GM soy making its way into the UK food chain through animal feed


NaturalNews) Labeling laws throughout the European Union (EU) require that all food containing genetically-modified (GM) ingredients be properly labeled. But a new report from The Telegraph explains that many big-name food brands in the UK that sell meat, dairy, and other animal products are sourcing from animals fed GMOs, but selling the final product without a proper GMO label.

The problem of GMOs sneaking in the back door through animal feed is not limited to the EU, as many other countries that require mandatory GMO labeling are experiencing similar scenarios. A recent NaturalNews report highlighted GMO contamination problems with Fonterra, a New Zealand-based cooperative that is the world’s largest exporter of dairy products, as well (…).

Some experts insist that animals fed GM feed do not end up producing GM-contaminated meat and dairy. But others say that GM traits are, indeed, passed on through animal feed into the animal itself, contaminating milk and meat with GM materials. They also say that the growing prevalence of GM feed — particularly GM soy — is destroying rainforests, introducing extreme amounts of new pesticides into the environment, and damaging animal and human health (…).

According to Barbara Gallani, Director of Food Safety and Science at the UK Food and Drink Federation, most major UK brands of conventional meat and dairy now come from animals fed GM soy. And more than three million tons of soy, most of which is GM, is imported into the UK every year.

Numerous groups continue to petition officials to correct this glaring loophole that allows GM-fed animals to have their meat and dairy sold as non-GM. Former Labour environment minister Michael Meacher told The Telegraph that safety studies on meat and dairy produced from GM-fed animals are inadequate, and that people are “entitled to know” what they are eating.

In the US, GMO labeling laws are nonexistent. The vast majority of conventional soy, corn, and cotton products are GM, and most consumers are unaware of it. Advocacy groups have been pushing for labeling laws in the US for years, but the US Food and Drug Administration (FDA) and the US Department of Agriculture (USDA) have sided with the Codex Alimentarius Committee position that falsely says there is no difference between GM and non-GM crops (…).

To see how GM soy is destroying the health, lives, and livelihoods of those who work or live near GM soy plantations, check out this sobering report recently aired on German television:…

Learn more:

More scientific evidence that antioxidants can fight cancer

(NaturalNews) For the first time, scientists have discovered a genetic pathway suggesting that antioxidants may help in the treatment of cancer, according to a study conducted by researchers from Thomas Jefferson University and published in the journal Cancer Biology & Therapy.

Scientists have known for a long time that diets high in antioxidant-containing foods are associated with a lower risk of cancer. This effect is widely attributed to the fact that antioxidants remove DNA-damaging free radicals from the body.

“Reactive oxygen species [free radicals] are formed in vivo during normal aerobic metabolism and can cause damage to DNA, proteins, and lipids, despite the natural antioxidant defense system of all organisms,” writes Erich Grotewold in the book The Science of Flavonoids.

“Diets high in flavonoids, fruits, and vegetables are protective against a variety of diseases, particularly cardiovascular disease and some types of cancer,” he writes.

Yet now researchers have shown that antioxidants may not just prevent, but also treat cancer. The Thomas Jefferson University researchers had previously discovered a protein called Caveolin-1 (Cav-1) that, when present in the human genome, suppresses tumor growth. Indeed, the presence or absence of the Cav-1 protein is the single best predictor of breast cancer outcome. Women with triple-negative breast cancer – the most deadly kind – are more than 75 percent likely to be alive 12 years after diagnosis if they have the protein, and less than 10 percent likely to be alive five years after diagnosis if they lack it.

The researchers further verified that Cav-1 plays an antioxidant role in the body. In the current study, the researchers confirmed that when Cav-1 is removed, oxidative stress in breast cancer tumors increases, leading to a 300 percent increase in tumor mass and volume.

“Antioxidants have been associated with cancer reducing effects – beta carotene, for example – but the mechanisms, the genetic evidence, has been lacking,” lead researcher Michael P. Lisanti said. “This study provides the necessary genetic evidence that reducing oxidative stress in the body will decrease tumor growth.”

The findings may actually cast doubt on the effectiveness of many chemotherapy drugs, which are actually known to increase oxidative stress. They also suggest that many antioxidant drugs used to treat other conditions, such as diabetes and malaria, may be effective in treating cancer.

To learn more about how to fight disease with a healthy diet, read th

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Scientists Link DNA ‘End-Caps’ Length to Diabetes Risk; New Role for Short Telomeres

Scientists Link DNA ‘End-Caps’ Length to Diabetes Risk; New Role for Short Telomeres

ScienceDaily (Mar. 24, 2011) — New evidence has emerged from studies in mice that short telomeres or “caps” at the ends of chromosomes may predispose people to age-related diabetes, according to Johns Hopkins scientists.

Telomeres are repetitive sequences of DNA that protect the ends of chromosomes, and they normally shorten with age, much like the caps that protect the end of shoelaces. As telomeres shorten, cells lose the ability to divide normally and eventually die. Telomere shortening has been linked to cancer, lung disease, and other age-related illnesses. Diabetes, also a disease of aging, affects as many as one in four adults over the age of 60.

The Johns Hopkins research, described in the March 10 issue of PLoS ONE, arose from scientist Mary Armanios’ observation that diabetes seems to occur more often in patients with dyskeratosis congenita, a rare, inherited disease caused by short telomeres. Patients with dyskeratosis congenita often have premature hair graying and are prone to develop early organ failure.

“Dyskeratosis congenita is a disease that essentially makes people age prematurely. We knew that the incidence of diabetes increases with age, so we thought there may be a link between telomeres and diabetes,” says Armanios, assistant professor of oncology at the Johns Hopkins Kimmel Cancer Center.

Armanios studied mice with short telomeres and their insulin-producing beta cells. Human diabetics lack sufficient insulin production and have cells resistant to its efficient use, causing disruption to the regulation of sugar levels in the blood.

Armanios found that despite the presence of plentiful, healthy-looking beta cells in the mice, they had higher blood sugar levels and secreted half as much insulin as the controls. “This mimics early stages of diabetes in humans where cells have trouble secreting insulin in response to sugar stimulus,” says Armanios.

“Many of the steps of insulin secretion in these mice, from mitochondrial energy production to calcium signaling, functioned at half their normal levels,” says Armanios.

In beta cells from mice with short telomeres, they found disregulation of p16, a gene linked to aging and diabetes. No such mistakes were found in the controls.

In addition, many of the gene pathways essential for insulin secretion in beta cells, including pathways that control calcium signaling, were altered in beta cells from mice with short telomeres.

Armanios says that some studies have suggested that diabetic patients may have short telomeres, but it was not clear whether this contributes to diabetes risk or is a consequence of the disease.

“Age is the most important risk factor for diabetes, and we also know that family heredity plays a very important role. Telomere length is an inherited factor and may make people more prone to develop diabetes,” says Armanios.

Based on this work, Armanios says that telomere length could serve as a biomarker for development of diabetes. Armanios and her colleagues are planning to conduct research to examine whether telomere length can predict the risk of diabetes prospectively.

The research was funded by the National Institutes of Health, a Ruth L. Kirschstein Award, the Maryland Stem Cell Research Fund, Sidney Kimmel Foundation, Doris Duke Charitable Foundation, the Swedish Research Council and the Family Erling-Persson Foundation.

Participants in the research included Nini Guo, Erin M. Parry, Frant Kembou, Naudia Lauder, Mehboob A. Hussain from Johns Hopkins; and Luo-Sheng Li and Per-Olof Berggren from the Karolinska Institutet in Sweden.

Walnuts Are Top Nut for Heart-Healthy Antioxidants

Walnuts Are Top Nut for Heart-Healthy Antioxidants

ScienceDaily (Mar. 28, 2011) — A new scientific study positions walnuts in the number one slot among a family of foods that lay claim to being among Mother Nature’s most nearly perfect packaged foods: Tree and ground nuts. In a report given in Anaheim, California at the 241st National Meeting & Exposition of the American Chemical Society on March 27, scientists presented an analysis showing that walnuts have a combination of more healthful antioxidants and higher quality antioxidants than any other nut.

“Walnuts rank above peanuts, almonds, pecans, pistachios and other nuts,” said Joe Vinson, Ph.D., who did the analysis. “A handful of walnuts contains almost twice as much antioxidants as an equivalent amount of any other commonly consumed nut. But unfortunately, people don’t eat a lot of them. This study suggests that consumers should eat more walnuts as part of a healthy diet.”

Vinson noted that nuts in general have an unusual combination of nutritional benefits — in addition those antioxidants — wrapped into a convenient and inexpensive package. Nuts, for instance, contain plenty of high-quality protein that can substitute for meat; vitamins and minerals; dietary fiber; and are dairy- and gluten-free. Years of research by scientists around the world link regular consumption of small amounts of nuts or peanut butter with decreased risk of heart disease, certain kinds of cancer, gallstones, Type 2 diabetes, and other health problems.

Despite all the previous research, scientists until now had not compared both the amount and quality of antioxidants found in different nuts, Vinson said. He filled that knowledge gap by analyzing antioxidants in nine different types of nuts: walnuts, almonds, peanuts, pistachios, hazelnuts, Brazil nuts, cashews, macadamias, and pecans. Walnuts had the highest levels of antioxidants.

Vinson also found that the quality, or potency, of antioxidants present in walnuts was highest among the nuts. Antioxidants in walnuts were 2-15 times as potent as vitamin E, renowned for its powerful antioxidant effects that protect the body against damaging natural chemicals involved in causing disease.

“There’s another advantage in choosing walnuts as a source of antioxidants,” said Vinson, who is with the University of Scranton in Pennsylvania. “The heat from roasting nuts generally reduces the quality of the antioxidants. People usually eat walnuts raw or unroasted, and get the full effectiveness of those antioxidants.”

If nuts are so healthful and nutritious, why don’t people eat more? Vinson’s research shows, for instance, that nuts account for barely 8 percent of the daily antioxidants in the average person’s diet. Many people, he said, may not be aware that nuts are such a healthful food. Others may be concerned about gaining weight from a food so high in fat and calories. But he points out that nuts contain healthful polyunsaturated and monosaturated fats rather than artery-clogging saturated fat. As for the calories, eating nuts does not appear to cause weight gain and even makes people feel full and less likely to overeat. In a 2009 U. S. study, nut consumption was associated with a significantly lower risk of weight gain and obesity. Still, consumers should keep the portion size small. Vinson said it takes only about 7 walnuts a day, for instance, to get the potential health benefits uncovered

People at Risk of Alzheimer’s May Now Be Able to Delay the Onset of Their First Symptoms

People at Risk of Alzheimer’s May Now Be Able to Delay the Onset of Their First Symptoms

ScienceDaily (Mar. 28, 2011) — The human brain loses 5 to 10% of its weight between the ages of 20 and 90 years old. While some cells are lost, the brain is equipped with two compensatory mechanisms: plasticity and redundancy. Based on the results of her most recent clinical study published March 23 in the online version of Brain: A Journal of Neurology, Dr. Sylvie Belleville, PhD in neuropsychology, the principal author of this study and Director of Research at the Institut universitaire de gériatrie de Montréal (IUGM), which is affiliated with the Université de Montréal, has found that for elderly subjects at risk of developing Alzheimer’s disease, hope may lie in brain plasticity.

“Brain plasticity refers to the brain’s remarkable ability to change and reorganize itself. It was long thought that brain plasticity declined with age, however, our study demonstrates that this is not the case, even in the early stages of Alzheimer’s disease,” declares Sylvie Belleville.

These findings open countless new avenues of research including the possibility of improving the plasticity of affected areas of the brain, and slowing the decline in plasticity through pharmacological means or lifestyle changes, thereby allowing subjects with Alzheimer’s disease to enjoy several more symptom-free years.

The hypothesis behind this research was that certain cells traditionally involved in other brain processes could, through a simple memory training program, temporarily take over since they themselves are not yet affected. According to Dr. Belleville: “Our research has validated our hypothesis. Not only were we able to use functional imaging to observe this diversification, but we also noted a 33% increase in the number of correct answers given during a post-training memory task by subjects with mild cognitive impairment (MCI) who, incidentally, are ten times more likely to develop Alzheimer’s disease.”

The training program that was used was designed to help elderly subjects with MCI develop strategies, such as the use of mnemonics, for example, and promote encoding and retrieval, such as word lists, for example, using alternative areas of the brain. “The hypothesis had already been raised, but our team was the first to provide scientific support, using a functional neuroimaging protocol,” added Sylvie Belleville.

Researchers worked with thirty elderly subjects: 15 healthy adults and 15 with MCI. Magnetic resonance imaging was used to analyse brain activity in the two groups 6 weeks prior to memory training, one week prior to training and one week after training. Before the memory training, magnetic resonance imaging in both the healthy elderly subjects and those with MCI showed activation in areas of the brain traditionally associated with memory. As expected, decreased activation was observed in subjects with MCI. After training, brain areas in elderly subjects with MCI showed increased activation in areas typically associated with memory, but also in new areas of the brain usually associated with language processing, spatial and object memory and skill learning.

According to Dr. Belleville: “Analysis of brain activity during encoding as measured before and after the training program, indicates that increased post-training activation in the right inferior parietal gyrus is associated with post-intervention improvement. The healthy area of the brain has taken over for the area that is compromised.”

Structure of DNA Repair Complex Reveals Workings of Powerful Cell Motor

Structure of DNA Repair Complex Reveals Workings of Powerful Cell Motor

ScienceDaily (Mar. 28, 2011) — Over the last years, two teams of researchers at The Scripps Research Institute have steadily built a model of how a powerful DNA repair complex works. Now, their latest discovery provides revolutionary insights into the way the molecular motor inside the complex functions — findings they say may have implications for treatment of disorders ranging from cancer to cystic fibrosis.

In a paper published in an Advance Online Edition of Nature Structural and Molecular Biology March 27, 2011, the scientists say that the complex’s motor molecule, known as Rad50, is a surprisingly flexible protein that can change shape and even rotate depending on the task at hand.

The finding solves the long-standing mystery of how a single protein complex known as MRN (Mre11-Rad50-Nbs1) can repair DNA in a number of different, and tricky, ways that seem impossible for “standard issue” proteins to do, say team leaders Scripps Research Professor John Tainer, Ph.D., and Scripps Research Professor Paul Russell, Ph.D., who also collaborated with members of the Lawrence Berkeley National Laboratory on the study.

They say the finding also provides a critical insight into the ABC-ATPase superfamily of molecular motors, of which Rad50 is a member.

“Rad50 and its brethren proteins in this superfamily are biology’s general motors,” said Tainer, “and if we know how they work, we might be able to control biological outcomes when we need to.”

For example, knowing that Rad50 changes its contour to perform a function suggests it might be possible to therapeutically target unique elements in that specific conformation. “There could be a new generation of drugs that are designed not against an active site, like most drugs now (an approach that can cause side effects, but against the shape the protein needs to be in to work,” Tainer said.

Russell added, “Proteins are often viewed as static, but we are showing the moving parts in this complex. They are dynamic. They move about and change shape when engaging with other molecules.”



The MRN complex is known as a first-responder molecule that rushes in to repair serious double-strand breaks in the DNA helix — an event that normally occurs about 10 times a day per cell due to ultraviolet light and radiation damage, etc. If these breaks are not fixed, dangerous chromosomal rearrangements can occur that lead to cancer. Paradoxically, the complex also mends DNA breaks promoted by chemotherapy, protecting cells against cancer treatment.

