients were infected by malaria parasite-carrying mosquitoes while travelling abroad, mostly in sub-Saharan Africa, home to 90 per cent of the one million people killed worldwide each year by malaria.—Study lead Professor Sanjeev Krishna said: “Artemether and ACTs are still very effective, but this study confirms our fears of how the parasite is mutating to develop resistance. Drug resistance could eventually become a devastating problem in Africa, and not just in south east Asia where most of the world is watching for resistance. Effective alternative treatments are currently unaffordable for most suffering from malaria[U8] . Finding new drugs is, therefore, crucial.” In the study, published online April 27, 2012 in BioMed Central’s open access journal Malaria Journal, the researchers tested samples from patients infected with the Plasmodium falciparum parasite. This parasite causes the deadliest form of malaria, and is responsible for nine out of 10 malaria deaths. [U9] The parasites were assessed for their sensitivity to four artemisinins — artemisinin itself, artemether, dihydroartemisinin and artesunate.—The 11 parasites showing artemether resistance had the same genetic mutations in an internal system called the calcium pump. This is used to transport calcium, crucial for the parasite to function. The researchers already suspected that the calcium pump — which they first showed was a target for artemisinins to work on in 2003 — had the potential to develop artemisinin resistance. But this had been difficult to confirm until now.—Artemether resistance was strongest in several cases where a separate mutation in another transport system — a protein called pfmdr1, already associated with drug resistance — also occurred.—The effectiveness of the other artemisinins was not significantly affected by the mutations. This may be because they were able to work on other transport systems in the parasite, compensating for the effects of resistance mutations in the calcium pump.—However, Professor Krishna added: “At the moment, we do not know if the other artemisinins will follow suit, but given the shared chemistry they have with artemether it is tempting to think that they would.”—He added that resistance could be a result of the increasing use of ACTs, 300 million doses of which were dispensed worldwide in 2011. Greater use could offer the parasites more opportunities to develop genetic mutations that provide resistance. This could, the researchers say, lead to a repeat of how the parasite developed resistance to pre-artemisinin drugs such as chloroquine. Incorrect use of anti-malarials, such as not completing the treatment course or taking sub-standard drugs, could aid this process.—Professor Krishna said: “New drug development is paramount, but it is vital that we also learn more about how artemisinins work so we can tailor ACT treatments to be effective for as long as possible.”—-Story Source-The above story is reprinted from materials provided by University of St George’s London, via AlphaGalileo. –Journal Reference-Dylan R Pillai, Rachel Lau, Krishna Khairnar, Rosalba Lepore, Allegra Via, Henry M Staines, Sanjeev Krishna. Artemether resistance in vitro is linked to mutations in PfATP6 that also interact with mutations in PfMDR1 in travellers returning with Plasmodium falciparum infections. Malaria Journal, 2012; 11 (1): 131 DOI: 10.1186/1475-2875-11-131

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Chernobyl Deaths Top a Million Based on Real Evidence
Medical records from contaminated areas speak for themselves; doctors, scientists and citizens bear witness to the devastating health impacts of radioactive fallout from nuclear accidents Dr. Mae-Wan Ho
Official denial by nuclear lobby—The Chernobyl disaster occurred on 26 April 1986 at the Chernobyl Nuclear Power -Plant near the city of Prypiat in Ukraine, then part of the Soviet Union, and close to the administrative border with Belarus.  A sudden power output surge prompted an attempt at emergency shutdown; but a more extreme spike in power output led to the rupture of a reactor vessel and a series of explosions. The graphite moderator was exposed, causing it to ignite, and the resulting fire sent a plume of highly radioactive fallout over large parts of the western Soviet Union and Europe. From 1986 to 2000, 350 400 people were evacuated
and resettled from the most contaminated areas of Belarus, Russia and Ukraine. According to official post-Soviet data, about 57 % of the fallout landed in Belarus [1]. Chernobyl is widely considered to have been the worst nuclear accident in history and one of only two classified as a level 7 event on the International Nuclear Event Scale, the other being the Fukushima Daiichi nuclear meltdown in 2011 (see [2] Fukushima Nuclear Crisis, SiS 50).— From the beginning, the official nuclear safety experts were at pains to minimise the projected health impacts, as they are doing now for the Fukushima accident.  The UNSCEAR (United Nations Scientific Committee on the Effects of Atomic Radiation) estimated a “global collective dose” of radiation exposure from the accident “equivalent on average to 21 additional days of world exposure to natural background radiation”. Successive studies reported by the IAEA (International Atomic Energy Agency) continued to underestimate the level of exposure and to understate health impacts other than [3] “psychosocial effects, believed to be unrelated to radiation exposure” resulting from the lack of information immediately after the accident, “the stress and trauma of compulsory relocation to less contaminated areas, the breaking of social ties and the fear that radiation exposure could cause  health damage in the future.”——The number of deaths attributed to Chernobyl varies widely [1]. Thirty-one deaths are directly attributed to the accident, all among the reactor staff and emergency workers. An UNSCEAR report places the total confirmed deaths from radiation at 64 as of 2008. The Chernobyl Forum [4] founded in February 2003 at the IAEA Headquarters in Vienna with representatives from IAEA and UN agencies including UNSCEAR, WHO,  the World Bank, and Belarus, Russia and Ukraine, estimates that the eventual death toll could reach 4 000 among those exposed to the highest levels of radiation (200 000 emergency workers, 115 000 evacuees and 270 000 residents of the most contaminated areas); the figure includes some 50 emergency workers who died of acute radiation syndrome, 9 children who died of thyroid cancer and an estimated total of 3950 deaths from radiation-induced cancer and leukemia. The Union of Concerned Scientists based in Washington in the United States estimates another 50 000 excess cancer cases among people living in areas outside the most contaminated, and 25 000 excess deaths. A Greenpeace report puts the figure at 200 000 or more. The Russian publication, Chernobyl, by scientists Alexey V. Yablokov, Vassily B Nesterenko, and Alexey V. Nesterenko, translated and published by the New York Academy of Sciences in 2009, concludes that among the billions of people worldwide who were exposed to radioactive contamination from the disaster, nearly a million deaths had already occurred between 1986 and 2004. Most of the deaths were in Russia, Belarus and Ukraine [5] (see Truth about Chernobyl, SiS 47). The report drew on thousands of published papers and internet and printed publications. Those publications and papers, written by leading Eastern authorities, were downplayed or ignored by the IAEA and UNSCEAR. These agencies minimised their estimates by several ploys including [6]—– Underestimating the level of radiation by averaging exposure over a large regions, such as an entire country; so high exposure doses and health statistics of the most contaminated areas are lumped together with the less and least exposed

