Round UP GENETIC DAMAGE
Dear Secretary Vilsack:
A team of senior plant and animal scientists have recently brought to my attention the discovery of an electron microscopic pathogen that appears to significantly impact the health of plants, animals, and probably human beings. Based on a review of the data, it is widespread, very serious, and is in much higher concentrations in Roundup Ready (RR) soybeans and corn – suggesting a link with the RR gene or more likely the presence of Roundup. This organism appears NEW to science!- This is highly sensitive information that could result in a collapse of US soy and corn export markets and significant disruption of domestic food and feed supplies. On the other hand, this new organism may already be responsible for significant harm (see below). My colleagues and I are therefore moving our investigation forward with speed and discretion, and seek assistance from the USDA and other entities to identify the pathogen’s source, prevalence, implications, and remedies.
We are informing the USDA of our findings at this early stage, specifically due to your pending decision regarding approval of RR alfalfa. Naturally, if either the RR gene or Roundup itself is a promoter or co-factor of this pathogen, then such approval could be a calamity. Based on the current evidence, the only reasonable action at this time would be to delay deregulation at least until sufficient data has exonerated the RR system, if it does.
For the past 40 years, I have been a scientist in the professional and military agencies that evaluate and prepare for natural and manmade biological threats, including germ warfare and disease outbreaks. Based on this experience, I believe the threat we are facing from this pathogen is unique and of a high risk status. In layman’s terms, it should be treated as an emergency.
A diverse set of researchers working on this problem have contributed various pieces of the puzzle, which together presents the following disturbing scenario:
Unique Physical Properties
This previously unknown organism is only visible under an electron microscope (36,000X), with an approximate size range equal to a medium size virus. It is able to reproduce and appears to be a micro-fungal-like organism. If so, it would be the first such micro-fungus ever identified. There is strong evidence that this infectious agent promotes diseases of both plants and mammals, which is very rare.
Pathogen Location and Concentration
It is found in high concentrations in Roundup Ready soybean meal and corn, distillers meal, fermentation feed products, pig stomach contents, and pig and cattle placentas.
Linked with Outbreaks of Plant Disease
The organism is prolific in plants infected with two pervasive diseases that are driving down yields and farmer income – sudden death syndrome (SDS) in soy, and Goss’ wilt in corn. The pathogen is also found in the fungal causative agent of SDS (Fusarium solani fsp glycines).
Implicated in Animal Reproductive Failure
Laboratory tests have confirmed the presence of this organism in a wide variety of livestock that have experienced spontaneous abortions and infertility. Preliminary results from ongoing research have also been able to reproduce abortions in a clinical setting.–The pathogen may explain the escalating frequency of infertility and spontaneous abortions over the past few years in US cattle, dairy, swine, and horse operations. These include recent reports of infertility rates in dairy heifers of over 20%, and spontaneous abortions in cattle as high as 45%- For example, 450 of 1,000 pregnant heifers fed wheatlege experienced spontaneous abortions. Over the same period, another 1,000 heifers from the same herd that were raised on hay had no abortions. High concentrations of the pathogen were confirmed on the wheatlege, which likely had been under weed management using glyphosate.
In summary, because of the high titer of this new animal pathogen in Roundup Ready crops, and its association with plant and animal diseases that are reaching epidemic proportions, we request USDA’s participation in a multi-agency investigation, and an immediate moratorium on the deregulation of RR crops until the causal/predisposing relationship with glyphosate and/or RR plants can be ruled out as a threat to crop and animal production and human health.
It is urgent to examine whether the side-effects of glyphosate use may have facilitated the growth of this pathogen, or allowed it to cause greater harm to weakened plant and animal hosts. It is well-documented thatglyphosate promotes soil pathogens and is already implicated with the increase of more than 40 plant diseases; it dismantles plant defenses by chelating vital nutrients; and it reduces the bioavailability of nutrients in feed, which in turn can cause animal disorders. To properly evaluate these factors, we request access to the relevant USDA data.