When MRN senses a break, it activates an alarm telling the cell to shut down division until repairs are made. Then, it binds to ATP (an energy source) and repairs DNA in three different ways, depending on whether two ends of strands need to be joined together or if DNA sequences need to be replicated. “The same complex has to decide the extent of damage and be able to do multiple things,” Tainer said. “The mystery was how it can do it all.”

To find out, Tainer, head of a structural biology group, and Russell, who leads a yeast genetics laboratory, began collaborating five years ago. With the additional help of team members at Lawrence Berkeley National Laboratory and its Advanced Light Source beamline, called SIBYLS, the collaboration has produced a series of high-resolution images of the crystal structure of parts of all three proteins (rad50, Mre11, and Nbs1), taken from fission yeast and archaea. The scientists also used the lab’s X-ray scattering tool to determine the proteins’ overall architecture in solution, which approximates how a protein appears in a natural state.

The scientists say that the parts of the complex, when imagined together as a whole unit, resemble an octopus: the head consists of the repair machinery (the Rad50 motor and the Mre11 protein, which is an enzyme that can break bonds between nucleic acids) and the octopus arms are made up of Nbs1 which can grab the molecules needed to help the machinery mend the strands.

In this study, Tainer and Russell were able to produce crystal and X-ray scattering images of parts of where Rad50 and Mre11 touched each other, and what happened when ATP bound to this complex and what it looked like when it didn’t.

In these four new structures, they showed that ATP binding allows Rad50 to drastically change its shape. When not bound to ATP, Rad50 is flexible and floppy, but bound to ATP, Rad50 snaps into a ring that presumably closes around DNA in order to repair it.

“We saw a lot of big movement on a molecular scale,” said Tainer. “Rad50 is like a rope that can pull. It appears to be a dynamic system of communicating with other molecules, and so we can now see how flexibly linked proteins can alter their physical states to control outcomes in biology.”

“We thought ATP allowed Rad50 to change shape, but now we have proof of it and how it works,” Russell said. “This is a key part of the MRN puzzle.”



Rad50 and ATP provide the motor and gas for a number of biological machines that operate across species. These machines are linked to a number of disorders, such as cystic fibrosis, which is caused by a defect in the cystic fibrosis transmembrane conductance regulator (CFTR) gene, which is a member of the ABC ATPase superfamily.

“Our study suggests that ABC ATPase proteins are used so often in biology because they can flexibly hook up to so many different things and produce a specific biological outcome,” Tainer said.

Given this new prototypic understanding of these motors, Tainer and Russell envision a future in which therapies might be designed that target Rad50 when it changes into a shape that promotes a disease. For example, chemotherapy could be coupled with an agent that prevents the MRN complex from repairing DNA damage, promoting death of cancer cells.

“There are some potentially very cool applications to these findings that we are only beginning to think about,” Russell said.

The study was funded by the National Cancer Institute, the National Institutes of Health, and the Department of Energy


An Engine for Many Vehicles


First Responder

AntiRadiation  and then click on the antiradiation link

and go to and type in herbspusbeadworks and llo at the SSKI and the Lugols Vid to see how to make your own—

with the nightmare of the Russian setting fire last year near the Ukraine border where there was Nuclear wast stockpiled it burned for almos 2 1/2 weeks this is what we are feeling today the japa nuclear is a sensationalized media script to get us off our truth in what we see—here is more info on anti radiation—


Subcategories– Baking soda (sodium bicarbonate) bathsCysteine hydrochloride —  Radioprotective Herbs —  Alkalinizers to Remove Uranium and other Radioisotopes  — Herbs and Supplements For Anti-Radiation –  Evaluation of silymarin as a promising radioprotectorFormulations and RecipesPlant Antioxidant May Protect Against Radiation Exposure The antiradiation properties of the ecdysteroid-containing compoundsNaringin, a citrus flavonone, protects against radiation-induced chromosome damage in mouse bone marrow– The grapefruit flavanone naringin protects against the radiation-induced genomic instability in the mice bone marrowEnhancement of antiradiation potential of some aminothiols by beta-caroteneCaffeine protects mice against whole-body lethal dose of gamma-irradiation –Radioprotective potential of Rosemarinus officinalis against lethal effects of gamma radiation Radioprotective effects of Aloe vera leaf extract on Swiss albino mice against whole-body gamma irradiation.  — Radioprotective influence of Mentha piperita (Linn) against gamma irradiation in mice: Antioxidant and radical scavenging activity

Radioprotective Health Supporting foods and Herbs and supplements

Baking soda (sodium bicarbonate) baths

If you had an X-rays or radiation treatments for cancer, A radionics practitioner said that you can soak your body in baking soda baths to help pull out the radiation from your body. If you look all over the web, there is talk about patients who have had mouth cancer who were given radiation treatments to get rid of the cancer, and then told to gargle with baking soda mixed in water.”

Baking soda (sodium bicarbonate) rinses

“The most effective measure to treat RT-induced mucositis in patients with head and neck cancer is frequent oral rinsing with a sodium bicarbonate rinse, to reduce the amount of oral microbial flora.” (Oncol Nurs Forum. 2002 Aug;29[7]:1063-80. A research review of the current treatments for radiation-induced oral mucositis in patients with head and neck cancer. Shih A, Miaskowski C, Dodd MJ, Stotts NA, MacPhail)

Baking soda (sodium bicarbonate) plus sea salt baths

Take 20-minute baking soda/ sea salt baths (mixing half a pound of salt and half a pound of baking soda) to drain the body of DOR (deadly orgone energy as described by Dr. Wilhelm Reich) etc. Since such baths are “energy draining”, she advises to be careful when getting up from them (circulation) and lie naked in the sun afterwards to recharge one’s battery. I would add the salt when the bathtub is somewhat filled already to avoid possible damage to any metal parts.

Cysteine hydrochloride

Dr. Donsbach writes: “An amino acid, cysteine protects against the damage of radiation by terminating the free radicals produced by ionizing radiation.” Cysteine, together with methionine, cystine,Alpha Lipoic Acid, Taurine and their derivatives, is numbered among the “sulphurated amino acids” due to the fact that these amino acids contain sulfur in addition to carbon, hydrogen, nitrogen and oxygen. Sulfurated amino acids.

Radioprotective Herbs: LarreaRx chapparal – Burdock root – Mint extract- Curcumin extract – Tumeric spice – Boswellia extract- Periwinkle- Basil- Celery Root- Dandelion-Milk Thistle  

Post Radiation Drug Therapies ACE inhibitors for the kidney, lung and CNS)- Growth factors (G-CSF, GM-CSF, KGF, EPO) to treat the–bone marrow, whole body–• Chelating and isotope-competing agents (Prussian Blue, 7–DTPA, EDTA, potassium iodide, penicillamine, alginates) for the thyroid and bone marrow Pentoxifylline, Vitamin E and SOD to treat fibrosis– Antiemetics to target the GI tract and CNS- Pentoxifylline for fibrosis– Amifostine (anticarcinogenic effects) for mutagenesis-carcinogenesis (given within 3 hours of exposure)- Tempol and other nitroxides for the whole body and fibrosis-• Stem cell transplants (bone marrow, umbilical cord blood, peripheral blood, liver, CNS) for the bone marrow, CNS, liver.

Alkalinizers to Remove Uranium and other Radioisotopes EO Electrolyzed Water — TriSalts – Ecological Formulas BioAlkalinizer – BioGenesis -Buffered Vitamin C – Ca, Mg, Zn – Ascorbates




Spirulina reduces the quantity of Radioactive Isotopes absorbed into the body and thereby helps to counteract the toxic effects of exposure to Radioactivity. 


Animal Organ Supplements


Spleen Extract counteracts the damage to the Immune System caused by exposure to Radioactivity.—


Herbs and Supplements For Anti-Radiation

 FAloe vera (gel applied topically) counteracts the ability of Radioactivity exposure to cause Radioepithelitis (destruction of the epithelium of the Skin). 

FChaparral protects the body from the toxic effects of Radioactivity.

FGinkgo biloba protects the body from the damage to Chromosomes associated with exposure to Radioactivity. 

FSarsaparilla protects against the toxic effects of Radioactivity.

FTea (especially Green Tea) protects the body’s Cells against the damage caused by Radioactivity (due to the Epigallo-Catechin-Gallate (EGCG) content of Green Tea).

FIodine alleviates Radiation Sickness (specifically the damage to the Thyroid) that occurs as a result of exposure to Radioactivity (Radioactive Iodine-131).  The best form of Iodine for protecting against Radiation Sickness after exposure to excessive Radioactivity is Potassium Iodate. 




FEpigallo-Catechin-Gallate (EGCG) protects the body’s Cells against the damage caused by Radioactivity.

FLuteolin protects the body’s Cells against the damage caused by Radioactivity. 



FZinc (especially Zinc Aspartate consumed orally) reduces the lethality of exposure to Radioactivity (when Zinc is consumed prior to exposure to Radioactivity).



 FLipoic Acid reduces the toxic effects of exposure to Radioactivity: Persons exposed to Radioactivity after the Chernobyl incident (in Russia) who received Lipoic Acid supplements exhibit greatly lowered oxidative damage as a result of Radioactivity exposure.

FVitamin C improves the ability of the body to resist the toxic effects of exposure to Radioactivity: Guinea pigs that were supplemented with large dosages of Vitamin C combined with Bioflavonoids were able to withstand twice the known lethal dosages of Radioactivity. Vitamin C (10 grams per day) decreases the Bleeding and Cell degeneration that occurs as a result of exposure to Plutonium Radioactivity.— Vitamin E and C reduced radiation-induced mutations and chromosomal damage in mammalian cells, and radiation-induced lethality


FCysteine protects the body’s Cells from many of the toxic effects of exposure to Radioactivity.

FFFRadiobiological studies have identified several radioprotective compounds some of which are non-toxic to humans. The identification that sulfhydryl (SH)-compounds are potent radioprotective agents is considered as one the most important discoveries in applied radiobiological research [5]. Unfortunately, most widely studied SH-compounds like cysteamine, cystamine and aminoethylisothiourea dihydrobromide (AET) and a cysteamine analogue, amifostine, were toxic to humans . However, compounds such as N-acetylcysteine (NAC) and alpha-lipoic acid that are rapidly absorbed and elevate intracellular levels of glutathione (perhaps the most ubiquitous endogenous SH-compound in ameliorating sources of oxidative stress), are of radioprotective value  and are, within certain dose ranges, non-toxic to humans. Dietary antioxidants such as vitamins E, C and beta-carotene are of radioprotective value, but very little attention has been given to these compounds with respect to their use in protecting normal tissue against radiation damage in humans. It is therefore possible to develop a non-toxic, cost-effective mixture of antioxidants (dietary and glutathione-elevating agents) that can provide biological protection against radiation damage based on several criteria including mutations. Indeed, such radioprotective products have been patented (pending), and are available commercially. In vitro, animal and human studies that support the rationale of these radioprotective products are described below.— Vitamin E and selenium reduced radiation-induced transformation in cell culture; the combination was more effective than the individual agents ..


FL-Dopa protects the body’s Cells from many of the toxic effects of exposure to Radioactivity.




FHyaluronic Acid (cream applied topically) counteracts the ability of Radioactivity exposure to cause Radioepithelitis. 




FBeta-Carotene protects against many of the toxic effects of exposure to Radioactivity. — Natural beta-carotene protected against radiation-induced neoplastic transformation in cell culture

FLycopene protects against many of the toxic effects of exposure to Radioactivity. 


FMelatonin protects the body’s Cells and tissues against the toxic effects of exposure to Radioactivity. 


Evaluation of silymarin as a promising radioprotector.

Z Naturforsch C. 2010 May-Jun;65(5-6):337-46

Authors: Adhikari M, Arora R, Chawla R, Sharma J, Dhaker AS, Gupta D, Dubey N, Kumar R, Ivanov V, Gadjeva V, Gevrenova R, Sharma RK

Silymarin, a purified extract of seeds of Silybum marianum L. and well known for its hepatoprotective abilities, has been evaluated for inherent utility as a radioprotective agent. A fraction (INM-7035) was authenticated by characterizing the percentage composition of silybin A and B (39.9% and 57.4%). Free radical scavenging activities of INM-7035 against superoxide radicals (>68%), hydroxyl radicals (>33.75%), DPPH (67.2%), and ABTS (32.4%) were also evaluated. The fraction chelated (>30%) ferrous ions, thereby able to restrict amplification. INM-7035 exhibited >50% peroxyl radical scavenging activity in the lipid phase along with dose-dependent (R2 = 0.990) reducing power in the aqueous phase. Radiation-induced free radical flux can lead to disruption of biomolecules like membrane lipids. INM-7035 completely inhibited lipid peroxidative stress in case of membranes against supralethal radiation stress in the liposomal system. The ability of INM-7035 to modulate the levels of NF-kappaB, indicated its inherent potential as a radioprotective bioactive constituent.—PMID: 20653235 [PubMed – indexed for MEDLINE]


Formulations and Recipes

Ø Recipe Liver and Thyroid and Brain Effect+—combine milk thistle seeds 1 teaspoon with sea weed 2 oz and sage 1 /2 tsp dry 2-3 sprigs fresh and dandelion ( luteolin ) 2 oz of root or leaf- and water 2-3 pints–make a tea with this as a protectant for radiation exposure—liver regenerator—hormone balancer—thyroid regulator—brain protectant—mood stabilizer—adrenal support—blood support—

Ø Recipe—Using lugols iodine or SSKI or Iodoral supplements at an 80 mg a day in divided small doses will assist the body in the removal of Radiation Poisoning—taking any of these supplements prior to taking a Anti Radiation bath will assist in not only detoxing the rads out but protecting the thyroid against the chlorine in the water

Ø Recipe—Anti Radiation Bathing —take ½ – 1 cup of baking soda and add ½ – 1 cup of Epsom salt and ½- 1 cup of bleach—mix well in the bath—as hot as you can tolerate this—and sit in minimally for 20 minutes—when arising from the bath Do not rinse or very superficially and allow to let sit on the surface of the skin

Ø Recipe Bioflavonoid Peel the Citrus fruits like a potato—what you will have is a higher amount of the components of the Bioflavonoid—you can as well use the essential oils of any citrus and spray them on the bioflavonoid and allow to dry—powder this down and sift after drying—and consume in juices — sprinkle on foods ( you can add potassium chloride or potassium iodide with this and apply it to your foods)  you can as well before you dry this apply either a lugols solution or a SSKI solution to this to have a added benefit by apply 1-2 drops of the sski or 2-4 drops of the lugols to each slice and then allow to dry and then powder down the and again apply this on your foods—




Plant Antioxidant May Protect Against Radiation Exposure

ScienceDaily (Sep. 24, 2008) — Resveratrol, the natural antioxidant commonly found in red wine and many plants, may offer protection against radiation exposure, according to a study by the University of Pittsburgh School of Medicine. When altered with acetyl, resveratrol administered before radiation exposure proved to protect cells from radiation in mouse models. The study, led by Joel Greenberger, M.D., professor and chairman of the Department of Radiation Oncology at the University of Pittsburgh School of Medicine, is overseen by Pitt’s Center for Medical Countermeasures Against Radiation. The center is dedicated to identifying and developing small molecule radiation protectors and mitigators that easily can be accessed and administered in the event of a large-scale radiological or nuclear emergency. “New, small molecules with radioprotective capacity will be required for treatment in case of radiation spills or even as countermeasures against radiological terrorism,” said Dr. Greenberger. “Small molecules which can be easily stored, transported and administered are optimal for this, and so far acetylated resveratrol fits these requirements well.””Currently there are no drugs on the market that protect against or counteract radiation exposure,” he added. “Our goal is to develop treatments for the general population that are effective and non-toxic.”Dr. Greenberger and his team are conducting further studies to determine whether acetylated resveratrol eventually can be translated into clinical use as a radioprotective agent. In 2004, this same team of researchers identified the drug JP4-039, which can be delivered directly to the mitochondria, the energy producing areas of cells. When this occurs, the drug assists the mitochondria in combating radiation-induced cell death. The results of the research were presented during the American Society for Therapeutic Radiology and Oncology’s (ASTRO) 50th Annual Meeting in Boston.The abstract, “Acetylated resveratrol: A new small molecule radioprotector,”was presented at a poster discussion session on  Sept. 23. The study was funded by a $10 million grant from the National Institute of Allergy and Infectious Diseases to establish the Center for Medical Countermeasures Against Radiation at the University of Pittsburgh.—Adapted from materials provided by University of Pittsburgh Schools of the Health Sciences, via EurekAlert!, a service of AAAS.