– Ignoring internal sources of radiation due to inhalation and ingestion of radioactive material from fallout
– Using an obsolete and erroneous model of linear energy transfer due to external sources of ionising radiation
– Not counting diseases and conditions other than cancers
– Overestimating the natural background radiation; today’s ‘background’ has been greatly increased by discharges from nuclear activities including tests of nuclear weapons, use of depleted uranium, and uranium mining
– Suppressing and withholding information from the public.
Nevertheless, the devastating health impacts did not escape the notice of the hundreds of doctors, scientists and other citizens who had to bear witness to the deformities, sicknesses and deaths of exposed babies, children and adults in their care. -Diversity of health impacts and their global extent over generations to come Alexei Yablokov, distinguished academician of the Russian Academy of Sciences in Moscow, spoke at the Scientific and Citizen Forum on adioprotection – From Chernobyl to Fukushima, 11-13 May 2012 in Geneva [7]. He is adamant that the consequences of the Chernobyl disaster can be clearly demonstrated by comparing the states of people’s health in areas receiving different amounts of additional radiation following the accident, instead of one based on average effective dose calculated by the ICRP and UNSCEAR which underestimates the true levels of irradiation. For example, there is a clear difference in mortality rates between highly contaminated provinces and less contaminated provinces of Russia (see Figure 1).  Yablokov is lead author of a massive report, now in its third enlarged 2011 edition [8], which has collated all the available evidence.

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Estriol- Benefits

Estriol, an estrogen that has virtually been ignored by the mainstream medical community, is one of the three principal estrogens produced by the body. Estriol was originally thought to have little significance due to its weak estrogenic activity when compared with estrone and estradiol. Nonetheless, research has found that its weakness may very well be its strength. –Studies suggest that when the lower-potency estrogen, estriol, is administered topically, it does not increase the risk of hormone-dependent cancers of the breast or endometrium (uterine lining).1-3 However, having weaker estrogenic effects does not mean that estriol has none of the benefits that come with more potent estrogens. Studies suggest that estriol reduces symptoms of menopause, such as hot flashes and vaginal dryness, but with a better safety profile compared with more potent estrogens.1,4,5 This makes estriol a better choice for bioidentical hormone-replacement treatment regimes. –That is not all this ‘weak’ hormone is good for! Research suggests that estriol has benefits for bone density, heart health, multiple sclerosis, and postmenopausal urinary tract health.6-12 In this article, we will review the attributes of this ‘weaker’ estrogen, and why this estrogen is currently in the news.

F     The body naturally makes three estrogen hormones—estradiol, estrone, and estriol. Since estriol possesses the weakest estrogenic effects of the three, it has been largely overlooked by the medical community.

  • Many studies show that estriol offers a wealth of potential health benefits—without the dangers that sometimes accompany higher-potency estrogens and synthetic or horse-derived hormones.
  • Studies suggest that estriol helps relieve menopausal symptoms while benefitting bone and urinary tract health. Estriol may also help improve cardiovascular risk factors and even shows promise in reducing the brain lesions of multiple sclerosis.
  • The most reliable way to measure estriol levels is through 24-hour urine collection.
  • Despite abundant evidence to the contrary, the FDA has recently claimed that estriol is not safe. You can act now to help preserve consumers’ access to bioidentical hormones such as estriol by visiting www.homecoalition.org.

Fear of cancer prevents many women from restoring youthful hormone levels. When applied through the topical (transdermal) route, estriol is not associated with increased cancer risk. Other methods women can use to prevent hormone-related cancers include consuming regular amounts of vitamin D,  and Bioflavonoids, Tumeric,Rosemary,Parsley,Dandelion, Hawthorn Berry, Black tea and Black Tea extracts, Celery Root, regulating meat and high-fat dairy intake.—