I have studied plant pathogens for more than 50 years. We are now seeing an unprecedented trend of increasing plant and animal diseases and disorders. This pathogen may be instrumental to understanding and solving this problem. It deserves immediate attention with significant resources to avoid a general collapse of our critical agricultural infrastructure.
COL (Ret.) Don M. Huber
Emeritus Professor, Purdue University
APS Coordinator, USDA National Plant Disease Recovery System (NPDRS)
Asthma Tied to Bacterial Communities in the Airway
ScienceDaily (Feb. 19, 2011) — Asthma may have a surprising relationship with the composition of the species of bacteria that inhabit bronchial airways, a finding that could suggest new treatment or even potential cures for the common inflammatory disease, according to a new UCSF-led study.
Using new detection methods, researchers learned that the diversity of microbes inside the respiratory tract is far vaster than previously suspected — creating a complex and inter-connected microbial neighborhood that appears to be associated with asthma, and akin to what has also been found in inflammatory bowel disease, vaginitis, periodontitis, and possibly even obesity.[U1]—Contrary to popular belief, the scientists also learned that the airways are not necessarily entirely sterile environments, even in healthy people, while the airways of asthmatics are infected by a richer, more complex collection of bacteria. These findings could improve understanding of the biology of asthma, and potentially lead to new and much-needed therapies.-“People thought that asthma was caused by inhalation of allergens but this study shows that it may be more complicated than that — asthma may involve colonization of the airways by multiple bacteria,” said study co-author Homer Boushey, MD, a UCSF professor of medicine in the division of Pulmonary and Critical Care Medicine.–The study is published online in the Journal of Allergy and Clinical Immunology.–Asthma is one of the most common diseases in the world, with approximately 300 million asthmatics globally, including 24 million in the United States, according to the Centers for Disease Control. The disease has been on the rise for the last 60 years.–“It has gone from 3 percent of the population to slightly more than 8 percent of the population in the U.S.,” said Boushey. “It is most prevalent in western, developed nations — and we don’t know why.”[U2]—In recent years, scientists began studying communities of mixed-species microorganisms (microbiome) found in both diseased and healthy people to better understand their role in a variety of diseases. But research on the microbiome in respiratory disease is relatively uncharted terrain.–“We know fairly little about the diversity, complexity and collective function of bacteria living in the respiratory tract, and how they might contribute to diseases like asthma,” said Yvonne J. Huang, MD, the paper’s first author. She is a research fellow and clinical instructor in the UCSF Pulmonary Division.–“Traditionally, the airways have been thought to be sterile. However, this study suggests this is not the case. Certain asthma patients who require inhaled corticosteroid therapy possess a great abundance of bacteria compared to healthy individuals, and have an increased relative abundance of specific organisms that is correlated with greater sensitivity of their airways.”In their three-year pilot project, the scientists collected samples from the airway linings of 65 adults with mild to moderate asthma and 10 healthy subjects. Then, using a tool that can identify approximately 8,500 distinct groups of bacteria in a single assay, the scientists profiled the organisms present in each sample to look for relationships between bacterial community composition and clinical characteristics of the patients’ asthma.–The researchers found that bronchial airway samples from asthmatic patients contained far more bacteria than samples from healthy patients. The scientists also found greater bacterial diversity in the asthmatic patients who had the most hyper-responsive or sensitive airways (a feature of asthma).–“People have viewed asthma as a misdirected immune reaction to environmental exposures, but few have thought of it in the context of airway microbiota composition,” said senior author Susan Lynch, PhD, an assistant professor of medicine and director of the UCSF Colitis and Crohn’s Disease Microbiome Research Core in the division of gastroenterology.—“We took an ecological approach, considering the bacteria in the context of their microbial neighborhoods to identify relationships between characteristics of these communities and features of the disease…This new approach will help us to better understand the microbiota-host relationships that define human health.”–The authors say that further studies are needed to determine how these specific bacteria identified in the study may influence the cause and development of asthma.