The antiradiation properties of the ecdysteroid-containing compounds]-

[Article in Russian]—Shevchenko OG, Zagorskaia NG, Kudiasheva AG, Shishkina LN. Abstract—The antiradiation properties of the ecdysteroid-containing preparations (“serpisten” and inokosterone) are studied under their application before or after the 22.6 cGy chronic low intensity gamma-irradiation of mice. It is shown that the antiradiation of these compounds depend on the dose of preparations and time of the application before or after irradiation of mice. “Serpisten” prevented the decrease of the growth of the body mass of irradiated mice. The normalization of the phospholipid composition of the mice liver and blood erythrocytes for the most investigated parameters revealed under the application of this compound at the dose of 50 mg/kg after the irradiation of animals. The capacity of “serpisten” to decompose of peroxides is shown in vitro. Inokosterone had the certain anabolic properties, caused the normalization of the total peroxidase activity of blood and intensity of the lipid peroxidation (LPO) in brain and in liver, and also the repair of the interrelation between the LPO intensity and catalase activity in the irradiated mice liver. The obtained results allow to conclude that the antiradiation properties of the ecdysteroid-containing preparations under the chronic low intensity irradiation of animals at the low dose due to their capacity to depend on the LPO regulatory system parameters.PMID: 17953438 [PubMed – indexed for MEDLINE]



 Naringin, a citrus flavonone, protects against radiation-induced chromosome damage in mouse bone marrow.

Jagetia GC, Venkatesha VA, Reddy TK.—-Department of Radiobiology, Kasturba Medical College, Manipal 576119, India.


Free radicals are responsible for the induction of damage to the cellular DNA that leads to the formation of chromosome aberrations. Antioxidants are known to scavenge free radicals, thereby decreasing the degree of such effects. Radiation is a well-known inducer of free radicals and compounds that can scavenge free radicals may reduce radiation-induced DNA damage. Naringin, a bioflavonoid predominant in grapefruit and other citrus fruits, has been found to scavenge free radicals, therefore it may also reduce radiation-induced damage. The aim of the present study was to evaluate the radioprotective action of 2 mg/kg naringin in the bone marrow of mice exposed to different doses of (60)Co gamma-radiation by scoring the frequency of asymmetrical chromosomal aberrations. The irradiation of mice resulted in a dose-dependent elevation in the frequency of aberrant cells, acentric fragments, chromatid and chromosome breaks, dicentrics and exchanges. All these aberrations were elevated with scoring time up to 24 h post-irradiation and declined thereafter, except chromatid breaks, which were maximum at 12 h post-irradiation. Treatment of mice with 2 mg/kg body wt naringin before exposure to various doses of gamma-radiation resulted in a significant reduction in the frequencies of aberrant cells and chromosomal aberrations like acentric fragments, chromatid and chromosome breaks, centric rings, dicentrics and exchanges. The evaluation of free radical scavenging activity of naringin revealed a dose-dependent scavenging of hydroxyl, superoxide and 2,2 equal to or precedes -diphenyl-1-picryl hydrazyl radical. Naringin at 5 microM scavenged the 2,2-azino-bis-3-ethyl benzothiazoline-6-sulphonic acid cation radical very efficiently, where a 90% scavenging was observed. Our study demonstrates that naringin can protect mouse bone marrow cells against radiation-induced chromosomal damage.– PMID: 12840107 [PubMed – indexed for MEDLINE]


The grapefruit flavanone naringin protects against the radiation-induced genomic instability in the mice bone marrow: a micronucleus study.

Jagetia GC,Reddy TK.

Department of Radiobiology, Kasturba Medical College, Manipal 576119, India.


The effect of various doses, viz. 0, 0.5, 1, 2, 4, 6 and 8 mg/kg body weight of naringin (NIN) (a citrus flavanone) was studied on the alteration in the radiation-induced micronucleated polychromatic (MPCE) and normochromatic (MNCE) erythrocytes in mouse bone marrow exposed to 2 Gy of 60Co gamma-radiation. The treatment of mice with various doses of NIN before exposure to 2 Gy resulted in a significant decline in the frequency of MPCE when compared to the non-drug-treated irradiated control. However, the greatest reduction in MPCE was observed for 2mg/kg body weight NIN, accompanied by a highest PCE/NCE ratio when compared with the non-drug-treated irradiated control. Therefore, further studies were carried out using this dose of NIN, where the animals were administered with 2mg/kg body weight of NIN before exposure to 0, 0.5, 1, 2, 3 and 4 Gy of gamma-radiation. The frequency of MPCE and MNCE increased in a dose-dependent manner in both the non-drug-treated irradiated control and NIN-pretreated irradiated groups up to a dose of 2 Gy, while a further increase in the irradiation dose resulted in a significant decline in MPCE and MNCE frequencies in both groups. Pretreatment of mice with 2mg/kg body weight of NIN resulted in a significant decline in the frequencies of MPCE and MNCE. NIN treatment not only reduced the frequency of MPCE with one micronucleus, but also of MPCE with multiple micronuclei (MN), indicating its ability to reduce complex chromosome aberrations. Conversely, the PCE/NCE ratio declined in a dose-dependent manner in both groups. The treatment of mice with NIN before exposure to different doses of gamma-radiation resulted in the inhibition in this decline in the PCE/NCE ratio. Our study demonstrates that NIN is able to protect mouse bone marrow cells against the radiation-induced DNA damage and decline in the cell proliferation as observed by a reduction in the micronucleus frequency and an increase in PCE/NCE ratio, respectively, in the NIN-pretreated irradiated group. –PMID: 12160890 [PubMed – indexed for MEDLINE]


Enhancement of antiradiation potential of some aminothiols by beta-carotene.

Al-Wandawi HK.

Division of Environmental Researches and Workers Protection, Iraqi Atomic Energy Commission, Baghdad, Iraq.


In the present study, protection of mice, BALB/c inbred as measured by survival at 30 days against whole-body gamma exposure at two dose levels, namely, 7.60 and 10.12 Gy by prior irradiation treatment with combination of beta-carotene, N-(2-mercapto-propionyl)-glycine (MPG) and S-(2-aminoethyl) isothiouroniumbromide hydrobromide (AET), is reported. It was found that administration of beta-carotene (1 mg per 20 g body mass) and 24 h before whole-body irradiation (7.60 Gy) had significantly improved the post-irradiation survival. It was also found that administration of a combination of AET (260 mg per kg body mass) and MPG (60 mg per kg body mass) 20 min before exposure to 7.60 Gy gamma irradiation to mice which have been treated with beta-carotene (1 mg per 20 g body mass) 24 h before exposure had resulted in 80% survival in comparison to 10% survival recorded for control animals. On the other hand, when the animals were exposed to a higher dose (10.12 Gy) under similar experimental conditions as above, a significant improvement in survival was observed during the first 10 days following the exposure but only a slight effect afterward. On the other hand, the response of male and female mice 10 days after exposure to the above radiation dose indicated that the females were more radioresistant than the males. Copyright 2003 S. Karger AG, Basel— PMID: 12743471 [PubMed – Indexed for MEDLINE]


Caffeine protects mice against whole-body lethal dose of gamma-irradiation.

George KC, Hebbar SA, Kale SP, Kesavan PC.

Biosciences Group, Bhabha Atomic Research Centre, Trombay, Mumbai, India.


Administration of caffeine (1,3,7-trimethylxanthine), a major component of coffee, to Swiss mice at doses of 80 or 100 mg/kg body weight 60 min prior to whole-body lethal dose of gamma-irradiation (7.5 Gy) resulted in the survival of 70 and 63% of animals, respectively, at the above doses in contrast to absolutely no survivors (LD-100/25 days) in the group exposed to radiation alone. Pre-treatment with a lower concentration of caffeine (50 mg/kg) did not confer any radioprotection. The protection exerted by caffeine (80 mg/kg), however, was reduced from 70 to 50% if administered 30 min prior to irradiation. The trend statistics reveal that a dose of 80 mg/kg administered 60 min before whole-body exposure to 7.5 Gy is optimal for maximal radioprotection. However, caffeine (80 mg/kg) administered within 3 min after irradiation offered no protection. While there is documentation in the literature that caffeine is an antioxidant and radioprotector against the oxic pathway of radiation damage in a wide range of cells and organisms, this is the first report demonstrating unequivocally its potent radioprotective action in terms of survival of lethally whole-body irradiated mice.


Radioprotective potential of Rosemarinus officinalis against lethal effects of gamma radiation : a preliminary study.

Jindal A, Soyal D, Sancheti G, Goyal PK.

Radiation and Cancer Biology Laboratory, Department of Zoology, University of Rajasthan, Jaipur – 302 004, India.


The radioprotective effect of Rosemarinus officinalis extract (ROE) was studied in mice exposed to 8 Gy of gamma radiation. The optimum dose for radioprotection was determined by administering 100, 200, 400, 800, 1000, 1500, and 2000 mg/kg body weight of ROE orally once daily, consecutively for five days before irradiation. Treatment of mice with ROE, delayed the onset of mortality and reduced the symptoms of radiation sickness when compared with the non-drug-treated irradiated controls. The dose of ROE found to be most effective against radiation was 1000 mg/kg body weight because this dose increased the survival time and reduced the mortality rate of mice significantly. Body weight loss in ROE administered irradiated animals was significantly less in comparison with animals who were given radiation treatment alone. Furthermore, irradiation of animals resulted in an elevation in lipid peroxidation (LPx) and a significant decrease in glutathione (GSH) in blood and liver. Conversely, administration of animals with ROE before irradiation caused a significant decline in LPx accompanied by a significant increase in GSH concentration. The present study demonstrates that Rosemarinus officinalis leave extract is a good radioprotector.— PMID: 17341204 [PubMed – indexed for MEDLINE]


Radioprotective effects of Aloe vera leaf extract on Swiss albino mice against whole-body gamma irradiationGoyal PK, Gehlot P.

Department of Zoology, University of Rajasthan, Jaipur – 302 004, India.


The skin, being a cell-renewal system, is one of the first organs to be affected in total-body irradiation during radiotherapy. An attempt has been made in the present study to explore radiation-induced biochemical alterations caused by whole-body gamma irradiation and their modulation in Swiss albino mice by Aloe vera leaf extract (AVE). Mice were selected for this study from an inbreed colony and divided into four different groups: I (double-distilled water-treated group): considered as normal; II (Aloe vera-treated group): the animals were administered 1 g/kg body-wt/day Aloe vera leaf extract; III (radiation-treated group): the animals were exposed to 6 Gy gamma radiation at the dose rate of 0.96 Gy/min; and IV (combination group): animals were administered Aloe vera leaf extract continuously for 15 consecutive days, and on the 15th day they were irradiated to 6 Gy gamma radiation after 30 minutes of extract administration. The animals from the above groups were autopsied after 6 hours, 24 hours, and at 3, 7, 14, and 21 days of radiation. Biochemical estimations of DNA, lipid peroxidation, glutathione, catalase, and superoxide-dismutase were made. Total DNA, catalase, superoxide dismutase (SOD) activity in the skin, and glutathione (GSH) in the liver and blood significantly decreased compared to normal, but lipid peroxidation (LPO) in the liver and blood increased in the irradiated control group. In contrast, in experimental animals, DNA, catalase, and SOD in the skin and GSH in the liver and blood increased significantly, whereas LPO in the liver and blood decreased in comparison to irradiated control animals. Thus, Aloe vera leaf extract is found to have damage-resistant properties against radiation-induced biochemical alterations in Swiss albino mice.– PMID: 19392655 [PubMed – indexed for MEDLINE]


Radioprotective influence of Mentha piperita (Linn) against gamma irradiation in mice: Antioxidant and radical scavenging activity.

Samarth RM, Panwar M, Kumar M, Kumar A.Radiation & Cancer Biology Laboratory, Department of Zoology, University of Rajasthan, Jaipur, India.

Retraction in:   Int J Radiat Biol. 2007 Nov-Dec;83(11-12):897.


PURPOSE: To evaluate the radiomodulatory influence of a leaf extract of Mentha piperita (Linn) on hepatic antioxidant status and lipid peroxidation in Swiss albino mice. MATERIALS AND METHODS: Animals were given either double distilled water or leaf extract of M. piperita orally (1 g/kg bwt/day) once a day for three consecutive days. Thirty min after the last treatment mice were exposed to 8 Gy of gamma radiation. Mice were autopsied at 30 min post-irradiation. Biochemical parameters were studied to assess the radioprotective effect of leaf extract of M. piperita. RESULTS: Animals pretreated with leaf extract of M. piperita and exposed to 8.0 Gy gamma radiation showed a significant increase in the activities of reduced glutathione content (p < 0.001), glutathione peroxidase (p < 0.005), glutathione reductase (p < 0.001), glutathione S-transferase (p < 0.005), superoxide dismutase (p < 0.005), and catalase (p < 0.005). Irradiated group pretreated with leaf extract of M. piperita showed significant decrease in malondialdehyde (MDA) formation in liver. The leaf extract of M. piperita showed strong radical scavenging activity in both the 1, 1 diphenyl-2-picryl hydrazyl radical (DPPH*) and 2, 2 azinobis (3-ethylbenzothiazoline-6-sulfonic acid) radical cation (ABTS*+) assays. CONCLUSIONS: The results of the present investigation suggest the antioxidant and free radical scavenging activities of leaf extract of M. piperita are the likely mechanism of radiation protection.