Estriol and Hormone Replacement Therapy

In addition, several studies suggest that bioidentical estrogen has less health risk when given with low doses of bioidentical progesterone.26,27— if you are on hormone-replacement therapy (HRT) and have never heard of estriol, you might be wondering why not? Before the 1970s, estriol was thought to have significance only during pregnancy we saw the beginning of hormone-replacement therapy with patented equine estrogens such as Premarin® and synthetic progestins as found in Provera®. By the 1990s, one-third of menopausal women were taking Premarin®. Research uncovered the increased incidence of breast cancer, increased risk of blood clotting, and increased cardiovascular risk associated with the use of these horse-derived and synthetic hormones (used in combination in the patented medication Prempro®).13 The medical community began to wonder if using hormones from pregnant horses was such a good idea. In an effort to find a safer alternative, many patients and practitioners began looking into ‘natural’ hormone-replacement treatment using bioidentical hormones, which are identical to those produced naturally within the body. Bioidentical-hormone replacement was pioneered in the 1980s as a treatment for menopause by Dr. Jonathan Wright in Washington state. ——-Interest in estriol increased as it was discovered that estriol was safer than horse-derived and synthetic hormones in relation to cardiovascular health and potentially cancer risk. Unfortunately, many doctors have not adopted its use, and many bioidentical hormone-replacement regimes use only estradiol, a more potent estrogen
with increased associated risks.-
– The benefits of estriol may, in part, be explained by the mixed pro-estrogenic and anti-estrogenic effects of this interesting estrogen hormone. Scientists Melamed et al. investigated the mixture of stimulating and non-stimulating effects posed by estriol upon estrogen receptors. When estriol is given together with estradiol, the estradiol-specific stimulation to cells is decreased. This little-appreciated scientific fact helps to explain how estriol can reduce pro-carcinogenic effects of more powerful estrogens like estradiol. However, when estriol is given alone over a long period of time, it can produce a more complete pro-estrogenic effect, explaining why symptom relief is achieved when menopausal women take estriol.2 Experimental studies suggest that both estriol and tamoxifen (a synthetic anti-estrogen) have protective effects against radiation-induced cancer of the breast[U10] .14— Most of the research cited in this article used oral estrogen as the route of administration. However, for enhanced safety, topical estriol would be a better choice. Several studies have shown that transdermal estrogen confers less health risk as a route of administration than oral estrogen.3,21-25 Clinical experience of many doctors over the past 20-30 years suggests that transdermal estrogen is also more effective for some women. This is largely thought to be due to the ‘first-pass effect’—meaning that orally ingested drugs are often first metabolized in the liver, before having any activity in the body. Orally ingested estrogen hormones are among these drugs that are first metabolized in the liver before exerting their effects in the body. Physicians experienced in hormone replacement often observe that women treated with oral estrogens show high levels of estrogen metabolites in 24-hour urine specimens, suggesting that most of the orally ingested hormones are being excreted.———- In a prospective study funded by the US Army and performed at the Public Health Institute, Berkeley, California, researchers compared estriol levels during pregnancy with breast cancer incidence 40 years later. Results revealed that of the 15,000 women entered in the study, those with the highest levels of estriol relative to other estrogens during pregnancy had the lowest cancer risk. In other words, as the relative level of estriol increased during pregnancy, risk of breast cancer decreased 40 years later. In fact, women with the highest level of estriol during pregnancy had 58% lower risk for breast cancer compared with women who had the lowest serum estriol levels. The authors also noted that Asian and Hispanic women had higher estriol levels compared with other racial groups. Interestingly, Asian and Hispanic women have the lowest breast cancer rates. The authors concluded, “If confirmed, these results could lead to breast cancer prevention or treatment regimens that seek to block estradiol estrogen action using estriol, similar to treatment based on the synthetic anti-estrogen tamoxifen.”15 —In another study, Takahashi et al. studied the safety of estriol treatment for Menopausal symptoms. Fifty-three women with either surgically induced or natural menopause were given 2 mg of oral estriol/day for 12 months. Endometrial and breast assessments done with endometrial biopsy and breast ultrasound, respectively, found normal results in all women. The authors concluded that over a 12-month period, “estriol appeared to be safe and effective in relieving symptoms of menopausal women.”1—In one investigation, 52 postmenopausal women were given 2 mg, 4 mg, 6 mg, or 8 mg/day of oral estriol for six months. In all patients, vasomotor symptoms of menopause (such as hot flashes) were decreased. The most improvement was experienced by women taking the highest dose of 8 mg. There were no signs of endometrial hyperplasia confirmed by endometrial biopsy over the six-month treatment period. Mammograms were obtained on six of the patients who had mammary hyperplasia at the study’s outset, and no further changes were seen.8- Although the oral route of administration of estriol appears relatively safe over the short-term, the transdermal route is preferred for long-term use. For example, Weiderpass et al. found an increased risk of endometrial atypical hyperplasia and endometrial cancer with oral use of estriol, but not with transdermal estriol over at least a five-year period. Compared with no use of estriol, those who took oral estriol for at least five years had a significantly greater risk, compared with individuals who did not take any estriol. Women using topical estriol for at least five years did not have any increased risk.

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New protein study could shake up sports nutrition market-Or Kill It!

A study out this week could add a new player to the protein market that’s long been dominated by whey.–At the Experimental Biology meeting in San Diego on Monday, Blake Rasmussen, PhD, of the University of Texas Medical Branch, presented findings that show a blend of protein sources—50 percent casein, 25 percent whey, 25 percent soy—was superior to whey alone for prolonging muscle building and recovery after exercise.[U11] “Whey protein has been given considerable notice as the gold standard ingredient after exercise to enhance muscle growth,” Rasmussen said. “The main problem with whey is it’s fast digesting—the anabolic response in muscle is only about an hour. We wanted to prolong the anabolic response with other protein sources. We found muscle protein synthesis is elevated for a longer amount of time with a protein blend versus whey protein.”[U12] The combination of protein blends was determined in Rasmussen’s previous preclinical work with rats.–Soy, whey and casein protein are all absorbed at different rates during digestion[U13] . Whey protein is referred to as a “fast” protein because it is rapidly absorbed, between 30 and 60 minutes, Rasmussen said. Soy is an intermediate protein, taking between 60 and 120 minutes to digest. And casein is a slow protein, requiring between three and five hours to digest.-“The combination gives you a quick increase in protein synthesis, and it gets sustained,” said Rasmussen. “It’s a prolonged delivery to muscle that the muscles use for recovery.”-The double-blind, randomized clinical trial followed 19 young adults before and after ingestion of about 19 grams of protein from the blend or about 17.5 grams of whey protein alone.