The study was supported by the National Heart, Lung and Blood Institute and by the Strategic Asthma Basic Research Center at UCSF, supported by the Sandler Family Foundation. Huang was funded by a National Institutes of Health grant and by a UC Tobacco-related Disease Research Program award; Lynch receives research support from the NIH; Boushey is an ad-hoc consultant for KaloBios Pharmaceuticals, Inc., is on the advisory committee for Pharmaxis, is on ad-hoc advisory committees for GlaxoSmithKline and Merck, and receives research support from GlaxoSmithKline.
UCSF is a leading university dedicated to promoting health worldwide through advanced biomedical research, graduate-level education in the life sciences and health professions, and excellence in patient care.
Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of California – San Francisco, via EurekAlert!, a service of AAAS.–Journal Reference-Yvonne J. Huang, Craig E. Nelson, Eoin L. Brodie, Todd Z. DeSantis, Marshall S. Baek, Jane Liu, Tanja Woyke, Martin Allgaier, Jim Bristow, Jeanine P. Wiener-Kronish. Airway microbiota and bronchial hyperresponsiveness in patients with suboptimally controlled asthma. Journal of Allergy and Clinical Immunology, 2011; 127 (2): 372 DOI: 10.1016/j.jaci.2010.10.048
Bacteria to Blame in Asthma Attacks in Children, Research Suggests
ScienceDaily (Oct. 7, 2010) — Doctors have long known that viral infections can bring about asthma attacks and the shortness of breath, coughing, and wheezing associated with them. But while viral infections cannot be treated, scientists at the Danish Paediatric Asthma Centre (DPAC) at the University of Copenhagen and GentofteHospital have discovered that treatable bacterial infections can also cause asthma attacks. The discovery could revolutionize treatment.—“We found a significant relationship between bacterial infections and acute asthma attacks — above and beyond the expected relationship between viral infections and attacks,” says Hans Bisgaard, a professor of pediatrics at the DPAC.–The study examined 361 children between the ages of four weeks and three years to determine the presence of viral and bacterial infections during severe asthma attacks. The results conclude that the number of attacks was just as high in children with bacterial respiratory infections as in those with viral infections.—
Using antibiotics to treat asthma attacks?–“This indicates that bacteria can exacerbate asthma symptoms even if they aren’t infected with a virus,” Professor Bisgaard says. “The findings open up an entirely new method for treating severe asthma attacks. We can’t treat viral infections, but scientists will now look into whether treatment with antibiotics can help children when they have an asthma attack if they are also suffering from a bacterial infection.”–“Being able to use antibiotics to treat asthma attacks in children would be revolutionary,” Professor Bisgaard says.–The effects of antibiotics in treating asthma attacks will now be examined in large-scale, clinical study by the DPAC. The research has been published in British Medical Journalon 4 October 2010.
Story Source–The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by University of Copenhagen.