Radiation Protocols Part 2

Go to the type in herbsplusbeadworks  and look at the SSKI and the Lugols Iodine Making—and you can go to Http://  and Look at Radiation Protocols Part 2


Radioprotective Herbs Plus Nutrients and Supplements


When and how to use multiple antioxidants for radiation protection in humans

Commonly used doses in diagnostic procedures are listed in Tables 1 and 2 . It is suggested that patients receiving diagnostic radiation doses take orally an appropriately prepared multiple antioxidants pill once 30 min to 60 min before radiation procedure. Radiation workers are advised to keep the body level of antioxidants high at all times by taking the appropriate multiple vitamins with antioxidants, B-vitamins and appropriate minerals, but no iron, copper or manganese. They may take a booster dose antioxidant pill 30 min to 60 min before exposure to higher radiation doses is anticipated. Frequent-flyers may also follow the same dose and dose schedule as radiation workers. The proposed strategy of increasing antioxidant levels in the body can also be implemented in populations living in regions with high background radiation that show increased levels of intermediate risk markers such as increased chromosomal damage or evidence of increased oxidative damage such as lipid peroxidation. Such a strategy may reduce the levels of intermediate risk markers, and thereby, protect against long-term adverse health consequences of radiation exposure among this population. The proposed antioxidant strategy, in addition to potassium iodide, should be adopted in the event of nuclear accident or explosion of a “dirty bomb“. The efficacy of antioxidant strategy for biological radiation protection in humans against low doses of radiation has not been tested; however, all proposed antioxidant ingredients have exhibited radiation protection in humans and/or in laboratory experiments. Direct experiments with radioprotective antioxidant formulations cannot be performed in humans for obvious ethical reasons. However, implementation of the proposed recommendation may allow prospective studies among radiation workers to determine its efficacy in reducing health risks of low doses of radiation in present and future generations

Potassium iodide as a radioprotective agent
The increased use of radioactive iodine (131I) in research, diagnosis and treatment of thyroid diseases, and the release of 131I during nuclear explosion made it imperative that all radiation workers working with 131I take potassium iodide pills. Since iodine selectively accumulates in the thyroid gland, and the thyroid is considered a very radiosensitive organ, it was thought that the intake of potassium iodide would saturate the thyroid gland with iodine, and thus would prevent the accumulation of radioactive iodine in the thyroid. Thus potassium iodide remains a valuable strategy for protecting the thyroid gland against damage produced by radioactive iodine. However, this agent does not protect any other radiosensitive organ against -radiation that is produced by radioactive iodine. In addition, potassium iodide does not protect against damage produced by other radioactive isotopes or other radiation sources such as X-irradiation or -irradiation.
Therefore, additional non-toxic agents must be identified to protect humans against radiation damage produced by sources other than radioactive iodine.

Multiple antioxidants as radioprotective agents
Although earlier identified radioprotective SH-compounds were found to be toxic in humans, glutathione-elevating agents such as N-acetylcysteine (NAC) and -lipoic acid, that are non-toxic (within certain concentration ranges) to humans, protect normal tissues against radiation damage [48, 60]. In addition to glutathione-elevating agents, dietary antioxidants such as vitamins E, C and -carotene are also of radioprotective value [57, 6082], but very little attention has been given to these agents with respect to their use in protecting normal tissue against radiation damage in humans. Based on published data on antioxidants and radiation protection, it is possible to develop a non-toxic, cost-effective mixture of antioxidants (dietary and glutathione-elevating agents) that can provide biological protection against radiation damage in humans. Indeed, such formulations, referred to as Bio-Shield, (patent pending) are available commercially (Premier Micronutrient Corporation, Nashville, TN). In vitro animal and human studies that support the rationale of using multiple antioxidants in reducing the risk of radiation damage in humans are described below.

In vitro studies supporting the scientific rationale for using antioxidants for radiation protection in humans
It has been reported that mitotic cells, which are most sensitive to radiation, have the lowest levels of SH-compounds, whereas S-phase cells, which are the most resistant to radiation, have the highest levels of these compounds [83]. The role of SH-compounds in radiation protection was further substantiated by the fact that an elevation of the intracellular levels of these compounds in mitotic cells makes them radioresistant to the same level as S-phase cells].
It has been shown that vitamin E and selenium reduced radiation-induced transformation in cell culture; the combination was more effective than the individual agents  Natural -carotene protected against radiation-induced neoplastic transformation in cell culture.  Vitamins E and C reduced radiation-induced mutations and chromosomal damage in mammalian cells, and radiation-induced lethality In vivo (animal) studies supporting the scientific rationale for using antioxidants for radiation protection in humans Alpha-lipoic acid, a glutathione-elevating agent, increases the LD50 in mice with a dose reduction factor (DRF) of 1.26  Vitamin E, Vitamin C and -carotene protected rodents against the acute effects of irradiation. Vitamin A and -carotene protected normal tissue during radiation therapy of cancer in an animal model . A combination of vitamin A, C and E protected against radiation-induced myelosuppression during radiation therapy of cancer in an animal model. It has been reported that L-selenomethionine and several different types of antioxidants (vitamin C, vitamin E, glutathione, n-acetylcysteine (NAC), lipoic acid and co-enzyme Q10) protected human cells in culture and rats in vivo against oxidative stress produced by photons, protons and 1 GeV iron ions [41].

Human studies supporting the scientific rationale for using antioxidants for radiation protection
Vitamin A and NAC may be effective against radiation-induced carcinogenesis [60]. Alpha-lipoic acid treatment for 28 days lowered lipid peroxidation among children chronically exposed to low doses of radiation in the area contaminated by the Chernobyl nuclear accident [48]. In another study, -carotene reduced cellular damage in the above population of children [47]. A combination of vitamin E and -lipoic acid was more effective than the individual agents [48]. -carotene also protected against radiation-induced mucositis during radiation therapy of cancer of the head and neck [80]. A combination of dietary antioxidants was more effective in protecting normal tissue during radiation therapy than the individual agents [81, 82].


Radioprotective Potential of Plants and Herbs against the Effects of Ionizing Radiation

C Jagetia G.

Department of Radiobiology, Kasturba Medical College, Manipal-576 104, India.

Ionizing radiations produce deleterious effects in the living organisms and the rapid technological advancement has increased human exposure to ionizing radiations enormously. There is a need to protect humans against such effects of ionizing radiation. Attempts to protect against the deleterious effects of ionizing radiations by pharmacological intervention were made as early as 1949 and efforts are continued to search radioprotectors, which may be of great help for human application. This review mainly dwells on the radioprotective potential of plant and herbal extracts. The results obtained from in vitro and in vivo studies indicate that several botanicals such as Gingko biloba, Centella asiatica ( gota kola ) , Hippophae rhamnoides ( Sea Buckthorn), Ocimum sanctum ( Holy Basil ) , Panax ginseng ( American or Canadian Ginseng ), Podophyllum hexandrum ( Himalayan May Apple ), Amaranthus paniculatus ( Foxtail ), Emblica officinalis ( Indian goose berry or AMLA ), Phyllanthus amarus, Piper longum ( Long Pepper ), Tinospora cordifoila ( Guduchii ), Mentha arvensis ( Wild Mint ), Mentha piperita ( Pepper Mint ), Syzygium cumini (Jambolan plum ), Zingiber officinale  ( Ginger ), Ageratum conyzoides ( Billy Goat  Weed ), Aegle marmelos (Indian Bael Fruit ) and Aphanamixis polystachya (amoora ) protect against radiation-induced lethality, lipid peroxidation and DNA damage. The fractionation-guided evaluation may help to develop new radioprotectors of desired activities.

 Recipe With Herbals To Increase Adequate Protection Against RadiationMake a tea with any of these or a combination—Use as a tincture—add to foods and soups or saladsthe idea is to eithr build this inside to have an offsetting effect or to increase the ability to remove the excess rads you may wind up with


Reducing Radiation Damages with Bicarbonate

So deep are the protective, buffering and neutralizing properties of bicarbonate that it is used even with radiation exposure to protect the kidneys and other tissues. In a world that is already overexposed to uranium and mercury, sodium bicarbonate becomes even more important because mercury and uranium oxide directly attack the nuclear material and mitochondria of the cells.  The oral administration of sodium bicarbonate diminishes the severity of the changes produced by uranium in the kidneys.[1] It does this for all the heavy metals and other toxic chemicals including chemotherapy agents, which are highly lethal even in low dosages. After depleted uranium weapons were used starting in the first Gulf War, the United States has polluted the world with uranium oxide and it is showing up more and more in tests doctors perform. With a half life of several billion years we had better be prepared to get used to dealing with the toxic effects and help our bodies clear it more easily through the kidneys. Sodium bicarbonate is an absolute must item in any field hospital and it should be in used and recommended in all clinics and be present in every home medicine cabinet. In reality we need a more descriptive image for bicarbonate. Its pharmacological characteristics, even though widely used, are not well understood. What does bicarbonate really do? Well, instead of a muscleman with a mallet, an even better image would be a strong janitor mopping up the messes and carrying the poisons away. This strong janitor protects tissues and leaves an alkaline film or trail behind to make sure everything stays safe. In medicine, sodium bicarbonate is the cleaning and security man proven loyal through decades of faithful service and he can be brought in to provide some sort of protection in cases where people are suffering from radiation toxicity. So useful and strong is sodium bicarbonate that at Los Alamos National Laboratory in New Mexico, researcher Don York has used baking soda to clean soil contaminated with uranium. Sodium bicarbonate binds with uranium, separating it from the dirt; so far, York has removed as much as 92 percent of the uranium from contaminated soil samples. I started writing about baking soda after discovering that the United States Army recommends the use of bicarbonate to protect the kidneys from radiation damage. Blaise W. LeBlanc, a former research chemist with the U.S. Department of Agriculture identified the byproduct hydroxymethylfurfural, (HMF) as a potential culprit in colony collapse disorder of bees. LeBlanc has a solution to minimize HMF toxicity: By adding bases (such as sodium bicarbonate, or baking soda, lime, potash or caustic soda) to HFCS, the pH rises and HMF levels drop. Sodium bicarbonate can safely remove paint, grease, oil and smoke residue, decreasing workers’ exposure to harsh chemicals and eliminating much of the hazardous waste associated with other cleaners. “Sodium bicarbonate is able to clean in areas where other substances pose fire hazards, because baking soda is a natural fire extinguisher,” says Kenneth Colbert, a general manager for Arm & Hammer. This is the reason it’s used by oncology centers to control chemo agent spills and its actually used intravenously to protect patients from the hazardous toxicity of chemotherapy. If the bombs start dropping anywhere on earth you will need to have a large amount of sodium bicarbonate on hand. Minimum stocks should be 25 or 50 pounds. You will also need iodine, magnesium chloride—-


Acute DU Deplete Uranium Exposure or Nuclear Event -Detox Bath


The Epson Salt Bath:

Use after exposure to DU, medical radiation,nuclear radiation or dirty bombs, high altitude radiation exposure or irradiated foods. Dissolve 1 pound of sea salt or rock salt and 1 pound of baking soda in a hot bath — as hot as can be tolerated — and soak into the water until the bath becomes cool. This usually takes about 20-25 minutes. Afterwards, do not shower or rinse the salt off your body for 4-8 hours

The Clorox Heavy Metal Bath:

Chorox bath can aid heavy metal removal from the body: DU and particulate radioisotopes from nuclear explosions or dirty bombs. Just add 1 cup of regular Clorox bleach to a tub of hot water — as hot as can be tolerated Soak in it until the water becomes cool or body temperature.Don’t wash off for at least 4 hours and make sure you’re using sufficient water


Inhalation Exposure Low Level Radiation Detoxification Bath:

Dissolve 2 pounds of baking soda in a tub of hot water Soak in it until the water becomes cool or body temperature. Drink Green tea or better still take Polyphenon 100mg 6 caps every four hours to chelate toxins and heavy metals Drink EO Electrolyted Alkaline water 1 oz per 8 to 10 ounces of filtered water ( Multi Pure Water or Pure Water Systems recommended-RO or Distilled with the alkali added

Higher Level Radiation Fallout Detoxification Baths:

Parcells recommended a stronger mixture of drinking an 8-ounce glass of water containing ¼-teaspoon natural sea salt and ¼-teaspoon baking soda. Drink every 2-3 hours and each glass was to be taken with 3 caps of calcium – magnesium taurate If symptoms in the head, sinuses, chest, glands,–neck, throat, they were to add ¼-teaspoon of cream of tartar to the mixture.–Don’t shower for at least 4 hours after the bath. Bathing in the evening is more effective Please don’t mix the different bath ingredients

Higher Level Radiation Fallout Detoxification Baths 2:

Alternate the baths on different evenings or one bath in the morning and the other at night. In more severe cases a baking soda bath in the morning and Clorox bath at night may be useful.


 Radioprotective Herbs Plus Nutrients and Supplements :

Selenium DNA protection -Lycopene antioxidant protection of DNA-Sulphurophane from brassica brussel sprouts and cabbage DNA-Protection –NAC, N-Acetyl Cysteine -Reduced L-Glutathione- SAMe S-Adenosyl Methionine – Polyphenon – FirstLine-Defense Caps —Longevinex – Red Wine Extract 30 capsules Polyphenol Antioxidant Alpha lipoic acid with 12 to 20 Times power with Alpha R-Lipoic (BioGenesis) Reishi mushrooms and Beta Glucans – Aloe Beta 1,3 Glucans Ambrotose – Arabinogalatan and Manapol  1,3 Beta Poly Manose – Alkalinizers to Remove Uranium and other Radioisotopes: EO Electrolyzed Water TriSalts – Ecological Formulas BioAlkalinizer – BioGenesis  Buffered Vitamin C – Ca, Mg, Zn Ascorbates  

Protocols For Radiation Part 1

Here are some things you can do for your benefit–and if you go to and type in herbsplusbeadworks you can see how to make a SSKI solution and a lugols

or go to  and look for Radiation Protocol 1


Radiation Toxicity Antidotes and USES

(C) 2010 by John W. Apsley, II, MD(E), DC –

Q: What can I do now to protect myself from nuclear fall-out arriving from the meltdown of Japan’s nuclear power plants?

A: History has taught us to “hope for the best,” but “plan for the worst” and to educate oneself better than the politicians or their official scientific spokespersons!

The radioactive metals uranium, plutonium, cesium and strontium are of primary concern, right alongside radioactive iodide. Once lodged into our tissues, all will induce lethal tissue ionization, which over decades will derange genetic functions and kill many cells. To avoid this, the metals need to be removed from the cells. Specifically over the long term, radioactive cesium will concentrate in the fatty tissues, radioactive iodine in the thyroid gland and ovaries, strontium in the bone and uranium and plutonium in the liver. For North America, over time this may/will lead to significantly greater levels of cancer or alternative forms of chronic degenerative disease in our children and young adults via the Petkau Effect.(A), (B), 18, 19, 22

The fallout will present in THREE forms or threats to our health:

The first will be the immediate airborne threat over the next several weeks. Depending upon which “starting date” you use (the date of the first detection of radioactive cesium being airborne or when helicopter crews from the USS Ronald Reagan were first documented to have been exposed, i.e., March 14th, 2011), we can expect the West Coast of the U.S. to begin to receive at least minimal fallout within 7 to 10 days or possibly on the weekend of March 19th – 20th, 2011 (see nucelar fallout map above). This may mean that by the 1st of April, the entire region of North America will have undergone the first round of fall-out exposure, even if at undetectable levels in many areas. As the upper air currents repetitively cycle around the globe over and over again, the stock of radioactive airborne materials will fall out as part of normal rainfall or as simple dust particles. For an enlargement of the above map, see:

To track up to the minute levels of excessive radiation here in the U.S., and/or join in this worthy effort, see:


The next threat will be when it enters into our water supply and food chain over the many weeks and perhaps months ahead.