“Your muscles don’t recover in 30 minutes. It takes at least 24 to 48 hours for your muscles to recover after a resistance exercise,” said Greg Paul, global marketing director for sports nutrition and weight management at Solae, a soy supplier that sponsored the study.[U14]

Not just for athletes anymore?—It was only five years ago when research showed that protein should be an important part of sports nutrition products. Before then, the game was typically provided by the likes of Gatorade-style drinks: with fast carbs and electrolytes such as potassium and sodium.–Whey became the go-to protein source in beverages because it quickly fed muscles. Whey also appears better than soy for producing muscle synthesis because of the presence in whey of the amino acid leucine, which has been shown to uniquely act as a stimulatory signal for muscle protein synthesis. But for athletes and weekend warriors alike, using a blend of protein sources that absorb in the body over time means muscles are being fed until the next meal.

The addition of soy is also important because of soy’s particular properties including as an antioxidant and as an anti-inflammatory, which are both key attributes in muscle building beyond anabolic[U15] effects.

And this could lead to the next great demographic for protein products: the elderly.–“Protein blends are useful for sports nutrition,” said Rasmussen, “but also for those interested in aging and maintaining muscle mass as we age. This could potentially be a great intervention for aging.”[U16] -To date, products targeting elderly nutrition with protein-centric value propositions are few and far between. The trend of aging baby boomers, coupled with research demonstrating the value of protein blends in maintaining muscle mass, ought to be of interest to marketers and product developers. [U17]

 

TOP D


[U1]Again Not Genetic as they perpetrate—but a preconditioning to create the anomaly intended—an experiment of control and dependency and debilitation

[U2]Disruptor of the endocrine system—then what happens when you add several different disruptors of the endocrine system? Sterilization!!

[U3]Imagine the duress our lineage will have as a result of the –soy poisoning—chemtrail poisoning-vaccination poisoning –genetically modified food poisoning-environmental—they will be beyond xenophobic and will be told they have a genetic disposition to XYZ infections or conditions or potential invasion from a pathogen specific to there DNA structure—all lies—a direct result of intervention of a evil kind causing this to occur in 3 generations

[U4]It would appear they would possibly chose as a direct result of those who also would have hade these inherited genes –further compounding the problem and  causing  more issues

[U5]A Binary attack to hit and violate at the same time causing a breakdown

[U6]How interesting —we have a GMM= genetically  modified malaria—doe that not make you go hmmmmm wonder how it got genetically enhanced to resist what once cured it !!?—Is it not interesting that we have a new species of Malaria now—apparently the old one was not killing people off fast enough so now the pharma’ have decided to make this  a better pathogen

[U7]WARNING WARNING—Understand this—Genetic Mutation–

[U8]Interesting—if they had the  money they could be relieved but no money and they are held hostage by the IMF—what a crock

[U9] 90% kill ratio—-am I the only one seeing this is not a natural cause—weoponized malaria??

[U10]Utilizing black tea in a concentrated form  has actually the same effect as tamoxifen without the unwanted side effects

[U11]You never Mix a casein with a Soy they are highly allergenic and will cause all kinds of intestinal break down–The soy itself is an idigestible protein that cause intestinal and pancreatic cancers—avoid this

[U12]This is true—but the issue is how it is blended—the idea that you can mix a good egg or animal protein wih the whey would make this far superior to there method and without the allergens associated  with the Soy

[U13]This is true whey is in and out in about 2 hours casein in and out in about 4-6 hours-Soy can take a month due to the fact it does not digest and in fact causes intestinal disruptions and pancreatic shutdown—so they are right in the blending but wrong in how they blend

[U14]NOW YOU SEE WHO DID THE STUDY- A CONFLICT OF INTREREST SINCE THEY  TRIED TO PATENT SOY-AND THE ISRAELI GOV’T STOPPED THEM FROM ATTAINING THIS AND IN FACT GAVE SOME OF THE HARSHEST CRITIQUE ON SOY AND WHO COULD EAT IT—ESSENTIALLY NO ONE BASED ON THOSE STUDIES

[U15]This is at  best  a fabrication go to the soy links at http://augmentinforce.50webs.com

and read for your self the 4 studies that are there—even the FDA since 1904  knew Soy was toxic and not meant to consume

[U16]If this is followed this will exacerbate an already compromised colon and or pancreas—this advice Should not be followed at all—this was obviously done by someone trying to get ahead at the elderly expense

[U17]This is a form of  euphanasia—food poisoning the elderly

Show Of The Week June 11 2012

Understanding Why Alkalinity Can Be Unhealthy

 

Bt Toxin Kills Human Kidney Cells

 

Green Tea Compound for Radioprotection

 

2 people dead after swarms of venomous spiders invade Indian town

 

Polyphenolics from various extracts/fractions of red onion (Allium cepa) peel with potent antioxidant and antimutagenic activities

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Understanding Why Alkalinity Can Be Unhealthy

If you took high school or college chemistry, you may remember that pH is a measure of the acidity or alkalinity of a solution. The pH scale ranges from 0 to 14 with 7 for neutral solutions [white, middle of scale] with:

  • Lower numbers representing increased acidity [red zone, top of scale at right]
  • Higher numbers representing increased alkalinity and [blue zone, bottom of scale at right]

In an overly alkaline environment [due to aliens closeby in Earth’s 4th dimension — aliens whose energy is highly alkaline], there is a need for the following strategies:

  • Emphasize acid-forming foods
  • Use white vinegar in bath water and washing machine [2 –  to 9 cups] depending on the severity of the alkaline energy.
  • Use soaps that have a neutral pH [e.g. glycerin soap] or contain mild acid that help preserve the skin’s acid mantle [e.g. goat milk soap]. Note: Miracle II is a pH neutral soap that is available through mail-order distributors and some retail stores.