Journal Reference-H. Bisgaard, M. N. Hermansen, K. Bonnelykke, J. Stokholm, F. Baty, N. L. Skytt, J. Aniscenko, T. Kebadze, S. L. Johnston. Association of bacteria and viruses with wheezy episodes in young children: prospective birth cohort study. BMJ, 2010; 341 (oct04 1): c4978 DOI:10.1136/bmj.c4978
Blocking Effects Of Viral Infections May Prevent Asthma In Young Children
ScienceDaily (Nov. 9, 2007) — Babies who get severe respiratory viral infections are much more likely to suffer from asthma as they get older. Now researchers at Washington University School of Medicine in St. Louis have pinpointed a key step in the development of asthma in mice after a severe respiratory infection. They suggest that medications designed to interfere with this mechanism could potentially prevent many cases of childhood asthma. –“A severe respiratory infection in infancy greatly increases the risk of developing asthma,” says the study’s lead author Mitchell Grayson, M.D., assistant professor of medicine in the Division of Allergy and Immunology. “Less than one in 30 people who don’t suffer a severe respiratory infection as a baby develop asthma, but of those who do get these infections, one in five goes on to have asthma.”Grayson and colleagues recently published their research in the Journal of Experimental Medicine. They found that mice that developed asthma-like symptoms after a severe respiratory viral infection had an unusual immune reaction. During the infection, the mice produced antibodies and immune signals similar to those produced during an allergic response, instead of those typically made in response to infection. That started a chain reaction that led to asthma. The researchers propose that a similar reaction occurs in some people who suffer severe respiratory viral infections. –“We think genetically predisposed[U3]individuals will tend to have this kind of immune reaction to a severe respiratory viral infection,” Grayson says. “In those people an allergic-type response could be part of their antiviral immune response. That sets them up to make antibodies against a lot of environmental substances, like pet dander or pollen, and they can go on to develop allergies or asthma.” –Reports by the Centers for Disease Control and Prevention indicate that the number of people with asthma in the United States rose from approximately 7 million in 1980 to about 20 million in 2003. The reasons for this trend are unclear, Grayson indicates. But he suggests that a growing population density and the resulting increase in transmission of respiratory viral infections might be a cause. —Respiratory syncytial virus (RSV) is a common source of respiratory infections. In the United States nearly all children have been infected with RSV by two or three years of age. Severe RSV infections, typified by persistent coughing, wheezing and gasping for breath, send many thousands of children to the hospital each year. –To investigate the connection between severe respiratory viral infections and subsequent asthma, the researchers used mice genetically selected to have an asthma susceptibility and infected them with a virus similar to RSV. They found that severe respiratory infections in the mice induced an allergic-type immune response and ultimately caused long-term changes in the airways of the lungs that are hallmarks of chronic asthma. —The researchers discovered that certain immune cells in the mouse lungs reacted to severe viral infections by releasing compounds that instigated an inflammatory response. That in turn induced many lung airway cells to transform into mucus-producing cells, which can cause the obstruction of lung passages and shortness of breath characteristic of asthma. —The researchers found that interfering with this process by altering the immune cells or removing the inflammatory compounds they secreted prevented overgrowth of mucus-producing cells. –The findings promise a new approach to asthma prevention, according to Grayson. “This offers a different way of thinking about what happens in the development of asthma,” Grayson says. “It may be possible to prevent many cases of asthma and other chronic inflammatory airway diseases by stopping allergic-type antibody production after a severe viral infection in infants.”
Journal reference: Grayson MH, Cheung D, Rohlfing MM, Kitchens R, Spiegel DE, Tucker J, Battaile JT, Alevy Y, Yan L, Agapov E, Kim EY, Holtzman MJ. Induction of high-affinity IgE receptor on lung dendritic cells during viral infection leads to mucous cell metaplasia. Journal of Experimental Medicine 2007 Oct 29;204(11):2759-69. –Funding from the National Institute of Allergy and Infectious Disease, the National Heart, Lung and Blood Institute, Genentech Inc., Novartis International AG, the Martin Schaeffer Fund and the Alan A. and Edith L. Wolff Charitable Trust supported this research.–Story Source-The above story is reprinted (with editorial adaptations by ScienceDaily staff) from materials provided by Washington University in St. Louis, viaEurekAlert!, a service of AAAS.
Monsanto’s Technology Stewardship Agreement shifts responsibility to growers
for any and all losses, injury or damages resulting from the use of Monsanto seeds. There is no expiration date on the contract. The grower may terminate the contract, but: “Grower’s responsibilities and the other terms herein shall survive…”
This includes contamination of other farms. Growers are purchasing seed for Spring planting right now. Alfalfa, America’s 4th largest crop, is a particular problem because it is a perennial plant and the seeds may lie dormant in the ground for 10-20 years, and WILL contaminate non-GM plants. Contaminated alfalfa cannot be recalled from the environment. The liability burden can follow the grower for decades. Farmers must be made aware of the danger of being sued before they plant GM crops (especially alfalfa because it is used for cattle feed and will affect dairy farmers).
Currently, Australian organic farmer Steve Marsh, who lost his organic certification due to contamination, is suing his GM crop-growing neighbor for the GM contamination.