The last threat stems from the extremely low levels of radiation which become ever present due to radioactive materials burrowing into our tissues. This will be by far the more deadly form of radiation poisoning to those living in North America, and is properly called The Petkau EffectThe Petkau Effect is in many ways identical to what happens when underground fires ignite that cannot be extinguished. Indeed, there are locations around the world where abandoned coal mines went ablaze and simply cannot be put out. Some of these have smoldered slowly out of control for 30 years or more. The same may occur within our very own bodies when radioactive iodide, uranium, plutonium, cesium and strontium enter our lungs as we breathe or in through our digestive tract when we eat or drink contaminated foods or water. The Petkau Effect applies to chronic low level exposures and accrues over a long time frame because these radioactive materials will continue to emit toxic ionizing radiation for thousands of years.(A), 22

You don’t need to worry about acute spikes in radiation which are fleeting, since few will ever experience such an event. You should be concerned about tiny scattered particles that settle into our tissues undetected, and not worry about amounts sufficient to trigger Geiger Counter alarms going off at airport check stations. The tiny amounts of exposure which are more likely to occur give off constant ionizing radiation that burns us at the molecular level over many years before causing a diagnosable issue.19

Also, the cloth-paper masks that many people use around their faces are useless to protect from these tiny particles. Sorry folks! You would need advanced charcoal filters that are quite bulky and more expensive to achieve any meaningful “up-front” protection for your lungs. Therefore, be forewarned that any official positions claiming that, “only low levels of radiation have been detected and therefore pose no real health threat to the American public,” are tainted and should be viewed as phraseology more properly akin to either misinformation or outright propaganda.(B), 18 

For scientists, the Petkau Effect may be illustrated as follows:

A long term exposure of extremely low radiation (i.e., one-ten millionth of a rad) was found to be 100 BILLION times MORE lethal than a short term exposure to exceedingly high level radiation (i.e., 10,000 rads per minute). As it turns out, Petkau discovered– that at exceedingly high radiation levels, the abundant free radicals generated in tissues tended to cancel each other out before they could do cellular damage. But at extremely low levels of radiation, these same free radicals – produced in minuscule quantities – remain unchecked. And any steady stream of unchecked free-radicals will efficiently and lethally cleave lipid cellular membranes like a hot knife slicing through butter once they overwhelm and exhaust cellular antioxidant defenses. This dramatically illustrates the non-linear aspects of dose (rads) to lethality. Most scientists specializing in this field are unaware of this fact. And most think strictly in terms of genetic damage, while the above presents its lethal affects upon cell membranes and only secondarily to the genetic core.18 


1.    N-Acetyl-Cysteine (NAC) is the most powerful short term quencher of ionizing radiation. For adults (weighing above 150lbs) , it is taken in dosages of up to 500mg daily. For younger adults weighing 100lbs to 149lbs, 400mg daily affords adequate protection. In children weighing less than 100lbs, but above 50lbs, 200mg daily is a suitable dose. For infants, toddlers or children weighing less than 50lbs, 50mg to 100mg daily may be used in juice, as long as no sensitivity to NAC arises (i.e., light skin rashes). In this manner, NAC may be used daily on an indefinite basis, as it is a harmless amino acid our bodies will use. It is also an excellent remover of toxic metals from the body, such as radioactive uranium. Rarely, some adults are sensitive to NAC, so be aware of special advisories regarding its long term use.6, 7

2.     Liquid iodide is also a first line of defense mineral supplement, since it can out-compete radioactive iodide from entering into our bodies.11 Up to 1,000mcg daily are used for several short weeks. Kelp, Irish Moss or Dulse can then replace the liquid iodide. For adults, up to 5 tablets per meal of any one of these may be wise. But for folks allergic to seafoods or iodide, taking Kelp or liquid iodide is not advised. Kelp is also an excellent remover of toxic metals from the body, especially if high fiber intake is also being incorporated into the diet.9, 10

3.     Chlorella (and other blue green algae) is a superior protector and remover of radioactive metals from the body contains no less than 20 superior neutralizers to– radioactive poisons. 5 per meal (250mg each) is a great dose for adults, 3 per meal for young adults and children, and 1 per meal for the very young. Make sure the Chlorella brand you buy has the outer cell wall “cracked” for best absorption. More may be taken, but it is suggested to not exceed 40 tablets daily.3, 4, 29

4.     High quality bone meal (rich in Calcium & Strontium Hydroxyapatite) will also protect against radioactive strontium poisoning. 3 per meal as labeled is suggested for adults, young adults and children, and 1-2 per meal for the very young.12

5.     Natural Vitamin E Complex – To stop cell membrane destruction. 800iu per day is an excellent dose for average adults, and 400iu per day for young adults and children. Toddlers and infants may be given 100iu per day in juice. (Side Note: Wheat Germ Oil CoQ10(H) and Melatonin…)

6.     Consuming High fiber and seaweed dishes on a regular basis must be used to maximize the best effects of the above tools. These will help insure removal of toxic radioactive metals from the body. On rare occassions, if exposures to radioactive metals become chronic or reaches what are considered high levels, baking soda is an efficient means to remove these metals quickly (especially uranium). For adults under doctor supervision, and when deemed medical necessary, up to 1 teaspoon of baking soda can be taken 2 hours after each meal, plus upon rising and upon retiring, for a maximum total of 7 teaspoons daily over a two week period.14, 15

LONG TERM ANTIDOTES (for maintenance):

  • Bone Meal (protects against radioactive Strontium);
  • Chlorella (protects against most radioactive metals);
  • Kelp (protects against radioactive iodide and other radioactive metals);
  • Probiotics – several billion per meal in capsule format (protects against radioactive Strontium).16, 17


Examples of Protection Schedules According to Body Weight & Budgetary Allowances



Q: What is the one, least expensive method to protect oneself from radioactive fall-out?

Q: What are the top three tools to protect oneself from initial stages of radioactive fall-out?

A: NAC, Kelp and Chlorella. ( Sea Weed and Sea Fungus and Sea Vegetables at a asian market will as well be considered )

Average adults (weighing +150lbs): 500mg NAC at breakfast, 5 Kelp tablets & 5 Chlorella tablets with each meal.

Young adults and larger children (weighing 100lbs – 149lbs): 400mg NAC at breakfast, 3 tablets of Kelp & 3 Chlorella tablets with each meal.

For smaller children (weighing 50lbs – 99lbs): 200mg NAC at breakfast, 1 tablet of Kelp & 1 tablet of Chlorella with each meal sprinkled into juice.

For the very young (weighing less than 50lbs): 50mg to 100mg NAC, 1 tablet Kelp & 1 tablet Chlorella daily sprinkled into juice.



Q: What are the top four tools to protect oneself from initial stages of radioactive fall-out?

A: NAC, Kelp, Chlorella and Vitamn E.

 Average adults (weighing +150lbs): 500mg NAC at breakfast, 5 Kelp tablets & 5 Chlorella tablets with each meal + 1 cap Krill Oil.

 Young adults and larger children (weighing 100lbs – 149lbs): 400mg NAC at breakfast, 3 tablets of Kelp & 3 Chlorella tablets with each meal + 1 cap Krill Oil.

 For smaller children (weighing 50lbs – 99lbs): 200mg NAC at breakfast, 1 tablet of Kelp & 1 tablet of Chlorella with each meal crushed & sprinkled into juice + 1 cap Krill oil squeezed & blended into milk or juice.

 For the very young (weighing less than 50lbs): 50mg to 100mg NAC, 1 tablet Kelp & 1 tablet Chlorella daily crushed & sprinkled into juice + 1 cap Krill Oil squeezed & blended into milk or juice.

Q: If the lipid membranes are the sole issue, what are the best tools to prevent membrane damage during initial stages of contamination?

A: Many physicians would chose R-alpha lipoic acid (thioctic acid) as their first choice since it will powerfully retard lipid membrane ionization. Lipoic acid tends to bind metals and deposit them into the nucleus of cells (as opposed to removing them from the body).24, 26 Therefore, it is best to use Krill oil, or ‘reduced’ co-enzyme Q10 [CoQ10(H)], or Vitamin E and Melatonin to accomplish a better end result. Krill oil, CoQ10(H) and Vitamin E would neutralize the ionizing effects onsite, while Melatonin would also seek to safely remove the offending radioactive particle from the body (via the bile route of elmination provided there is adequate fiber).7, 21, 23, 25  In order of strength, Krill Oil reigns supreme, then CoQ10(H), then Melatonin, and finally Vitamin E. But for the kinds of low dose exposures North America is likely to receive, natural Vitamin E complex should provide adequate protection if taken ahead of exposures. Melatonin is a superior multitasking nutrient and so serves purposes beyond the others. Therefore, this is a prudent tool to include if budget allows. In summary, budget prudently for the suggested schedules below, and if necessary, eliminate Krill oil in favor of Vitamin E, eliminate NAC in favor of extra Chlorella, and substitute Spirulina over Chlorella to save money as necessary.

Average adults (weighing +150lbs): 500mg NAC + 800iu Natural Vit. E at breakfast + 1 cap Krill oil; 5 Kelp tablets, 5 Chlorella tablets with each meal; and 10mg Melatonin at bedtime.

Young adults and larger children (weighing 100lbs – 149lbs): 400mg NAC + 400iu Natural Vit. E at breakfast + 1 cap Krill oil; 3 tablets of Kelp & 3 Chlorella tablets with each meal, and 3mg Melatonin at bedtime.

For smaller children (weighing 50lbs – 99lbs): 200mg NAC + 200iu Natural Vit. E at breakfast + 1 cap Krill oil squeezed & mixed into milk or juice; 1 tablet of Kelp & 1 tablet of Chlorella with each meal; and 1mg Melatonin at bedtime.

For the very young (weighing less than 50lbs): 50mg to 100mg NAC, 100iu Natural Vit. E, 1 cap Krill Oil squeezed & mixed into milk or juice, 1 tablet Kelp & 1 tablet Chlorella daily sprinkled into juice; and 500mcg chewable Melatonin at bedtime.



Q: In view of the long term consequences from nuclear power plant meltdown, what maintenance schedules should be incorporated to best help us all prevent cancer decades down the road?

A: Kelp, Spirulina, Natural Vitamins C & E, high-quality Bone Meal, high-end probiotics (acidophilus), and Melatonin + selenium along with my Getting Started tab above for complete menu planning.

Average adults (weighing +150lbs): 400iu Natural Vit. E plus 1 capsule high potency Probiotics at breakfast; 2 Kelp tablets, 3 Spirulina tablets & 2 Bone Meal tablets with each meal; and 10mg Melatonin at bedtime with 400mcg selenium from selenomethionate. Also, one effervescent Vitamin C drink is to be ingested daily.

Young adults and larger children (weighing 100lbs – 149lbs): 400iu Natural Vit. E plus 1 capsule high potency Probiotics at breakfast; 2 tablets of Kelp, 2 Spirulina tablets & 2 Bone meal tablets with each meal, and 3mg Melatonin at bedtime with 400mcg selenium from selenomethionate. Also, one effervescent Vitamin C drink is to be ingested daily. 

For smaller children (weighing 50lbs – 99lbs): 200iu Natural Vit. E plus 1 capsule high potency Probiotics at breakfast; 1 tablet of Kelp, 1 tablet of Chlorella and one Bone Meal with each meal (all sprinkled into juice as appropriate); and 1mg chewable Melatonin at bedtime with 100mcg – 200mcg selenium from selenomethionate. Also, one effervescent Vitamin C drink is to be consumed daily. Chewable Vitamin C tablets may be substituted IF the brand is LOW in sugar.

For the very young (weighing less than 50lbs): 100iu Natural Vit. E, 1 tablet Kelp & 1 tablet Chlorella plus 1 capsule high potency Probiotics plus 2 Bone Meal capsules opened & sprinkled into juice daily; and 500mcg chewable Melatonin at bedtime with 50mcg selenium from selenomethionate. Lastly, one effervescent Vitamin C drink is attempted to be given daily. Chewable Vitamin C tablets may be substituted IF the brand is LOW in sugar.


Best Long Term Protocol: I (Dr. Apsley) have an extended family, with children ranging in ages from 34 down to 11, and three grandkids ages 4 & 2 years of age. My recommended maintenance schedules to my family are provided below:

Average adults (weighing +150lbs): 250mg NAC + 400iu Natural Vitamin E plus 1 capsule high potency Probiotics & 3 capsules Krill Oil at breakfast; 2 Kelp tablets, 3 Chlorella tablets & 2 Bone Meal tablets with each meal; and 10mg Melatonin at bedtime with 400mcg selenium from selenomethionate. Also, one to three effervescent Vitamin C drinks are to be ingested daily.

Young adults and larger children (weighing 100lbs – 149lbs): 250mg NAC + 400iu Natural Vit. E plus 1 capsule high potency Probiotics & 2 caps Krill Oil at breakfast; 2 tablets of Kelp, 2 Chlorella tablets & 2 Bone meal tablets with each meal, and 3mg Melatonin at bedtime with 400mcg selenium from selenomethionate. Also, one effervescent Vitamin C drink is to be ingested daily.

For smaller children (weighing 50lbs – 99lbs): 200mg NAC + 200iu Natural Vit. E plus 1 capsule high potency Probiotics & 1 cap Krill Oil at breakfast; 1 tablet of Kelp, 1 tablet of Chlorella and one Bone Meal with each meal (all sprinkled into juice as appropriate); and 1mg chewable Melatonin at bedtime with 100mcg – 200mcg selenium from selenomethionate. Also, one effervescent Vitamin C drink is to be consumed daily. Chewable Vitamin C tablets may be substituted IF the brand is LOW in sugar.

For the very young (weighing less than 50lbs): 50mg to 100mg NAC, 100iu Natural Vit. E, 1 tablet Kelp & 1 tablet Chlorella plus 1 capsule high potency Probiotics plus 2 Bone Meal capsules opened & sprinkled into juice daily; and 500mcg chewable Melatonin at bedtime with 50mcg selenium from selenomethionate. Lastly, one effervescent Vitamin C drink is attempted to be given daily. Chewable Vitamin C tablets may be substituted IF the brand is LOW in sugar.

Q: What can I do to antidote my exposures to radiation therapy?

A: Radiation toxicity from any source, including the after effects of radioablative therapy, can be detoxified or lessened and even reversed by the use of colloidal Regeneration Factors or cRFsTM.38, 39, 40 Just be sure to take after radioablative therapy has completed or is placed on hold. cRFsTM derive from select raw foods which contain high amounts of RNA or nucleoproteins as found in organ meats, nuts & seeds, sprouts, food-grade algae and bee pollen.(C)  Folks who tend to get gouty cannot take high amounts of these select foods sources, but usually can handle some intake providing the source is raw and not cooked. For example, food grade algae products, which are raw, can serve such folks well if lots of SAW is being ingested as well. All protocols for diet under this website are high in these factors, i.e., SAW plus RNA-cRFsTM.


For an average size adult, the foremost factors one should focus on to reduce the toxic after affects from radioablative therapy are:

A. NAC (as above);6

B. Chlorophyll (found in sea vegetables, and richest in Chlorella or Spirulina – 5 per meal for adults);3, 4

C. cRFsTM –  As found in Chlorella, Spirulina and organic organ meats such as liver, spleen and thymus in freeze-dried format. When taking capsules of organic

    glandulars without algae products as above, up to a combined 12 to 18 capsules per day for an average adult is optimal. When taking with algae products

    according to the above dosages, cut this amount in 1/2);1, 2

D. Krill oil or Vitamin E Complex (including delta and gamma tocopherols and tocotrienols taken as above). Krill oil is 300 times more powerful than Vitamin E

    and 34 times more powerful than CoQ10 for reducing free-radical damage, mainly to cellular lipid membranes in the body;7, 21, 27, 28

E. Lipoic acid as R-alpha lipoic acid (100mg three times daily for adults, 100mg daily for young adults and children, 25mg – 50mg daily for the very young – when

    no toxic metal particles are involved, just gamma rays);7

F. Melatonin (5mg to 20 mg at bedtime for adults, 1mg to 3mg for children and young adults respectively);5

G. Reduced CoQ10 or Ubiquinol (100mg daily for adults, 50mg per day for smaller adults and children, 25mg daily for the very young);7, 21

H. Vitamin C and Quercetin (1,000mg three times daily and 100mg three times daily respectively);7, 21

I. Organic Selenium as selenomethionate or when grown into kelp or other foods (400mcg – 600mcg daily for adults, 200mcg – 400mcg daily for young adults,    200mcg daily for children, 50mcg – 100mcg daily for the very young).21

If any confusion remains, your holistic trained doctor can adjust the above dosages down or up according to your weight.