Acids and alkalines (also known as bases) neutralize each other forming salt and water. By emphasizing acids in your diet and on your skin, you will neutralize the extreme alkaline energy that is all around us [due to the presence of aliens]. I will explain more details about these concepts later in this article.

Why Does Alkalinity Cause Aging? The key to why alkalinity causes aging is in the definition of acids and bases that was developed by a scientist named Svante August Arrhenius (1859 – 1927): –An acid is a substance that increases the concentration of hydrogen ions (H+), which are carried as hydronium ions (H3O+) when dissolved in water, while bases are substance that increase the concentration of hydroxide ions (OH-). -Hydrogen ions are involved in growth (a daytime function). They’re also needed in the body to convert adenosine diphosphate (ADP) to adenosine triphosphate (ATP). ATP has been called the “energy currency” of the cell. —The illustration Below shows a high concentration of hydrogen ions (H+) that are needed for growth and the production of energy used in the human body. See: “Selecting Foods That Provide Hydrogen Ions.” A substance with a high concentration of hydrogen ions would be extremely acid showing little or no hydroxide ions.–In contrast, the illustration at right shows a high concentration of hydroxide ions that are present in alkaline substances. Hydroxide ions will slow growth and thwart metabolic processes in the body because they gobble hydrogen ions to form salt and water [setting up a condition for accelerated aging].–A substance with a high concentration of hydroxide ions would be extremely alkaline showing little or no hydrogen ions.

Food Lies When you read what foods provide hydrogen ions for growth and energy production, you’ll say: –Hey, wait a minute, is there an extermination agenda? It turns out, most of the health advice we’re given is false. Examples include:

  • [#1] Alkaline-forming foods are healthy [fruits and vegetables] and acid-forming foods are harmful.

The truth: Alkaline-forming foods [and energy] cause accelerated aging and promote the growth of parasites that cause disease [Note: If energy was more normal, fruits and vegetables would provide a balance].

  • [#2] Fat is bad and low- fat or no-fat is healthy [Note: It has become almost impossible to find whole milk products in food stores and children are being fed low-fat dairy in schools.

The truth: Half of every cell wall is made of saturated fat, the fat that we’re told not to eat. With decreased energy and not enough fat to make new cells, you can imagine the effect these two combined factors have on aging. Children need fat for brain development. In addition, fat is required to make hormones. Infertility rates are very high.

These ideas are so deeply embedded, they’re hard to dislodge. Aren’t all the sources of information providing the same information? That’s right — they are.

 

Selecting Foods That Provide Hydrogen Ions Selecting foods that provide hydrogen ions can be tricky because the key is to select foods leave minerals behind that the body can use to make acids.

Some foods, such as lemons, seem to contain an acid (citric acid), yet they do not leave the minerals behind to make acids.–To correct mistakes that you may be making — will require memorization. I recommend Herman Aihara’s book that is shown in the upper left corner of this page [some authors make mistakes concerning acid- forming and alkaline forming]. –Look closely at the following chart and notice what foods we’re told to eat and not eat:

Acid-forming Minerals Alkaline-forming Minerals
Chloride (makes hydrochloric acid) Magnesium (makes magnesium hydroxide)
Phosphorus (makes phosphoric acid) Calcium (makes calcium hydroxide)
Sulfur (makes sulfuric acid) Potassium (makes potassium hydroxide)
Acid-forming Foods Alkaline-forming Foods
Apple Cider Vinegar (strong) Citrus Fruit (strong)
Peanut Butter (strong) Green Vegetables (strong)
Chocolate (strong) Seaweed (strong)
Meat (medium) Green Tea (strong)
Bread (medium) Sea Salt (medium)
Eggs (medium) Coffee (medium)
Black tea (medium) Wine (medium)
Milk and Cheese (Weak) Ginger (Medium)

Dr. Carey Reams: Parasites Thrive in Alkalinity Dr. Carey Reams [1904-1985], a medical doctor who also had a career as a soil scientist, has a devoted following in modern agricultural circles that could be described as alternative. Just as there are alternative medical doctors who strive to heal the body without drugs, there are farmers who prefer to use natural methods instead of chemicals.–In the area of human health, Reams is most known for an ionization theory that consists of seven mathematical equations that provide a framework for balancing the body’s chemistry. The equations are referred to as Reams Biological Theory of Ionization [RBTI] and pH was an important aspect of Reams’ work.—During his lifetime, Reams  taught workshops, but was too busy avoiding the medical establishment to write his own books. Fortunately, an oral surgeon named Dr. Alexander Beddoe, has written several books about Reams’ work. –Dr. Beddoe’s RBTI text book lists the following health conditions that are due to alkalinity:

Parasites
Acne Arthritis Slow digestion Breakdown of discs in the back Body odor Deminerialization of bones Dental decay
Ear deterioration Fever Gall stonesHeart stress Hot flashes Lower GI gas Mental confusion Moody/depression Muscle soreness Increase in skin pigmentation

If alkalinity reflects an abundance of hydroxide ions, this may lead to a buildup of excess water in the body. When a hydroxide ion encounters a hydrogen ion [a base meeting an acid], there is a neutralization reaction that forms salt and water. If you add the popular health advice to eat alkaline-forming foods [fruits and vegetables], and the fact that hydrogen ions [from acid-forming foods] are required for metabolism-driving energy, I believe that acid-base chemistry is the reason that many businesses are selling very large products.