Contamination of processing equipment is another risk.
There is evidence from India that GM crops are linked to livestock deaths. The Monsanto Technology Stewardship Agreement contract holds growers responsible for injuries, so this is another potential consequence for farmers planting Monsanto GM crops to consider.
The Monsanto Technology Stewardship Agreement has another clause that farmers will find disturbing:
it appears that the growers agree that in order to sell their farm, the new purchaser must also sign a Monsanto Technology Stewardship Agreement. According to a top real estate broker, the contract places a covenant, condition or restriction (CCR) on the farmer’s land:
“GROWER AGREES: To accept and continue the obligations of this Monsanto Technology/Stewardship Agreement on any new land purchased or leased by Grower that has Seed planted on it by a previous owner or possessor of the land; and to notify in writing purchasers or lessees of land owned by Grower that has Seed planted on it that the Monsanto Technology is subject to this Monsanto Technology/Stewardship Agreement and they must have or obtain their own Monsanto Technology/Stewardship Agreement.”
Environmental attorney Anthony Patchett further elaborated on Monsanto’s contract in a letter that states“Monsanto’s agreement shifts all liability to the growers, including contamination issues or any potential future liability. All the grower receives is the price of the seed.” He further stated that this contract appears to be “Unconscionable”. Click here to view the letter.
For more information about the perils of contamination, please go to MorphCity.com to read the interview with alfalfa seed grower Phil Geertson who opposed Monsanto in the GM case heard in the Supreme Court last summer. Geertson said that Monsanto’s GM seeds are more expensive and after a few years, weeds can become tolerant to Roundup Ready and other glyphosate herbicides so farmers must return to conventional farming practices anyway. Therefore, there is no benefit to planting GM crops.
You can alert farmers to the hazard of growing GM crops and how growers can be hurt by Monsanto’s contract, if you would like to take action in opposing GM crops. Please share this article and video.
Recipe For Anti Bacterial Resistance
Antimicrobials: Silver (and Copper) Bullets to Kill Bacteria
ScienceDaily (Nov. 10, 2009) — Dana Filoti of the University of New Hampshire is presenting thin films of silver and copper she has developed that can kill bacteria and may one day help to cut down on hospital infections.The antimicrobial properties of silver and copper have been known for centuries — last year, the U.S. Environmental Protection Agency officially registered copper alloys, allowing them to be marketed with the label “kills 99.9% of bacteria within two hours.” Copper ions are known to penetrate bacteria and disrupt molecular pathways important for their survival.–Using zeolite ceramic structures, Filoti is testing the hypothesis that the combination of silver and copper might work synergistically to better kill bacteria, work that she will present on November 12 at a meeting of the scientific society AVS in San Jose. “The hard ceramic structure looks like Swiss cheese and inside the holes there are ions of silver and copper,” says Filoti.–By experimenting with the ratio of the two metals and the texture of the thin films, she has been able to reduce the amount of microbes present on the surface by 99 percent. One application of these antimicrobials, which Filoti is developing in partnership with a company in New Hampshire, is an antimicrobial face mask designed to protect against pathogens that cause many hospital-acquired infections.
Adapted from materials provided by American Institute of Physics
øøAn Idea to try—
Make a green drink with parsley and dandelion—add ¼ dropper of colloidal silver with this—should see an impact The mix will not be as refined— but you may still see results—-again this is something to try Dandelion and Parsley are loaded with copper—and enter the blood stream very efficiently—-colloids are extremely penetrating due to the way our system absorbs them—
[U1]LEAVE STARCHY foods –grains–pastas–rice–corn–polentas–cakes–pastries–bananas–grapes–watermelon-raisons–soy–soy derivatives–tvp-hvp-avp-hpp msg autolysed yeast—all Problematic
[U2]GMO and SOY might be the problem anddd the lack of nutrients in the soil
[U3]Genetically altered food would cause a predispposition to having asthma!!??? is this what they are saying!!!