Strongly suggested additions to the diet:

All manner and selections of sea vegetables (seaweed dishes) are the first choice.9, 10 Additionally, two herbal medicines which are very potent antidotes to high radiation exposure are: high-quality Aloe vera juice 20 (for any radiation burns, internal or external), and Noni juice 13 (Morinda citrifolia). Also, eliminate all refined sugar from the diet. Lastly, emerging evidence suggests that one species of structured water (Fullerene-C60) may be able to protect normal cells from harmful effects of radiation therapy while simultaneously enhancing results of radiation therapy.8 More studies are needed to confirm this theory.

 In the unlikely event of an emergency concerning acute radiation poisoning, baking soda may be used when directly exposed to high levels of radioactive nuclei (americium, cesium, plutonium, strontium, uranium, etc…):

 Under a doctor’s supervision, baking soda is the tool of choice to remove these toxic metals from the body quickly. Up to 7 teaspoons daily for adults may be used short-term (over 6 consecutive days) to accomplish thorough decontamination, with route & rate of adminstration carefully considered by the physician. Now, couple this protocol to the lipid membrane protocol listed above with added probiotic supplementation and as appropriate use Aloe and Noni juice with a strict diet of no refined carbohydrates, and plenty of SAW.7, 12, 14, 15, 21, 23, 25 Where run-a-way inflammation has taken hold, quadruple NAC intake & follow the natural aspirin, omega 3 & 6 oil protocols with cherry concentrate, curcumin and boswellia (see:

Alternatively, intravenous administration of 5% baking soda (in 500cc), glutathione (2gm), reduced CoQ10 (300mg), sodium ascorbate (10gm), Omega 3 oils (2gm), NAC (5gm) and selenium (2mg) can be administered daily over the course of a week or two, with good success where patient is incapable of ingesting oral supplements. Patient should sip Aloe and Noni juice ad lib.

 To those who may dissent from the above science:

Currently there are a group of esteemed physicists who are suggesting that low levels of radiation from any source, can be beneficial. They are citing some very compelling evidence which suggests that cancer rates can actually go down, not up, with long term, low level exposures to toxic radioactive nuclei such as cobalt or cesium. They also link this kind of radiation to the principles of “hormesis” something of which Eclectic Physicians are quite familiar with. And finally, they cite mineral rich hot springs which are known to emit low level radiation from a naturally occurring radioisotope called radon.30 In fact, the hot springs in Ikaria, Greece and select hot springs in Misasa Hot Springs District of Japan are famous for healings reportedly arising from proper use of these therapeutic waters.

 Eclectic physicians often specialize in BioEnergetics, the Fourth Pillar to The Regeneration Effect. And what these experts will tell you is that select low levels of radiation that disrupt cell membranes will trigger attempts in tissues to regenerate the damage, which is termed autophagy. Just enough autophagy with just enough antioxidants and other essential factors like oxygen and you are, “In like Flint. But too much autophagy with too little antioxidants or under conditions of hypoxia, and you run into cancer, premature cellular death or autoimmune disease. Therefore, only looking at cancer death rates without the other diseases that can substitute in cancer’s place, is misleading to say the least when examining radiation hormesis.

 For example, since WWII, over 555,000 TRILLION picocuries of radiation have been released into the Earth’s biosphere with a half life exceeding 10,000 years of toxicity.18 This helps to explain the dramatic rise, for example, of Cancer, but also Hashimoto’s Disease, Hypothyroidism Type 2, Diabetes Type 2, Colitis, Crohn’s Disease, Lupus, Scleroderma, ALS, MS, R.A., Osteoporosis, Fibromyalgia, Chronic Fatigue Syndrome, Universal Allergic Reactors, Alzheimer’s Disease, infertility, Autism, Low Birth Weight, the rise in Birth Defects, Infant Failure to Thrive Syndromes (i.e., SIDS),  etc… These emerging studies so far do nothing to track these substitutes for cancer which may arise from chronic, low level radiation poisoning. For further confirmation of the above, according to Gabriel Cousens, MD, in his book, Conscious Eating (regarding the Chernobyl incident), reports:41 

“(A top expert, Dr. Ernest J. Sternglass of the University of Pittsburgh) presented at the First Global Radiation Victims Conference in New York in September 1987, impressively conveyed the seriousness of the radiation problem. The infant mortality rate following the arrival of the Chernobyl fallout in early May of 1986 showed a 54% increase in June 1986 in the Pacific region of the United States. Washington State had the highest rate in the region with a 245% increase in deaths per thousand live births. California was next highest with a 48% increase in infant mortality as compared to June of the year before. These high rates lasted for July and August. Massachusetts led the nation in post-Chernobyl increase of infant mortality rate with an increase of 900% per thousand live births! Massachusetts also had a decline of 70% in newborns. The rate of live births also decreased throughout the country in response to the Chernobyl fallout. The US fertility rate decreased 8.3% in July and August to the lowest level ever observed in United States history.

So the above illustrates what happens when everyone is looking in the wrong direction, even if for the right reason. Therefore in my book, there is no such thing as safe, low level exposures to “man-made” sources of radiation. Indeed, any short term benefits will be offset by untoward effects in the future, even if we are too ignorant at present to catch it in advance. Without the proper reference points to link back such differing kinds of data, confusion will prevail when trying to gauge radiation morbidity and mortality statistics.  So, what are the proper reference points that will tell us here and now if low levels of any kind of radiation are potentially beneficial to human tissues? These reference points derive from those discovered by Gilbert N. Ling, and two other experts in BioEnergetics, relating to seven factors:31, 32, 33


  • Does the radiation improve aerobic metabolism of the cell or tissue or lead to healthy cell or tissue regeneration which displays optimal aerobic


  • Does the radiation improve the pH profile of the external and internal milieu of the cell or tissue or lead to healthy cell or tissue regeneration which

           displays optimal pH?

  • Does the radiation improve the structured water (polarized multilayers of water or PM water) within the cell or lead to healthy cell or tissue regeneration which display optimal PM water?
  • Does the radiation improve or harm the normal spectrum of mineral elements in the cell and their proper locations or lead to healthy cell or tissue regeneration which displays optimal mineral reserves?
  • Does the radiation improve or harm the special proline-rich-peptides within cells that are the scafolding to the PM water in the cell or lead to healthy cell or tissue regeneration which displays optimal proline-rich-peptide reserves?
  • Is the ‘Phase Angle’ of the body improved by the radiation exposure or lead to healthy cell or tissue regeneration which displays optimal Phase Angle?
  • And finally, is the negative millivolts of the cell and tissues improved from the radiation exposure or lead to healthy cell or tissue regeneration which displays optimal millivolts?


Physicists who doubt that man-made low level radiation cause harm must verify their opinion by addressing the above questions. Until they do, they stand on inadequately circumspect data points. 


For a comprehensive, well-documented report on U.S. government official misinformation campaigns and outright propaganda policies regarding radiation threats to the American public,




Always restore the body’s own self-healing system first. Then the “footing” of all other natural healing tools employed will have the advantage of driving forward from the high-ground. cRFsTM are key to this strategy when dealing with all radiation toxicity situations, as is abundant SAW and plenty of fresh air. With so much to gain, and so little to lose, why not at least consider a sound maintenance schedule for you and your family?




(A). Abram Petkau, PhD, is a former nuclear physics researcher assigned to Atomic Energy of Canada, Whiteshell Nuclear Research Establishment in Pinowa, Manitoba during the early 1970’s.

        Also see:

(B). For a complete review on low-level radiation dangers and cover-ups, please see: Gould JM, Sternglass EJ, Mangano JJ, and McDonnell W. The Enemy Within. Four Walls Eight Windows, New York, NY 


(C). See upcoming eBook The Regeneration Effect, Volume 1, for complete documentation covering this subject and the items listed above. See:



1. Lourau, M. and Lartigue, O., Experientia, 1950;6:25.

2. Duplan, Influence of Dietary Regimen on Radiosensitivity of the Guinea Pig, Compt. Rend. Acad. Sc., 1953;236:424.

3. Colloway S. Reduction of X-Radiation Mortality by Cabbage and Broccoli, Proc. Soc. Exptl. Bio. Med., 1959;100:405.

4. Colloway S, et al. Quartermaster Food and Container Institute for the Armed Forces Report, N.R., 1961:12-61.

5. Vijayalaxmi et al. Melatonin as a radioprotective agent: a review. Int J Radiat Oncol Biol Phys. 2004 Jul1;59(3):639-53. Also See first abstract below.

6. Chung SW, et al. Molecular delineation of gamma-Ray-Induced NF-kappaB activation and pro-inflammatory genes in SMP30 knockout mice. Radiat Res. 2010 May;173(5):629-34.

7. Brown SL, et al. Antioxidant diet supplementation starting 24 hours after exposure reduces radiation lethality. Radiat Res. 2010 Apr;173(4):462-8.

8. Kateb B, et al. Nanoplatforms for constructing new approaches to cancer treatment, imaging, and drug delivery: What should be the policy? Neuroimage, 2010;105.

9. Maruyama H, Yamamoto I. Suppression of 125I-uptake in mouse thyroid by seaweed feeding: possible preventative effect of dietary seaweed on internal radiation injury of the thyroid by radioactive iodine.

    Kitasato Arch Exp Med. 1992 Dec;65(4):209-16.

10. Gong YF, et al. Suppression of radioactive strontium absorption by sodium alginate in animals and human subjects. Biomed Environ Sci. 1991 Sep;4(3):273-82.

11. Weiss JF, Landauer MR. History and development of radiation-protective agents. Int J Radiat Biol. 2009 Jul;85(7):539-73.

12. Apostoaei AI. Absorption of strontium from the gastrointestinal tract into plasma in healthy human adults. Health Phys. 2002 Jul;83(1):56-65.

13. Qiao ZS, Wu H, Su ZW. [Comparison with the pharmacological actions of Morinda officinalis, Damnacanthus officinarum and Schisandra propinqua]. Zhong Xi Yi Jie He Za Zhi. 1991 Jul;11(7):415-7, 390.

14. Henge-Napoli MH, et al. Efficacy of 3,4,3-LIHOPO for reducing the retention of uranium in rat after acute administration. Int J Radiat Biol. 1995 Oct;68(4):389-93.

15. Fatome M. [Management of accidental internal exposure]. J Radiol. 1994 Nov;75(11):571-5. And see:

16. Chaitow L, Trenev N. Probiotics. Thorsons, Hammersmith, London, 1990;pp. 15, 118-20 & 144.

17. Blom H, Mortvedt T. Anti-microbial substances produced by food associated micro-organisms. Biochem Soc Trans Food Biotech. 1991;694-98.

18. Null G, et al. What physicians should know about the biological effects of ingested fission products. Townsend Letter for Doctors & Patients. Aug/Sep 1993;(121/122):812.

     See: page 21, second paragraph.

19. Petkau A. Effect of Na-22 on phospholipid membranes. Health Physics 1972 Mar. (See reference above.) Also see:

20. Bakuridze AD, et al. [Radio protective drug production from fresh leaves of Aloe arborescens Mill]. Georgian Med News. 2009 Jun;(171):80-3.

21. Wambi CO, et al. Protective effects of dietary antioxidants on proton total-body irradiation-mediated hematopoietic cell and animal survival. Radiat Res. 2009 Aug;172(2):175-86.

22. Graeub R. The Petkau Effect, 2nd edition, Four Walls Eight Windows, New York, NY (1994).

23. Bellés M, Melatonin reduces uranium-induced nephrotoxicity in rats. J Pineal Res. 2007 Aug;43(1):87-95.

24. Aposhian HV, et al. Vitamin C, glutathione, or lipoic acid did not decrease brain or kidney mercury in rats exposed to mercury vapor. Toxicol Clin Toxicol, 2003;41(4):339-47.

25. Reiter RJ, et al. Mechanisms for the Protective Actions of Melatonin in the Central Nervous System. Annals of the New York Academy of Sciences 2001;939:200-215.

26. Hultberg G, Andersson A, Isaksson A. Lipoic acid increases glutathione production and enhances the effect of mercury in human cell lines. Toxicology, 2002 Jun14;175(1-3):103-10.

27. See:

28. Kidd PM. Omega-3 DHA and EPA for cognition, behavior, and mood: clinical findings and structural-functional synergies with cell membrane phospholipids. Altern Med Rev. 2007 Sep;12(3):207-27.


29. Apsley J. The Regeneration Effect: A Professional Treatise on Self Healing, Volume 2, Genesis Communications, LLC, Northport, AL, 1996;p.75. ISBN: 0-945704-02-X.

30. See:

31. Ling GN. Life at the Cell and Below – Cell Level: A Hidden History of a Fundamental Revolution in Biology. Pacific Press, NY. 2001.

32. Tennant J. Healing is Voltage: The Key to Pain Control and Chronic Disease, Tennant Institute for Integrative Medicine and Pain Control, Irving, TX. 972-580-1156.


33. The Phase Angle is the angle between resistance and impedence, and is measured in degrees. See:

34. Rotkovska D, et al. The radioprotective effects of aqueous extract from Chlorococcal freshwater algae (Chlorella kessieri) in mice and rats. Strahlenther Onkol. 1989;165:813.

35. Vacek A, et al. Radioprotection of hemopoisesis conferred by aqueous extract from Chlorococcal algae (Ivastimul) administered to mice before radiation. Exp Heamotol. 1990;18:237-73.

36. Qishen P, et al. Radioprotective effect of extract from Spirulina platensis in mouse bone marrow cells studied by using the micronucleus test. Toxicology Let.  1989;48:165.

37. Horikoshi T, et al. Uptake of uranium by various cell fractions of Chlorella regularis. Radioisotopes,  1979 Aug;28(8):485-87.

38. Maisin J, et al. Yeast ribonucleic acid and its nucleotides as recovery factors in rats receiving an acute whole-body dose of X-rays. Nature, 1960;186:487-95.

39. Ebel JP, et al. Study on the therapeutic effect on irradiated mice of substances contained in RNA preparations. Int J Radiat Biol. 1969;16:201-09.

40. Wagner R, Silverman EC. Chemical protection against X-radiation in the Guinea-Pig. I. Radioprective action of RNA and ATP. Int J Radiat Biol. 1967;12:101-12.

41. Cousens G. Conscious Eating. North Atlantic Books; 2 edition (April 11, 2000). ISBN13: 9781556432859



 Seaweeds & Kelp:

Kitasato Arch Exp Med. 1992 Dec;65(4):209-16.

Suppression of 125I-uptake in mouse thyroid by seaweed feeding: possible preventative effect of dietary seaweed on internal radiation injury of the thyroid by radioactive iodine.

Maruyama H, Yamamoto I.

Department of Pathology, Kitasato University School of Hygienic Sciences, Kanagawa, Japan.