Hydrochloric Acid Kills Parasite Eggs Disease-causing parasites are the most serious result of the alkalinity problem. The solution is to make the body inhospitable to parasites using food chemistry and herbs.—Chloride, the mineral that’s needed to make hydrochloric acid, provides an example of why we need to be careful when selecting the foods that we eat. You’ll notice that chloride is a mineral that is needed to make hydrochloric acid [that kills parasite eggs in the stomach]. This mineral is left behind by acid-forming foods [that we’re told not to eat]. –Parasites that cause disease lay eggs that are very small. If the eggs are present in food and if we have enough hydrochloric acid, the eggs will not mature and cause health problems.—Alkaline-forming foods create an environment in the body that is very healthy for parasites.—To neutralize alkaline energy, I drink apple cider vinegar in ice water, drink mostly black tea and I eat a lot of peanut butter. Peanut butter is also rich in nutrition [26 minerals, 14 vitamins and monounsaturated fat that’s used for energy and converted to saturated fat as needed].

Killing Large Parasites Dr. Hulda Clark [1928-2009], who wrote The Cure and Prevention of All Diseases, discovered herbs that kill large parasites. —Because there are no lab tests for large parasites that cause serious diseases such as cancer and heart disease, most medical doctors are clueless about the connection between parasites and disease.–Hulda was a meticulous researcher and she made a great contribution. The following list of herbs form the foundation of a group of herbs that Hulda wrote about for fifteen years. See: Antiparasite Tea Recipe

Herb Action
Cloves Hydrochloric acid (HCL) in the stomach kills parasite eggs. If stomach acid is low, the eggs may survive. Cloves are known to kill:

• Parasite eggs • Pseudomonas aeruginosa • Staphylococcus • Streptococcus • Malaria • Tuberculosis • Cholera • Scabies • Candida • Shigella

Black Walnut [Green] Hull Tincture Kills adult parasites and developmental stages. This tincture contains organic iodine that is required by every cell in the body. The green hull around the nut of the black walnut has antiparasitic properties.
Wormwood (Artemesia absinthium) Wormwood works to anesthetize worms so they detach from body tissues. It is most effective against roundworms, hookworms, whipworms and pinworms.
Thyme Thyme kills hook-worms, roundworms, threadworms and skin parasites. Thyme also destroys Cryptococcus neoformans, Aspergillus, Saprolegnia, Salmonella typhimurium, Staphylococcus aureas, and Escherichia coli.

Antiparasite Tea Recipe Hulda’s basic herbs are Cloves, Black Walnut Hull Tincture, and Wormwood. I make tea using the following recipe [frequently].

2-3 Cups of Water 2-3 Tea Bags Black Tea [e.g. Earl Grey] 48 Drops of Black Walnut Hull Tincture 12 Drops of Wormwood Tincture 1/4 teaspoon of Ground Cloves (approximately) 2-3 Sprigs of Fresh Thyme

  1. Use Distilled or Reverse Osmosis If you have a weak immune system, you may want to purchase Hulda’s book and learn how to sterilize food — with a freezer or sound waves. Chest freezers that can be set to -20 degrees kill parasites and microorganisms in 24 hours. — I use sound waves generated by a jewelry cleaner that Hulda renamed a “sonicator.” The Haier Jewelry Cleaner has a 25 oz. water tank (HDPE bottles of herbs are submerged to apply sound waves). Jewelry cleaners kill bugs in 20 minutes [$29.99 at Amazon.com].

A jewelry cleaner tank can accomodate two Nalgene, four-ounce, wide-mouth HDPE bottles containing loose herbs [available at camping stores such as REA or Eastern Mountain Sports].

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Bt Toxin Kills Human Kidney Cells

Cry1Ab biopesticide kills human cells at low doses as does Roundup herbicide Dr Eva Sirinathsinghji
A new study shows that low doses of Bt biopesticide CryA1b as well as the glyphosate herbicide, Roundup, kill human kidney cells. The Bt biopesticide conferring insect resistance and the glyphosate tolerance trait tied to the use of glyphosate herbicides account for almost all the GM crops grown worldwide. Bt crops already constitute 39 % of globally cultivated genetically modified (GM) crops, yet this is the first study that provides evidence on the toxicity of Bt protein in human cells.  This work comes at a time when the French environment and agricultural ministers are seeking an EU-wide ban of Monsanto’s MON810 Bt corn variety that is already outlawed in Hungary, Austria, Germany, Greece, and Luxembourg[U1] . The EU commission approved this crop in 2009, concluding that it “is as safe as its conventional counterpart with respect to potential effects on human and animal health”. In response to their publication the research team raised questions about the safety assessment procedure stating that their findings were a “surprising outcome and this risk was somehow overlooked” in past assessments of such crops. [1]. — The research team led by Gilles-Eric Séralini at the University of Caen, France, is already well-known for their investigations on the endocrine disrupting effects of glyphosate herbicides (see [2] Glyphosate Kills Rat Testis Cells[U2] , SiS 54).The researchers tested the effects of Cry1Ab and Cry1Ac proteins as well as their combined effects with the herbicide Roundup on the human kidney cell line HEK293 [3]. Humans are exposed to hundreds of chemicals in a day, and their combined effects need to be understood. This is particularly important when considering the new generation of ‘stacked’ genetically modified (GM) crops now on the market, which carry multiple resistance genes for Bt toxins and glyphosate tolerance together. [U3] ———Experiments were performed to assess both cell death and cell membrane integrity, as the pesticidal activity of Bt toxins results from creating pores in the membrane of cells in the insect gut. Cell death was measured using three parameters: 1) mitochondrial succinate dehydrogenase enzyme activity as a general cell death marker, 2) activity of the membrane-bound enzyme adenylate kinase  (AK) to assess membrane integrity as a marker of necrotic cell death and 3) caspase 3/7 activity, as a marker of apoptosis (programmed cell death). They found that Cry1Ab caused cell death at concentrations of 100 parts per million (ppm), according to mitochondrial succinate dehydrogenase activity. The membrane-bound enzyme adenylate kinase (AK) goes up in activity when the membrane disintegrates and releases the enzyme into the culture medium. Cry1Ab at 100 ppm induced a 2-fold increase in AK activity. No effects were seen with Cry1Ac.