We conducted an animal experiment to determine how dietary seaweeds rich in iodine and dietary fibers suppress radioactive iodine uptake by the thyroid, using mice and four kinds of experimental diets, three with 1% or 2% powdered fronds of the kelp Laminaria religiosa and 2% powdered laver Porphyra yezoensis, and one with cellulose. Iodine content of a hot-water extract of the kelp was 0.530 +/- 0.001%, and its dietary fiber (DF) values were 52.8 +/- 1.2%. Iodine in an extract of the laver was 0.008 +/- 0.001%, and its DF values were 41.4% +/- 0.7%. A statistically significant reduction of 125I uptake by the thyroid, 3 hours after intragastric administration of the radionuclide at a dosage of 18.5 kBq or 185 kBq in 0.3 ml aqueous solution per mouse, was observed in mice previously fed the experimental diets containing 1% and 2% kelp during periods varying from 24 hours to 7 days. The degree of the suppression was observed to depend on the amount of iodine in the diet or in the injected sample, no matter whether organic or inorganic, judging from the results of an additional experiment. Thus, we conclude that previously fed iodine-rich material, especially dietary seaweeds rich in iodine and other minerals, vitamins, and beta-carotene, such as kelps or laver supplemented with inorganic iodine, may be effective in prevention of internal radiation injury of the thyroid. PMID: 1344008

 Biomed Environ Sci. 1991 Sep;4(3):273-82.

Suppression of radioactive strontium absorption by sodium alginate in animals and human subjects.

Gong YF, Huang ZJ, Qiang MY, Lan FX, Bai GA, Mao YX, Ma XP, Zhang FG.

Institute of Radiation Medicine, Beijing, China.


The effect of 23 sodium alginate preparations from different species of algae (Sargassum sp.) and kelp (Laminaria sp.) on reducing the absorption of strontium was studied in detail. A pilot production procedure has been established. Na alginate from S. siliquastrum was proven to be a potent agent for reducing Sr absorption, with high efficiency and virtually no toxicity. It reduced the body burden of strontium 3.3-4.2 fold in rats. Strontium absorption in human subjects was reduced by 78% (+/- 8.9) or completely suppressed the increase of serum Sr at 2 h after ingestion of stable Sr in volunteers and decrease 24 h urine Sr to similar extent. No undesirable effects on gastrointestinal function was observed nor were Ca, Fe, Cu and Zn metabolism changed, both in the animal experiments and in human. It was concluded that alginate preparations derived from Sargassum species are a suitable antidote against radiostrontium absorption on a long-term basis, when added to bread at a 6% level. In cases of emergency, an alginate syrup preparation appears to be more suitable because of its rapid action. PMID: 1764217

  Studies Associated with NAC, Selenium, Vit. C & E, Lipoic acid and CoQ10 supplementation:

 Radiat Res. 2010 Apr;173(4):462-8.

Antioxidant diet supplementation starting 24 hours after exposure reduces radiation lethality.

Brown SL, Kolozsvary A, Liu J, Jenrow KA, Ryu S, Kim JH.

Henry Ford Hospital, Department of Radiation Oncology, Detroit, Michigan 48202, USA.


Antioxidants mitigate radiation-induced lethality when started soon after radiation exposure, a delivery time that may not be practical due to difficulties in distribution and because the oral administration of such agents may require a delay beyond the prodromal stage of the radiation syndrome. We report the unexpected finding that antioxidant supplementation starting 24 h after total-body irradiation resulted in better survival than antioxidant supplementation started soon after the irradiation. The antioxidant dietary supplement was l-selenomethionine, sodium ascorbate, N-acetyl cysteine, alpha-lipoic acid, alpha-tocopherol succinate, and co-enzyme Q10. Total-body irradiation with 8 Gy in the absence of antioxidant supplementation was lethal by day 16. When antioxidant supplementation was started soon after irradiation, four of 14 mice survived. In contrast, 14 of 18 mice receiving antioxidant supplementation starting 24 h after irradiation were alive and well 30 days later. The numbers of spleen colonies and blood cells were higher in mice receiving antioxidant supplementation starting 24 h after irradiation than in mice receiving radiation alone. A diet supplemented with antioxidants administered starting 24 h after total-body irradiation improved bone marrow cell survival and mitigated lethality, with a radiation protection factor of approximately 1.18. PMID: 20334518 

Studies Associated with Calcium Intake (and therefore pertaining to Bone Meal supplementation):

Health Phys. 2004 Jun;86(6):557-64.

Dietary intakes of seven elements of importance in radiological protection by asian population: comparison with ICRP data.

Iyengar GV, Kawamura H, Dang HS, Parr RM, Wang J, Akhter P, Cho SY, Natera E, Miah FK, Dojosubroto J, Nguyen MS.

Nutrition and Health-Related Environmental Studies Section Division of Human Health, International Atomic Energy Agency, Vienna, Austria.


Within the framework of a Coordinated Research Project (CRP) organized by the International Atomic Energy Agency, Vienna, the daily dietary intakes of seven elements by adult populations living in nine Asian countries were estimated. The countries that participated in the study were Bangladesh, China, India, Indonesia, Japan, Pakistan, Philippines, South Korea (Republic of Korea, ROK), and Vietnam and together they represented more than half of the world population. The seven elements studied were calcium, cesium, iodine, potassium, strontium, thorium, and uranium. These elements have chemical and biological similarity to some of the radionuclides abundantly encountered during nuclear power production and therefore data on these elements could provide important information on their biokinetic behavior. Analyses of diet samples for these seven elements were carried out using highly sensitive and reliable analytical techniques. One thousand one hundred and sixty analytical determinations were made on two hundred and twenty samples of typical diets consumed in these countries to estimate the daily intakes of these elements by the adult Asian population. The median daily dietary intakes for the adult Asian population were found to be 0.45 g calcium, 7 microg cesium, 90 microg iodine, 1.75 g potassium, 1.65 mg strontium, 1 microg thorium, and 1 microg uranium. When compared with the intakes proposed for ICRP Reference Man by International Commission for Radiological Protection, these intakes were lower by factors of 0.41 for calcium, 0.7 for cesium, 0.45 for iodine, 0.53 for potassium, 0.87 for strontium, 0.33 for thorium, and 0.52 for uranium. The lower daily intakes of calcium, cesium, and iodine by Asian population could be due to significantly lower consumption of milk and milk products, which are rich in these elements. The significantly lower intake of calcium in most of the Asian countries may lead to higher uptake of fission nuclide 90Sr and could result in perhaps higher internal radiation dose. The use of highly sensitive and reliable analytical methods resulted in accurate and lower intake values obtained for thorium and uranium, which suggest that radiation dose from their ingestion at natural background levels is likely to be lower than what may be concluded from ICRP data. PMID: 15167119

Health Phys. 2002 Jul;83(1):56-65.

Absorption of strontium from the gastrointestinal tract into plasma in healthy human adults.

Apostoaei AI.

SENES, Oak Ridge, Inc, TN 37830, USA.


The radioactive isotopes of strontium have always been a major concern in radiation protection. Currently, radiostrontium is of interest for evaluation of the health effects of the Chernobyl accident and for epidemiological studies in populations exposed to releases from the Mayak nuclear facilities in Russia. Ingestion is one of the most important exposure pathways involving radioactive strontium. The main sources of published data on the fraction of the ingested strontium that is transferred to plasma (f1) are summarized. For some of these studies, the original data had to be reanalyzed and a new iterative method to account for the elimination in feces of strontium of endogenous origin (i.e., that was absorbed to blood and has already been returned into feces) was employed. Data indicate no significant dependence of the absorbed fraction on sex or age at exposure within the adult group, but absorption of (radioactive) strontium is reduced if the intake of stable calcium is very high and is enhanced if the intake of calcium is very low. The probability distribution function of f1 values is well represented by a lognormal curve with a geometric mean of 22.3% and a geometric standard deviation of 1.44 (95% confidence interval 10.9% to 45.6%, or about a factor of 2 around the geometric mean). This distribution can be considered representative for the variability of the f1 values in a population of healthy adults. PMID: 12075684


 Int J Radiat Oncol Biol Phys. 2004 Jul 1;59(3):639-53.

Melatonin as a radioprotective agent: a review.

Vijayalaxmi, Reiter RJ, Tan DX, Herman TS, Thomas CR Jr.

Department of Radiation Oncology, The University of Texas Health Science Center, San Antonio, 78229, USA.


Melatonin (N-acetyl-5-methoxytryptamine), the chief secretory product of the pineal gland in the brain, is well known for its functional versatility. In hundreds of investigations, melatonin has been documented as a direct free radical scavenger and an indirect antioxidant, as well as an important immunomodulatory agent. The radical scavenging ability of melatonin is believed to work via electron donation to detoxify a variety of reactive oxygen and nitrogen species, including the highly toxic hydroxyl radical. It has long been recognized that the damaging effects of ionizing radiation are brought about by both direct and indirect mechanisms. The direct action produces disruption of sensitive molecules in the cells, whereas the indirect effects ( approximately 70%) result from its interaction with water molecules, which results in the production of highly reactive free radicals such as *OH, *H, and e(aq)- and their subsequent action on subcellular structures. The hydroxyl radical scavenging ability of melatonin was used as a rationale to determine its radioprotective efficiency. Indeed, the results from many in vitro and in vivo investigations have confirmed that melatonin protects mammalian cells from the toxic effects of ionizing radiation. Furthermore, several clinical reports indicate that melatonin administration, either alone or in combination with traditional radiotherapy, results in a favorable efficacy:toxicity ratio during the treatment of human cancers. This article reviews the literature from laboratory investigations that document the ability of melatonin to scavenge a variety of free radicals (including the hydroxyl radical induced by ionizing radiation) and summarizes the evidence that should be used to design larger translational research-based clinical trials using melatonin as a radioprotector and also in cancer radiotherapy. The potential use of melatonin for protecting individuals from radiation terrorism is also considered. PMID: 15183467 [PubMed – indexed for MEDLINE]


 Zhong Xi Yi Jie He Za Zhi. 1991 Jul;11(7):415-7, 390.

[Comparison with the pharmacological actions of Morinda officinalis, Damnacanthus officinarum and Schisandra propinqua].

[Article in Chinese]

Qiao ZS, Wu H, Su ZW.

College of Pharmacy, Second Military Medical University, Shanghai.


There are three kinds of plants, Morinda officinalis (1), Damnacanthus officinarum (2), and Schisandra propinqua (3) whose roots have been used since the ancient time. In this paper, some of their pharmacological actions that are related to tonifying and invigorating Yang were examined and compared. The body weight, the thymus weight, the amount of leukocyte in the blood, and the continuing swimming times of the young mice could be increased with the oral administration of the water extractions of (1) and (2) (P less than 0.05-0.001). The Rt of M-receptor in the brains of the hypothyroidism mice were decreased after administration of the water extracts of (1) and (2) (P less than 0.05). (1) could also increased the amount of leukocyte in the blood of leukocytopenia mice caused by radiation of gamma-ray (P less than 0.01). (3) has not shown the obvious effects (P greater than 0.05). The results indicate that (1) and (2) have the ability of anti-fatigue, improving the immunological action of the young mice, and reducing the excitability of the para-sympathetic nervous system of the hypothyroidism mice through decreasing the Rt of M-receptor in their brains. All of them did not show acute toxicity, inducing mutation, and sexual hormone like actions. PMID: 1914038


Georgian Med News. 2009 Jun;(171):80-3.

[Radio protective drug production from fresh leaves of Aloe arborescens Mill].

[Article in Russian]

Bakuridze AD, Nikolaev SM, Berashvili DT, Bakuridze KA, Tsomaia IV.


Nowadays, phytogenous drugs are wildly used as radio protective substances. The aim of the research was to study radio protective characteristics of aloe juice fraction and to develop new technology for radio protective drug production. Technological scheme for getting the drug in two stages. The first stage – extraction of juice from fresh leaves; the second stage – extracting bagasse have been developed and optimal environment for bagasse extraction are defined: Infusion of bagasse with 96 % ethyl spirit (1:1) during 30 minutes, continuation of extracting with water on correlation to raw materials 10:1 at temperature of 70 degrees C during 30 minutes. For the basis of the first series of balanced loading there are taken the optimal parameters of extracting process, on the basis of which in its turn was developed technological scheme of getting dry extract of aloe. Dry extract is a fine-dispersed reddish-yellow (brownish-yellow) powder, which can be easily dissolved in warm (40-60 degrees C) water. Pharmacological researches were conducted in the Institute of General and Experimental Biology, Siberian Branch, Russian. Academy of Sciences. The remarkable radio protective effect of the drug was revealed. PMID: 19578223

 Structured Water: 

J Photochem Photobiol B. 2010 Feb 12;98(2):144-51. Epub 2009 Dec 5.

Defensive effects of fullerene-C60/liposome complex against UVA-induced intracellular reactive oxygen species generation and cell death in human skin keratinocytes HaCaT, associated with intracellular uptake and extracellular excretion of fullerene-C60.

Kato S, Kikuchi R, Aoshima H, Saitoh Y, Miwa N.

Laboratory of Cell-Death Control BioTechnology, Faculty of Life and Environmental Sciences, Prefectural University of Hiroshima, 562, Nanatsuka, Shobara, Hiroshima 727-0023, Japan.


The UVA-irradiation of 10 J/cm(2) on HaCaT keratinocytes increased 59.1% of the intracellular reactive oxygen species (ROS) by NBT assay and the cell viability decreased to 31.5% by WST-1 assay, comparing to the non-irradiated control. In the presence of fullerene-C60 (C60) incorporated in phospholipid membrane vehicle (LiposomeFullerene: Lpsm-Flln) of 250-500 ppm, they were restored to -9.1% to +2.3% of the ROS and 83.0-84.8% of the cell viability, but scarcely restored by the liposome without C60 (Lpsm). In HaCaT cells administered with Lpsm-Flln (150 ppm), C60 was ingested at the intracellular concentrations of 1.4-21.9 ppm for 4-24 h, and, intracellular C60 was excreted by 80% at 4h after rinsing-out, and decreased to 2-10% after 24-48 h. C60 was predominantly distributed around the outside of nuclear membrane without deterioration of intact cell morphology according to fluorescent immunostain. Thus Lpsm-Flln is found to be an effective antioxidant that could preserve HaCaT keratinocytes against UVA-induced cellular injury. Lpsm-Flln has a potential to serve as a cosmetic material for skin protection against UVA. Copyright 2009 Elsevier B.V. All rights reserved.

PMID: 20060738 [PubMed – in process]

 J Radiat Res (Tokyo). 2008 May;49(3):321-7. Epub 2008 Feb 16.

Fullerenol C60(OH)24 effects on antioxidative enzymes activity in irradiated human erythroleukemia cell line.

Bogdanović V, Stankov K, Icević I, Zikic D, Nikolić A, Solajić S, Djordjević A, Bogdanović G.

Institute of Oncology, Department of Experimental Oncology, Sremska, Kamenica, Serbia.


Radiotherapy-induced toxicity is a major dose-limiting factor in anti-cancer treatment. Ionizing radiation leads to the formation of reactive oxygen and nitrogen species (ROS/RNS) that are associated with radiation-induced cell death. Investigations of biological effects of fullerenol have provided evidence for its ROS/RNS scavenger properties in vitro and radioprotective efficiency in vivo. Therefore we were interested to evaluate its radioprotective properties in vitro in the human erythroleukemia cell line. Pre-treatment of irradiated cells by fullerenol exerted statistically significant effects on cell numbers and the response of antioxidative enzymes to X-ray irradiation-induced oxidative stress in cells. Our study provides evidence that the pre-treatment with fullerenol enhanced the enzymatic activity of superoxide dismutase and glutathione peroxidase in irradiated K562 cells. PMID: 18285660 [PubMed – indexed for MEDLINE]

 Sodium Bicarbonate (Baking Soda):

 JEM. 1917;25(5):693-719.

A study of the acidosis, blood urea, and plasma chlorides in uranium nephritis in the dog, and of the protective action of sodium bicarbonate. 

Goto K.


Methods: The presence of an acidosis in dogs with experimental uranium nephritis is demonstrable by the Van Slyke-Stillman-Cullen method and that of Marriott. It is detected more readily by the former method. This acidosis is associated with increase in the blood urea and plasma chlorides and with the appearance of albumin and casts in the urine. Results: The oral administration of sodium bicarbonate diminishes the acidosis, the increase in plasma chlorides, the amount of albumin and casts in the urine, and, to a lesser degree, the increase in the blood urea following the administration of uranium. It also diminishes the severity of the changes produced by uranium in the kidneys. Conclusions: The oral administration of sodium bicarbonate to normal dogs raises the carbon dioxide content of the plasma as determined by the Van Slyke-Stillman-Cullen method, and reduces the nephrotoxicity associated with uranium exposure in dogs.

 J Radiol. 1994 Nov;75(11):571-5.

[Management of accidental internal exposure].

[Article in French]

Fatome M.


Radionucleides can penetrate into the body via the lung, the digestive tract, wounds and sometimes through healthy skin. Once they have penetrated the body, they can either remain localized at the site of entry or be rapidly metabolized. The risk is late effects. Radioelements must be eliminated as rapidly as possible decreasing the exposure proportionally. The effectiveness of the treatment depends on early institution. Nevertheless, emergency intensive care or surgery may be required. As soon as possible, explorations must be carried out to evaluate the level of contamination (human spectrometry, radiotoxicological examinations) and to start treatment. Modalities include non-specific techniques (lavage, insolubilization, laxatives) and specific techniques such as complexation or isotopic dilution (iodine for iodine, Prussian blue for cesium, DTPA for plutonium, Diamox or sodium bicarbonate for uranium). Surgical cleaning of wounds and burns is an excellent means of decontamination. External contamination is often associated. Further contamination must be prevented immediately. PMID: 7844774



Int J Radiat Biol. 1995 Oct;68(4):389-93.

Efficacy of 3,4,3-LIHOPO for reducing the retention of uranium in rat after acute administration.

Henge-Napoli MH, Archimbaud M, Ansoborlo E, Metivier H, Gourmelon P.

Institute de Protection et de Sûreté Nucléaire, IPSN, Fontenay aux Roses, France.


Decorporation therapy is the only known effective method of reducing the radiation dose to persons following accidental internal contamination with transportable radionuclides. Deposits of actinides in bone should be minimized because development of osteosarcoma appears to be related to internal exposure. In contrast with other actinides, such as plutonium or americium where chelating agent treatment is efficient, the therapeuric approaches used for cases of uranium contamination are widely ineffective. This is the first report on in vivo efficacy of a chelating agent, a siderophore analogue code named 3,4,3-LIHOPO, after systematic exposure to natural uranium in the rat. Using the classical antidotal therapy (sodium bicarbonate) for comparison, this ligand has been investigated for its ability to remove uranium from rats after intravenous or intramuscular injection as nitrate. Following an immediate single intramuscular or intravenous injection of 3,4,3-LIHOPO (30 urinary excretion of uranium was greatly enhanced with a corresponding reduction 24 h later in kidney and bone uranium content (to about 20 and 50% of the control rat respectively). Under identical experimental conditions, sodium bicarbonate (640 reduced the uranium content in kidney in kidney and bone only to about 90 and 70% of controls respectively, and there was less enhancement of uranium excretion. However, when treatment was delayed by 30 min and administered intraperitoneally, there was no marked difference in retention and excretion of uranium between the two compounds. As this ligand showed no apparent irreversible toxicity at effective dosages, it is concluded that the administration of the 3,4,3-LIHOPO chelating agent represents potentially a most significant advance for prompt treatment of uranium contamination, while a more detailed investigation is necessary on the possible advantage when treatment delayed. PMID: 7594963 


Ann Pharm Fr. 1999 Sep;57(5):397-400.

[Reduction of renal uranium uptake by acetazolamide: the importance of urinary elimination of bicarbonate].

[Article in French]

Destombes C, Laroche P, Cazoulat A, Gérasimo P.

Centre d’Etudes du Bouchet, Clamart.


Acetazolamide was compared with bicarbonate for the treatment of contamination with uranium. Uranium was injected peritoneally in rats, and its distribution was investigated. Acetazolamide was three times more efficient than bicarbonate in reducing the renal content of uranium. On the other hand, it had no effect on hepatic or skeletal content. In this study, renal physiology provides the basis for understanding the mode of action of acetazolamide and bicarbonate. In this context, it is of interest to determine the alkalinity of the urine, with the aim of knowing whether bicarbonate is present to mobilize uranium. PMID: 10520511

J Cell Physiol. 2005 Dec;205(3):428-36.

Alkaline induces metallothionein gene expression and potentiates cell proliferation in Chinese hamster ovary cells.

Lin KA, Chen JH, Lee DF, Lin LY.

Institute of Radiation Biology and Department of Life Science, National Tsing Hua University, Hsinchu, Taiwan.


Metallothionein (MT) gene expression is increased in cadmium resistant Chinese hamster ovary cells (CHO Cd(R)) upon medium (regular or serum-free) change during culturing. Among the major components of the medium, NaHCO3 was found to be able to induce MT gene expression in a dose- and time-dependent manner. The same effect was observed with other alkaline solutions, such as HEPES and NaOH. Using MT promoter-luciferase reporter gene constructs, we found that the presence of metal response elements (MREs) in the promoter region is necessary for NaHCO3-induced MT gene transcription. This finding is further supported by the observation that the binding activity between the metal-responsive transcription factor 1 (MTF-1) and the MRE were increased after NaHCO3 treatment. Following NaHCO3 treatment, an increase in cell proliferation was observed in CdR cells but not in the parental CHO K1 cells that do not express MT transcripts due to MT gene methylation. Using synchronized cells, an increase in cell proliferation was observed 9 h after NaHCO3 addition. Notably, proliferation of CHO K1 cells was increased when transfected with an MT gene. The effect of MT on cell growth was affirmed by treating CHO K1 cells with 5-azacytidine (Aza) to demethylate the MT gene. Proliferation increased in Aza-treated CHO K1 cells after NaHCO3 treatment. These results demonstrate that NaHCO3 stimulates MT gene expression and causes an enhancement of cell proliferation in CHO cells. (c) 2005 Wiley-Liss, Inc. PMID: 15965962

Toxicol Appl Pharmacol. 2006 Jun 15;213(3):245-55. Epub 2006 Jan 4.

Effects of 12 metal ions on iron regulatory protein 1 (IRP-1) and hypoxia-inducible factor-1 alpha (HIF-1alpha) and HIF-regulated genes.

Li Q, Chen H, Huang X, Costa M.

Nelson Institute of Environmental Medicine, New York University, School of Medicine, 57 Old Forge Road, NY 10987, USA.


Several metal ions that are carcinogenic affect cellular iron homeostasis by competing with iron transporters or iron-regulated enzymes. Some metal ions can mimic a hypoxia response in cells under normal oxygen tension, and induce expression of HIF-1alpha-regulated genes. This study investigated whether 12 metal ions altered iron homeostasis in human lung carcinoma A549 cells as measured by an activation of IRP-1 and ferritin level. We also studied hypoxia signaling by measuring HIF-1alpha protein levels, hypoxia response element (HRE)-driven luciferase reporter activity, and Cap43 protein level (an HIF-1alpha responsive gene). Our results show the following: (i) Ni(II), Co(II), V(V), Mn(II), and to a lesser extent As(III) and Cu(II) activated the binding of IRP-1 to IRE after 24 h, while the other metal ions had no effect; (ii) 10 of 12 metal ions induced HIF-1alpha protein but to strikingly different degrees. Two of these metal ions, Al(III) and Cd(II), did not induce HIF-1alpha protein; however, as indicated below, only Ni(II), Co (II), and to lesser extent Mn(II) and V(V) activated HIF-1alpha-dependent transcription. The combined effects of both [Ni(II) + As(III)] and [Ni(II) + Cr(VI)] on HIF-1alpha protein were synergistic; (iii) Addition of Fe(II) with Ni(II), Co(II), and Cr(VI) attenuated the induction of HIF-1alpha after 4 h treatment; (iv) Ni(II), Co(II), and Mn(II) significantly decrease ferritin level after 24 h exposure; (v) Ni(II), Co(II), V(V), and Mn(II) activated HRE reporter gene after 20 h treatment; (vi) Ni(II), Co(II), V(V), and Mn(II) increased the HIF-1-dependent Cap43 protein level after 24 h treatment. In conclusion, only Ni (II), Co (II), and to a lesser extent Mn(II) and V(V) significantly stabilized HIF-1alpha protein, activated IRP, decreased the levels of ferritin, induced the transcription of HIF-dependent reporter, and increased the expression of Cap43 protein levels (HIF-dependent gene). The mechanism for the significant stabilization and elevation of HIF-1alpha protein which drives these other parameters was  previously shown by us and others to involve a loss of cellular Fe as well as inhibition of HIF-1alpha-dependent prolyl hydroxylases which target the binding of VHL ubiquitin ligase and degrade HIF-1alpha. Even though there were small effects of some of the other metals on IRP and HIF-1alpha, downstream effects of HIF-1alpha activation and therefore robust hypoxia signaling were only observed with Ni(II), Co(II), and to much lesser extents with Mn(II) and V(V) in human A549 lung cells. It is of interest that the metal ions that were most effective in activating hypoxia signaling were the ones that were poor inducers of metallothionein protein and also decreased Ferritin levels, since both of these proteins can bind metal ions and protect the cell against toxicity in human lung cells. It is important to study effects of these metals in human lung cells since this represents a major route of human environmental and occupational exposure to these metal ions. PMID: 16386771




Precautionary Measures for Possible Radiation Plume

By The School of Natural Healing


In answer to the many inquiries of what to do about the recent news and reports (New York Times) of a radiation plume reaching the west coast by Friday, here are some things to know and ways in which you can be prepared.

At this point the levels of radiation prospected to be in the plume that would actually fall on estimated California, Oregon, Washington, British Columbia, Alaska, etc. is very minimal and is not to pose a serious risk to health. However, “if” the situation in Japan worsens (ie. the Fukushima reactors have a rod “meltdown” – creating a massive release of radiation into the atmosphere) taking the following precautions/preparations would be our suggestion, especially for those living in the pacific and on the west coast.

1) Take the time to reevaluate your emergency plan and supplies as a family. Make sure your vehicles are not empty of fuel in case you need to evacuate and do not count on phones working in case of actual emergency.

2) If suggestion is and you plan to, stay put and to stay within your home for an allotment of time make sure you have plastic and duct tape to seal your windows and doors as well as the supplies needed in the case of a usual emergency: water, food, etc.

3) If you plan to evacuate, have at least a 72-hour kit ready to go at a moment’s notice. This is good to have no matter what you plans are.

4) Staring now, significantly increase your intake of naturally sourced iodine-containing foods and superfoods: (*estimated amounts of naturally occurring iodine)

    Icelandic Kelp – *8000ppm
    Kelp, Dulse, etc. (Seaweeds) – *5400ppm
    Chlorella – *100ppm
    Spirulina – *70ppm
    Pistacios – *51ppm
    Dark Greens in all there variety
    Colorful vegetables & fresh fruits
    Onions & Garlic

5) The purpose of increasing your intake of iodine-containing foods is to raise the amount of stable iodine in the blood. This will increase the likelihood that the thyroid will absorb the stable iodine instead of the radioactive iodine (iodine-129 and iodine-131) released during a nuclear accident.

6) During the intake of iodine (kelp capsules SNH recommendation: 8-12 capsules a day depending on the size and health of the individual) further precautionary measures would be to take the four Dr. Christopher’s Cleansing Formulas to keep things cleaned out (Lower Bowel Formula, Kidney Formula, Liver & Gall Bladder Formula, and the Blood Stream Formula) and then to add the Heavy Mineral Bugleweed Formula for additional cleansing




Prostate Cancer Spreads to Bones by Overtaking the Home of Blood Stem Cells

Prostate Cancer Spreads to Bones by Overtaking the Home of Blood Stem Cells

ScienceDaily (Mar. 23, 2011) — Like bad neighbors who decide to go wreck another community, prostate and breast cancer usually recur in the bone, according to a new University of Michigan study.

, U-M researchers believe they know why. Prostate cancer cells specifically target and eventually overrun the bone marrow niche, a specialized area for hematopoietic stem cells, which make red and white blood cells, said Russell Taichman, professor at the U-M School of Dentistry and senior author of the study.

Once in the niche, the cancer cells stay dormant and when they become active again years later, that’s when tumors recur in the bone. The implication is that this may give us a window as to how dormancy and recurrence take place.

Taichman and a team of researchers looked in the bone marrow and found cancer cells and hematopoietic stem cells next to one another competing for the same place. The finding is important because it demonstrates that the bone marrow niche plays a central role in bone metastasis — cancers that spread into the bone — giving researchers a new potential drug target.

Drugs could be developed to keep the types of cancers that likely recur in the bone from returning, Taichman said. For example, these drugs could either halt or disrupt how the cancer cells enter or behave in the niche, or keep the cancer cells from out-competing the stem cells.

Cancer cells act a lot like stem cells in that they must reproduce, so the U-M research group hypothesized that prostate cancer cells might travel to the niche during metastasis. One of the jobs of the niche is to keep hematopoietic stem cells from proliferating — which may be the case for cancer cells, as well, the researchers found.

So why does cancer recur? Say a person has a tumor and surgeons cut it out or do radiation, but it recurs in the bone marrow five years later, Taichman said. Those cancer cells had been circulating in the body well before the tumor was discovered, and one place those cancer cells hid is the niche.

“So what have the cancer cells been doing during those five years? Now we have a partial answer — they’ve been sitting in this place whose job it is to keep things from proliferating and growing,” Taichman said.

“Our work also provides an explanation as to why current chemotherapies often fail in that once cancer cells enter the niche, most likely they stop proliferating,” said Yusuke Shiozawa, lead author of the study. “The problem is that most of the drugs we use to try to treat cancer only work on cells that are proliferating.”

Metastases are the most common malignant tumors involving the skeleton, and nearly 70 percent of patients with breast and prostate cancer have bone involvements. Roughly 15 percent to 30 percent of patients with lung, colon, stomach, bladder, uterus, rectum, thyroid or kidney cancer have bone lesions.

Researchers aren’t quite sure how the cancer cells out-compete the stem cells in the niche. However, they do know the stem cells were displaced because when cancer cells were in the niche scientists also found evidence of immature blood stem cells in the blood stream, instead of in the marrow where they were supposed to be, Taichman said.

“Eventually the entire blood system is going to collapse,” he said. “For example, the patient ultimately becomes anemic, gets infections, and has bleeding problems. We really don’t know why people with prostate cancer die. They end up dying from different kinds of complications in part because the marrow is taken over by cancer.”

The next step is to find out how the tumor cell gets into the niche and becomes dormant, and exactly what they do to the stem cells when they are there. Researchers also want to know if other types of cancer cells, such as breast cancer, also go to the niche.

The study appears online in the Journal of Clinical Investigation.

Co-authors are: Elizabeth Pedersen, Aaron Havens, Younghun Jung, Anjali Mishra, Jeena Joseph, Jin Koo Kim, Anne Ziegler, Michael Pienta, Jingcheng Wang, Junhui Song and Paul Krebsbach of the U-M School of Dentistry; Lalit Patel, Chi Ying, Robert Loberg and Kenneth Pienta of the departments of Urology and Internal Medicine at the U-M Medical School.