 

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Antidiabetic potentials of Momordica charantia: multiple mechanisms behind the effects.

J Med Food. 2012 Feb;15(2):101-7–Authors: Chaturvedi P

Abstract Momordica charantia fruits are used as a vegetable in many countries. From time immemorial, it has also been used for management of diabetes in the Ayurvedic and Chinese systems of medicine. Information regarding the standardization of this vegetable for its usage as an antidiabetic drug is scanty. There are many reports on its effects on glucose and lipid levels in diabetic animals and some in clinical trials. Reports regarding its mechanism of action are limited. So in the present review all the information is considered to produce some concrete findings on the mechanism behind its hypoglycemic and hypolipidemic effects. Studies have shown that M. charantia repairs damaged β-cells, increases insulin levels, and also enhance the sensitivity of insulin. It inhibits the absorption of glucose by inhibiting glucosidase and also suppresses the activity of disaccharidases in the intestine. It stimulates the synthesis and release of thyroid hormones and adiponectin and enhances the activity of AMP-activated protein kinase (AMPK). Effects of M. charantia like transport of glucose in the cells, transport of fatty acids in the mitochondria, modulation of insulin secretion, and elevation of levels of uncoupling proteins in adipose and skeletal muscles are similar to those of AMPK and thyroxine. Therefore it is proposed that effects of M. charantia on carbohydrate and fat metabolism are through thyroxine and AMPK.—PMID: 22191631 [PubMed – indexed for MEDLINE][U4]

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Green Tea Compound for Radioprotection
Green tea polyphenol antioxidant protects against bystander effects of low dose ionizing radiation that damage cells and cause numerous diseases including cancer. Dr. Mae-Wan Ho–The recent discovery of bystander effects from low levels of ionizing radiation has thrown risk assessment and radioprotection into disarray [1] (Bystander Effects Multiply Dose and Harm from Ionizing Radiation, SiS 55). However, it has also led to the discovery of potential mitigating measures against exposure to radioactivity, especially from nuclear accidents like Chernobyl (and Fukushima), the devastation health impacts of which are still surfacing 25 years later [2] (Chernobyl Deaths Top a Million Based on Real Evidence. SiS 55). ——Ionizing radiation has been known to produce free radicals and reactive oxygen species (ROS), predominantly by ionizing water, the most abundant molecules in tissues and cells (see [1] for an explanation of ROS). ROS are responsible for oxidative damage to DNA, proteins, and lipids, initiating cell death, genomic instability and other consequences of radiation, both in cells that have been directly targeted, and in bystander cells that have not been irradiated [1]. There is evidence that various antioxidants can protect cells against bystander radiation damages, and new findings published online in Mutation Research appear particularly promising. —-shu Tiku and Benila Richi at Jawaharlal Nehru University, New Delhi and Roasaheb Kale at Central University of Gujarat in India may have found the ideal antioxidant for radioprotectopm [3].
Non-toxic compound needed for radioprotection—–One main problem in radioprotection is to find compounds that are non-toxic or minimally so, and natural compounds fit the bill in being both non-toxic and easily available.  Green tea is a rich source of polyphenols with strong antioxidant activities. Green tea extracts and its polyphenols have been shown to possess many health benfits attributed to their antioxidant and anti-inflammatory properties (see [4, 5] Green Tea, The Elixir of Life? and Green Tea Against Cancers, SiS 33). Most of the health benefits of green tea have been credited to the major polyphenol EGCG (epigallocatechin-3-gallate) which constitutes 55 – 70 % of total polyphenols in green tea extract. Its antioxidant potential is believed to be far greater than vitamin E and vitamin C, the two main antioxidants among vitamins [6]. The team exposed both pBR322 plasmid DNA as well as spleen cells from mice to g-radiation at different concentrations of EGCG. Preliminary experiments found that EGCG concentrations above 125 mM were toxic to the cells, so the highest concentration used was restricted to 100 mM. The effects of quercetin – another polyphenol found in fruits, vegetables, leaves and grains – and vitamin C were also investigated. The plasmid DNA and cells were incubated for 2 hours with EGCG at different concentrations or quercetin and vitamin C, both at 100 mM, before being irradiated. Afterwards, the plasmid and cells were assessed for DNA damage, and the cells for viability, lipid peroxidation, membrane fluidity, and for activities of enzymes and cofactors involved in detoxification and scavenging of ROS.  Tea compound protects against DNA breaks and cell death–The intact plasmid is supercoiled in a compact form, while the cut plasmid is circular, and the two forms can be clearly distinguished and quantified by electrophoresis. The control (unexposed) sample is about 85% supercoiled. EGCG was found to protect plasmid DNA against breaks at high (50 Gy) or low (3 Gy) dose radiation: >82.5 % protection even at the lowest concentration of EGCG tested (10 mM) and complete 100 % protection at 50 mM. EGCG was better at protection against DNA breaks than quercetin or vitamin C at the same concentration of 100 mM. The viability of cells was determined with a vital dye that depends on active mitochondria.  At 3 to 7 Gy of g-irradiation, cell viability was significantly decreased, and at the highest dose, to 53 % of unexposed controls; but pre-incubation with EGCG protected the cells and restored viability in a concentration dependent manner, at 100 mM, viability was restored to >96 % of control. Single cell comet assay was used to determine the extent of DNA degradation in the cells. In this assay, cells are trapped in agar gel on a microscope slide, lysed to expose their DNA for electrophoresis, and stained with a fluorescent dye. Cells with intact DNA will appear as a small compact bright spot, while cells with degraded DNA will appear as a diffuse spot with a tail, like a comet, hence the name of the assay.  The bigger the tail, the greater is the extent of degradation, which can be quantified with computer software under a fluorescent microscope. Exposing the cells to 3 Gy led to substantial DNA degradation, which was reduced in a concentration dependent manner by EGCG. Quercetin and vitamin C -also protected the cells against DNA damage, though not as effectively as EGCG.

 

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2 people dead after swarms of venomous spiders invade Indian town

( Playing with genetics- )–Published June 03, 2012

SADIYA, India –  A town in India has suddenly been overrun by swarms of venomous spiders, leaving two people dead after being bitten.–It may sound like a B-grade horror movie, but residents of the town of Sadiya, in Assam state, say that on the evening of May 8 as they were celebrating a Hindu festival swarms of spiders suddenly appeared and attacked them, The Times of India reported.—Over the next few days two people — a man, Purnakanta Buragohain, and an unnamed school boy — died after being bitten by the spiders. Scores more turned up at the town’s hospital with spider bites.–Local resident Jintu Gogoi spent a day in the hospital complaining of excruciating pain and nausea after being bitten. He said weeks later his finger was still blackened and swollen.—District authorities are also panicking — and they are considering spraying the town with the insecticide DDT.—Locals say the most terrifying aspect is that spiders appear in swarms and their behavior is highly aggressive.[U5] –“It leaps at anything that comes close. Some of the victims claimed the spider latched on to them after biting. If that is so, it needs to be dealt with carefully. The chelicerae and fangs of this critter are quite powerful,” head of the department of life sciences at Dibrugarh University Dr. L.R. Saikia said.–Teams of Indian arachnid experts have flocked to the town, hoping to identify the species, but so far they have drawn a blank.[U6] They say it could be a tarantula, a black wishbone or even a funnel-web spider — or it could be a whole new species.—One thing they agree on is that it is not native to the area as there is no record of venomous spiders in Assam. The black wishbone and funnel-web are native to Australia. Researchers are also still running tests to find out the toxicity of the spiders’ venom.–Dr. Anil Phatowali, superintendent of the town’s hospital, said they had not administered antivenin as they could not be certain the spider was venomous at all.—He also pointed out other factors may have contributed to the two reported fatalities.–“All the bite patients first went to witch doctors, who cut open their wounds with razors, drained out blood and burnt it. That could have also made them sick,” Phatowali said.

Read more: http://www.foxnews.com/world/2012/06/03/2-people-dead-after-swarms-venomous-spiders-invade-indian-town/print#ixzz1wmE3t1TL

 

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Polyphenolics from various extracts/fractions of red onion (Allium cepa) peel with potent antioxidant and antimutagenic activities.

Singh BN, Singh BR, Singh RL, Prakash D, Singh DP, Sarma BK, Upadhyay G, Singh HB.

Source

Nutraceutical Chemistry, National Botanical Research Institute, Lucknow 226 001, India.

Abstract

In order to determine antioxidant activity, the five extracts/fractions of red onion peel were studied for their total content of phenolics (TPC), flavonoids (TFC), antioxidant activity (AOA), free radical scavenging activity (FRSA), assayed by DPPH radical in the terms of anti-radical power (ARP) and reducing power (RP), expressed as ascorbic acid equivalents (ASE)/ml. High TPC (384.7 +/- 5.0 mg GAE/g), TFC (165.2+/- 3.2 mg QE/g), AOA (97.4 +/- 7.6%), ARP (75.3 +/-4.5) and RP (1.6 +/-0.3 ASE/ml) were found for the ethyl acetate (EA) fraction. EA fraction had markedly higher antioxidant capacity than butylated hydroxytoluene (BHT) in preventive or scavenging capacities against FeCl3-induced lipid peroxidation, protein fragmentation, hydroxyl (site-specific and non-site-specific), superoxide anion and nitric oxide radicals. EA fraction also showed dose dependent antimutagenic activity by following the inhibition of tobacco-induced mutagenicity in Salmonella typhimurium strains (TA102) and hydroxyl radical-induced nicking in plasmid pUC18 DNA. HPLC and MS/MS analysis showed the presence of ferulic, gallic, protocatechuic acids, quercetin and kaempferol. The large amount of polyphenols contained in EA fraction may cause its strong antioxidant and antimutagenic properties. This information shows that EA fraction of red onion peel can be used as natural antioxidant in nutraceutical preparations.

 

 

TOP C


[U1]Observe these countries who have rejected this GMO—watch them all go financially broke—or economically devastated—germany is broke from bailing out half of Europe and Greece is totally in economic ruin—keep your eye on the other countries and watch them become devastated

[U2]Apparently this is what is going on with the  Human Race as well

[U3]And you wonder why you have headaches—endocrinal disruptions-heart issues-skin issues-brain issues—thyroid and liver and lung issues-glandular issues-why you seem to be aging far more rapidly then you should—why you are having difficulty in things you should not be having especially at the ages of the mid 20’s to 40 and upward ( downward if you are afflicted )

[U4]SO this maybe  good to take with tyrosine and iodne as welll

[U5]Genetically Modified Spider?—is it possible after all these years of planting a genetic disrupting compounds into the earth that we have finally made a genetically aggressive species—Was it done in a lab and now this particular area was or is being targeted!!!

[U6]A BLANK!!!!!so is it an alien specied fallen off a space ship—is it a lab made spider—is it been genetically changed due to either experimentation or careless dumping of genetic  materials—and no hypothesis?? A blank—something smells bad and it ain’t socks off a sweaty feet!!